Pellino-1 selectively regulates epithelial cell responses to rhinovirus. - Wikidata - wikimedia - TriplyDB

Pellino-1 selectively regulates epithelial cell responses to rhinovirus.

Pellino-1 selectively regulates epithelial cell responses to rhinovirus.

http://www.wikidata.org/entity/Q36086724

about

Phosphoinositide-3 kinase inhibition modulates responses to rhinovirus by mechanisms that are predominantly independent of autophagy Functional conservation of an ancestral Pellino protein in helminth species Pellino-1 Positively Regulates Toll-like Receptor (TLR) 2 and TLR4 Signaling and Is Suppressed upon Induction of Endotoxin Tolerance.Human rhinovirus-induced inflammatory responses are inhibited by phosphatidylserine containing liposomes Pellino-1 confers chemoresistance in lung cancer cells by upregulating cIAP2 through Lys63-mediated polyubiquitination.Airway gene expression of IL-1 pathway mediators predicts exacerbation risk in obstructive airway disease.Rhinoviral infection and asthma: the detection and management of rhinoviruses by airway epithelial cells.The roles of Pellino E3 ubiquitin ligases in immunity.Molecular and physiological roles of Pellino E3 ubiquitin ligases in immunity.E3 ubiquitin ligases Pellinos as regulators of pattern recognition receptor signaling and immune responses.Interaction of CSFV E2 protein with swine host factors as detected by yeast two-hybrid system Novel immune genes associated with excessive inflammatory and antiviral responses to rhinovirus in COPD.Differential modulation of Helicobacter pylori lipopolysaccharide-mediated TLR2 signaling by individual Pellino proteins.

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P2860

Pellino-1 selectively regulates epithelial cell responses to rhinovirus.

http://www.wikidata.org/entity/Q36086724

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

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2012年論文

@zh-tw

2012年论文

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2012年论文

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2012年论文

@zh-cn

name

Pellino-1 selectively regulates epithelial cell responses to rhinovirus.

@ast

Pellino-1 selectively regulates epithelial cell responses to rhinovirus.

@en

type

label

Pellino-1 selectively regulates epithelial cell responses to rhinovirus.

@ast

Pellino-1 selectively regulates epithelial cell responses to rhinovirus.

@en

prefLabel

Pellino-1 selectively regulates epithelial cell responses to rhinovirus.

@ast

Pellino-1 selectively regulates epithelial cell responses to rhinovirus.

@en

P1433

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P1433

Journal of Virology

P1476

Pellino-1 selectively regulates epithelial cell responses to rhinovirus

@en

P2093

Harold G de Bruin

http://www.w3.org/2001/XMLSchema#string

Irene H Heijink

http://www.w3.org/2001/XMLSchema#string

Julie A Bennett

http://www.w3.org/2001/XMLSchema#string

Lisa C Parker

http://www.w3.org/2001/XMLSchema#string

Maarten van den Berge

http://www.w3.org/2001/XMLSchema#string

Moira K Whyte

http://www.w3.org/2001/XMLSchema#string

P2860

Pellino proteins are more than scaffold proteins in TLR/IL-1R signalling: a role as novel RING E3-ubiquitin-ligases

Pellino 1 is required for interleukin-1 (IL-1)-mediated signaling through its interaction with the IL-1 receptor-associated kinase 4 (IRAK4)-IRAK-tumor necrosis factor receptor-associated factor 6 (TRAF6) complex

Pellino Proteins Contain a Cryptic FHA Domain that Mediates Interaction with Phosphorylated IRAK1

Peli1 facilitates TRIF-dependent Toll-like receptor signaling and proinflammatory cytokine production

Toll-like receptors and their crosstalk with other innate receptors in infection and immunity

Co-ordinated role of TLR3, RIG-I and MDA5 in the innate response to rhinovirus in bronchial epithelium

Diesel exhaust particles override natural injury-limiting pathways in the lung.

RIP1 is an essential mediator of Toll-like receptor 3-induced NF-kappa B activation.

Kinase-active interleukin-1 receptor-associated kinases promote polyubiquitination and degradation of the Pellino family: direct evidence for PELLINO proteins being ubiquitin-protein isopeptide ligases.

Experimental rhinovirus infection as a human model of chronic obstructive pulmonary disease exacerbation.

P304

6595-6604

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P31

scholarly article

P356

10.1128/JVI.06755-11

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P433

12

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P478

86

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P577

2012-04-18T00:00:00Z

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22514342

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P932

3393558

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