NK1.1+ cells and IL-22 regulate vaccine-induced protective immunity against challenge with Mycobacterium tuberculosis. - Wikidata - wikimedia - TriplyDB

NK1.1+ cells and IL-22 regulate vaccine-induced protective immunity against challenge with Mycobacterium tuberculosis.

NK1.1+ cells and IL-22 regulate vaccine-induced protective immunity against challenge with Mycobacterium tuberculosis.

http://www.wikidata.org/entity/Q36083904

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IMMUNOLOGY OF TUBERCULOSIS Interleukin-10 Family and Tuberculosis: An Old Story Renewed NK cells in immunotolerant organs Regulatory T-Cells at the Interface between Human Host and Pathogens in Infectious Diseases and Vaccination Quest for correlates of protection against tuberculosis Boosting vaccine efficacy the natural (killer) way Memory-Like Antigen-Specific Human NK Cells from TB Pleural Fluids Produced IL-22 in Response to IL-15 or Mycobacterium tuberculosis Antigens IL-21-dependent expansion of memory-like NK cells enhances protective immune responses against Mycobacterium tuberculosis.Directing traffic: IL-17 and IL-22 coordinate pulmonary immune defense.IL-22 is mainly produced by IFNγ-secreting cells but is dispensable for host protection against Mycobacterium tuberculosis infection.The contribution of non-conventional T cells and NK cells in the mycobacterial-specific IFNγ response in Bacille Calmette-Guérin (BCG)-immunized infants.Different subsets of macrophages in patients with new onset tuberculous pleural effusion.Interleukin-22: immunobiology and pathology.A rho GDP dissociation inhibitor produced by apoptotic T-cells inhibits growth of Mycobacterium tuberculosis.NK-CD11c+ Cell Crosstalk in Diabetes Enhances IL-6-Mediated Inflammation during Mycobacterium tuberculosis Infection.Nasal vaccination stimulates CD8(+) T cells for potent protection against mucosal Brucella melitensis challenge A TLR9 agonist promotes IL-22-dependent pancreatic islet allograft survival in type 1 diabetic mice.Interleukin 22 inhibits intracellular growth of Mycobacterium tuberculosis by enhancing calgranulin A expression.Th17 cell based vaccines in mucosal immunity.TB or not TB: that is no longer the question.Novel role for IL-22 in protection during chronic Mycobacterium tuberculosis HN878 infection.Cytokines and Chemokines in Mycobacterium tuberculosis Infection.CD4+ and γδ T Cells are the main Producers of IL-22 and IL-17A in Lymphocytes from Mycobacterium bovis-infected Cattle.Memory of Natural Killer Cells: A New Chance against Mycobacterium tuberculosis?Protective immunity against tuberculosis: what does it look like and how do we find it?Strategies to Improve Vaccine Efficacy against Tuberculosis by Targeting Innate Immunity.IL-21 regulates NK cell responses during Mycobacterium tuberculosis infection.New Players in Immunity to Tuberculosis: The Host Microbiome, Lung Epithelium, and Innate Immune Cells.Autologous tumor cells/bacillus Calmette-Guérin/formalin-based novel breast cancer vaccine induces an immune antitumor response.

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NK1.1+ cells and IL-22 regulate vaccine-induced protective immunity against challenge with Mycobacterium tuberculosis.

http://www.wikidata.org/entity/Q36083904

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

@zh-hans

2012年学术文章

@zh-my

2012年学术文章

@zh-sg

2012年學術文章

@yue

2012年學術文章

@zh

2012年學術文章

@zh-hant

name

NK1.1+ cells and IL-22 regulat ...... th Mycobacterium tuberculosis.

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NK1.1+ cells and IL-22 regulat ...... th Mycobacterium tuberculosis.

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type

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NK1.1+ cells and IL-22 regulat ...... th Mycobacterium tuberculosis.

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NK1.1+ cells and IL-22 regulat ...... th Mycobacterium tuberculosis.

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prefLabel

NK1.1+ cells and IL-22 regulat ...... th Mycobacterium tuberculosis.

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NK1.1+ cells and IL-22 regulat ...... th Mycobacterium tuberculosis.

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JIMMUNOL.1102833

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Journal of Immunology

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NK1.1+ cells and IL-22 regulat ...... th Mycobacterium tuberculosis.

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P2093

Amanda B Barnes

http://www.w3.org/2001/XMLSchema#string

Amy Tvinnereim

http://www.w3.org/2001/XMLSchema#string

Ankita Garg Jaiswal

http://www.w3.org/2001/XMLSchema#string

Padmaja Paidipally

http://www.w3.org/2001/XMLSchema#string

Peter F Barnes

http://www.w3.org/2001/XMLSchema#string

Ramakrishna Vankayalapati

http://www.w3.org/2001/XMLSchema#string

Rohan Dhiman

http://www.w3.org/2001/XMLSchema#string

Sivakumar Periasamy

http://www.w3.org/2001/XMLSchema#string

P2860

Activation of NK cells and T cells by NKG2D, a receptor for stress-inducible MICA

Interleukin (IL)-22, a novel human cytokine that signals through the interferon receptor-related proteins CRF2-4 and IL-22R

ULBPs, novel MHC class I-related molecules, bind to CMV glycoprotein UL16 and stimulate NK cytotoxicity through the NKG2D receptor

The biological functions of T helper 17 cell effector cytokines in inflammation

Stage 3 immature human natural killer cells found in secondary lymphoid tissue constitutively and selectively express the TH 17 cytokine interleukin-22

The NKp46 receptor contributes to NK cell lysis of mononuclear phagocytes infected with an intracellular bacterium

IL-22 produced by human NK cells inhibits growth of Mycobacterium tuberculosis by enhancing phagolysosomal fusion

A human natural killer cell subset provides an innate source of IL-22 for mucosal immunity

Interleukin-22 mediates early host defense against attaching and effacing bacterial pathogens

Biology of interleukin-22.

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897-905

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scholarly article

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10.4049/JIMMUNOL.1102833

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P407

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2

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189

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2012-06-18T00:00:00Z

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22711885

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Akafuba bulwadde

Mycobacterium tuberculosis

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3392427

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