Multiple Rad52-Mediated Homology-Directed Repair Mechanisms Are Required to Prevent Telomere Attrition-Induced Senescence in Saccharomyces cerevisiae
about
A sharp Pif1-dependent threshold separates DNA double-strand breaks from critically short telomeres.Rnr1, but not Rnr3, facilitates the sustained telomerase-dependent elongation of telomeres.In medio stat virtus: unanticipated consequences of telomere dysequilibrium.Histone depletion prevents telomere fusions in pre-senescent cells.Upregulation of dNTP Levels After Telomerase Inactivation Influences Telomerase-Independent Telomere Maintenance Pathway Choice in
P2860
Multiple Rad52-Mediated Homology-Directed Repair Mechanisms Are Required to Prevent Telomere Attrition-Induced Senescence in Saccharomyces cerevisiae
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Multiple Rad52-Mediated Homolo ...... ce in Saccharomyces cerevisiae
@ast
Multiple Rad52-Mediated Homolo ...... ce in Saccharomyces cerevisiae
@en
type
label
Multiple Rad52-Mediated Homolo ...... ce in Saccharomyces cerevisiae
@ast
Multiple Rad52-Mediated Homolo ...... ce in Saccharomyces cerevisiae
@en
prefLabel
Multiple Rad52-Mediated Homolo ...... ce in Saccharomyces cerevisiae
@ast
Multiple Rad52-Mediated Homolo ...... ce in Saccharomyces cerevisiae
@en
P2860
P1433
P1476
Multiple Rad52-Mediated Homolo ...... ce in Saccharomyces cerevisiae
@en
P2093
Clémence Claussin
Michael Chang
P2860
P304
P356
10.1371/JOURNAL.PGEN.1006176
P577
2016-07-18T00:00:00Z