Transcription factor FoxO1, the dominant mediator of muscle wasting in chronic kidney disease, is inhibited by microRNA-486. - Wikidata - wikimedia - TriplyDB

Transcription factor FoxO1, the dominant mediator of muscle wasting in chronic kidney disease, is inhibited by microRNA-486.

Transcription factor FoxO1, the dominant mediator of muscle wasting in chronic kidney disease, is inhibited by microRNA-486.

http://www.wikidata.org/entity/Q36087285

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Cancer Cachexia and MicroRNAs Noncoding RNAs, Emerging Regulators of Skeletal Muscle Development and Diseases Skeletal Muscle Regeneration and Oxidative Stress Are Altered in Chronic Kidney Disease MicroRNAs as regulators and mediators of forkhead box transcription factors function in human cancers MicroRNA-29 induces cellular senescence in aging muscle through multiple signaling pathways MicroRNA-486-dependent modulation of DOCK3/PTEN/AKT signaling pathways improves muscular dystrophy-associated symptoms Mega roles of microRNAs in regulation of skeletal muscle health and disease Cancer affects microRNA expression, release, and function in cardiac and skeletal muscle Melanocortin antagonism ameliorates muscle wasting and inflammation in chronic kidney disease.Akt1-mediated fast/glycolytic skeletal muscle growth attenuates renal damage in experimental kidney disease.Mechanisms of muscle wasting in chronic kidney disease MicroRNA-486-5p is an erythroid oncomiR of the myeloid leukemias of Down syndrome Regulation of autophagy and the ubiquitin-proteasome system by the FoxO transcriptional network during muscle atrophy Comparative Analyses between Skeletal Muscle miRNAomes from Large White and Min Pigs Revealed MicroRNAs Associated with Postnatal Muscle Hypertrophy.CKD Stimulates Muscle Protein Loss Via Rho-associated Protein Kinase 1 Activation.Circulating micrornas associated with glycemic impairment and progression in Asian Indians Selection of urinary sediment miRNAs as specific biomarkers of IgA nephropathy.The role of microRNAs in skeletal muscle health and disease Chronic kidney disease accelerates endothelial barrier dysfunction in a mouse model of an arteriovenous fistula.Extracellular vesicle microRNAs mediate skeletal muscle myogenesis and disease Muscle-Enriched MicroRNAs Isolated from Whole Blood Are Regulated by Exercise and Are Potential Biomarkers of Cardiorespiratory Fitness.Role of microRNAs in skeletal muscle hypertrophy.MicroRNA in myogenesis and muscle atrophy Suppression of muscle wasting by the plant-derived compound ursolic acid in a model of chronic kidney disease.Muscle wasting from kidney failure-a model for catabolic conditions.MicroRNA in skeletal muscle development, growth, atrophy, and disease.Bone is Not Alone: the Effects of Skeletal Muscle Dysfunction in Chronic Kidney Disease.F-BOX proteins in cancer cachexia and muscle wasting: Emerging regulators and therapeutic opportunities.Recombinant myostatin reduces highly expressed microRNAs in differentiating C2C12 cells.Chronic kidney disease induces autophagy leading to dysfunction of mitochondria in skeletal muscle.Muscle atrophy in patients with Type 2 Diabetes Mellitus: roles of inflammatory pathways, physical activity and exercise.Posttranscriptional regulation of FOXO expression: microRNAs and beyond.Clinical relevance of sarcopenia in chronic kidney disease.Direct repression of the oncogene CDK4 by the tumor suppressor miR-486-5p in non-small cell lung cancer Noncoding RNAs in the regulation of skeletal muscle biology in health and disease.MicroRNA-23a and MicroRNA-27a Mimic Exercise by Ameliorating CKD-Induced Muscle Atrophy.Does proteasome regulate the level of microRNA-1 in cardiomyocytes? Application to anoxia-reoxygenation.The miR-486-5p plays a causative role in prostate cancer through negative regulation of multiple tumor suppressor pathways.Uremic osteoporosis.Obestatin controls the ubiquitin-proteasome and autophagy-lysosome systems in glucocorticoid-induced muscle cell atrophy.

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P2860

Transcription factor FoxO1, the dominant mediator of muscle wasting in chronic kidney disease, is inhibited by microRNA-486.

http://www.wikidata.org/entity/Q36087285

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Biruh Workeneh

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Jing Xu

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Rongshan Li

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Xiaonan Wang

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Yanlan Dong

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Zhaoyong Hu

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P2860

Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy

microRNA-1 and microRNA-206 regulate skeletal muscle satellite cell proliferation and differentiation by repressing Pax7

Distinctive patterns of microRNA expression in primary muscular disorders

A Foxo/Notch pathway controls myogenic differentiation and fiber type specification

Identification of tissue-specific microRNAs from mouse

Protein degradation by the ubiquitin-proteasome pathway in normal and disease states

Multiple types of skeletal muscle atrophy involve a common program of changes in gene expression

Identification of ubiquitin ligases required for skeletal muscle atrophy

The widespread impact of mammalian MicroRNAs on mRNA repression and evolution

Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy

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