Role of the glucocorticoid receptor in the recurrence of primary nephrotic syndrome.

about

Role of the glucocorticoid receptor in the recurrence of primary nephrotic syndrome.

Item

http://www.wikidata.org/entity/Q36081427

description

2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

Role of the glucocorticoid receptor in the recurrence of primary nephrotic syndrome.

@ast

Role of the glucocorticoid receptor in the recurrence of primary nephrotic syndrome.

@en

type

Item

label

Role of the glucocorticoid receptor in the recurrence of primary nephrotic syndrome.

@ast

Role of the glucocorticoid receptor in the recurrence of primary nephrotic syndrome.

@en

prefLabel

Role of the glucocorticoid receptor in the recurrence of primary nephrotic syndrome.

@ast

Role of the glucocorticoid receptor in the recurrence of primary nephrotic syndrome.

@en

P1476

Role of the glucocorticoid receptor in the recurrence of primary nephrotic syndrome.

@en

P2093

Ying Chen

http://www.w3.org/2001/XMLSchema#string

Yinyin Chen

http://www.w3.org/2001/XMLSchema#string

Yumei Liang

http://www.w3.org/2001/XMLSchema#string

Yuting Gong

http://www.w3.org/2001/XMLSchema#string

P2860

Upregulation of nuclear factor-related kappa B suggests a disorder of transcriptional regulation in minimal change nephrotic syndrome.

Dose- and time-dependent glucocorticoid receptor signaling in podocytes.

Glucocorticoid modulation of cytokine signaling.

Cytokine-effects on glucocorticoid receptor function: relevance to glucocorticoid resistance and the pathophysiology and treatment of major depression.

Familial forms of nephrotic syndrome.

New insights into the pathophysiology of idiopathic nephrotic syndrome.

Physiopathology of idiopathic nephrotic syndrome: lessons from glucocorticoids and epigenetic perspectives.

Treatment of nephrotic syndrome with adrenocorticotropic hormone (ACTH).

Recent advances and prognosis in idiopathic membranous nephropathy.

Inhibition of Jun N-terminal kinase (JNK) enhances glucocorticoid receptor-mediated function in mouse hippocampal HT22 cells.

P304

1556-1562

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.3892/ETM.2015.2665

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2015-07-30T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

282462379

http://www.w3.org/2001/XMLSchema#string

P698

26622525

http://www.w3.org/2001/XMLSchema#string

P932

4578096

http://www.w3.org/2001/XMLSchema#string