BLM ortholog, Sgs1, prevents aberrant crossing-over by suppressing formation of multichromatid joint molecules.
about
Frequent and efficient use of the sister chromatid for DNA double-strand break repair during budding yeast meiosisControlling meiotic recombinational repair - specifying the roles of ZMMs, Sgs1 and Mus81/Mms4 in crossover formationMechanisms of change in gene copy numberRecQ helicase translocates along single-stranded DNA with a moderate processivity and tight mechanochemical couplingHomologous recombination in DNA repair and DNA damage toleranceMus81 nuclease and Sgs1 helicase are essential for meiotic recombination in a protist lacking a synaptonemal complexMeiotic development in Caenorhabditis elegansTOPping off meiosisThe SMC-5/6 Complex and the HIM-6 (BLM) Helicase Synergistically Promote Meiotic Recombination Intermediate Processing and Chromosome Maturation during Caenorhabditis elegans MeiosisSmc5/6 coordinates formation and resolution of joint molecules with chromosome morphology to ensure meiotic divisionsJoint molecule resolution requires the redundant activities of MUS-81 and XPF-1 during Caenorhabditis elegans meiosisConditional inactivation of the DNA damage response gene Hus1 in mouse testis reveals separable roles for components of the RAD9-RAD1-HUS1 complex in meiotic chromosome maintenanceMammalian BTBD12 (SLX4) protects against genomic instability during mammalian spermatogenesisYeast Mph1 helicase dissociates Rad51-made D-loops: implications for crossover control in mitotic recombination.Yeast Pch2 promotes domainal axis organization, timely recombination progression, and arrest of defective recombinosomes during meiosisCsm4, in collaboration with Ndj1, mediates telomere-led chromosome dynamics and recombination during yeast meiosis.Genetic analysis of mlh3 mutations reveals interactions between crossover promoting factors during meiosis in baker's yeast.Crossover assurance and crossover interference are distinctly regulated by the ZMM proteins during yeast meiosis.Remodeling of the Rad51 DNA strand-exchange protein by the Srs2 helicase.Rad51 is an accessory factor for Dmc1-mediated joint molecule formation during meiosisYeast axial-element protein, Red1, binds SUMO chains to promote meiotic interhomologue recombination and chromosome synapsis.Polo-like kinase Cdc5 drives exit from pachytene during budding yeast meiosis.High-resolution mapping of meiotic crossovers and non-crossovers in yeast.RecQ helicase, Sgs1, and XPF family endonuclease, Mus81-Mms4, resolve aberrant joint molecules during meiotic recombination.Mus81/Mms4 endonuclease and Sgs1 helicase collaborate to ensure proper recombination intermediate metabolism during meiosis.Meiotic crossover control by concerted action of Rad51-Dmc1 in homolog template bias and robust homeostatic regulation.Multiple mechanisms limit meiotic crossovers: TOP3α and two BLM homologs antagonize crossovers in parallel to FANCMBLM helicase ortholog Sgs1 is a central regulator of meiotic recombination intermediate metabolismDelineation of joint molecule resolution pathways in meiosis identifies a crossover-specific resolvaseThe consequences of Rad51 overexpression for normal and tumor cellsFANCM limits meiotic crossoversTemporally and biochemically distinct activities of Exo1 during meiosis: double-strand break resection and resolution of double Holliday junctionsMlh1-Mlh3, a meiotic crossover and DNA mismatch repair factor, is a Msh2-Msh3-stimulated endonucleaseTopoisomerase 3alpha and RMI1 suppress somatic crossovers and are essential for resolution of meiotic recombination intermediates in Arabidopsis thalianaRNF212 is a dosage-sensitive regulator of crossing-over during mammalian meiosisAlignment of Homologous Chromosomes and Effective Repair of Programmed DNA Double-Strand Breaks during Mouse Meiosis Require the Minichromosome Maintenance Domain Containing 2 (MCMDC2) ProteinFault tolerance in protein interaction networks: stable bipartite subgraphs and redundant pathways.Effects of mutations in SGS1 and in genes functionally related to SGS1 on inverted repeat-stimulated spontaneous unequal sister-chromatid exchange in yeastMUS81 generates a subset of MLH1-MLH3-independent crossovers in mammalian meiosis.The Arabidopsis BLAP75/Rmi1 homologue plays crucial roles in meiotic double-strand break repair.
P2860
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P2860
BLM ortholog, Sgs1, prevents aberrant crossing-over by suppressing formation of multichromatid joint molecules.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
BLM ortholog, Sgs1, prevents a ...... ultichromatid joint molecules.
@ast
BLM ortholog, Sgs1, prevents a ...... ultichromatid joint molecules.
@en
type
label
BLM ortholog, Sgs1, prevents a ...... ultichromatid joint molecules.
@ast
BLM ortholog, Sgs1, prevents a ...... ultichromatid joint molecules.
@en
prefLabel
BLM ortholog, Sgs1, prevents a ...... ultichromatid joint molecules.
@ast
BLM ortholog, Sgs1, prevents a ...... ultichromatid joint molecules.
@en
P2093
P2860
P1433
P1476
BLM ortholog, Sgs1, prevents a ...... ultichromatid joint molecules.
@en
P2093
Andrew F Taylor
Jessica P Lao
Neil Hunter
Patty Yi-Hwa Hwang
Steve D Oh
P2860
P304
P356
10.1016/J.CELL.2007.05.035
P407
P577
2007-07-01T00:00:00Z