Histamine receptor H1 is required for TCR-mediated p38 MAPK activation and optimal IFN-gamma production in mice.
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Quetiapine, an atypical antipsychotic, is protective against autoimmune-mediated demyelination by inhibiting effector T cell proliferationIdentification of Orch3, a locus controlling dominant resistance to autoimmune orchitis, as kinesin family member 1CEndothelial histamine H1 receptor signaling reduces blood-brain barrier permeability and susceptibility to autoimmune encephalomyelitis.Multiple linked quantitative trait loci within the Tmevd2/Eae3 interval control the severity of experimental allergic encephalomyelitis in DBA/2J mice.A single nucleotide polymorphism in Tyk2 controls susceptibility to experimental allergic encephalomyelitisHistamine H(3) receptor integrates peripheral inflammatory signals in the neurogenic control of immune responses and autoimmune disease susceptibilityIdentification of genetic determinants of the sexual dimorphism in CNS autoimmunity.Combination treatment of mice with CRx-153 (nortriptyline and desloratadine) decreases the severity of experimental autoimmune encephalomyelitis.Activation of p38 MAPK in CD4 T cells controls IL-17 production and autoimmune encephalomyelitisThe H1-receptor antagonist cetirizine ameliorates high-fat diet-induced glucose intolerance in male C57BL/6 mice, but not diabetes outcome in female non-obese diabetic (NOD) mice.Copy number variation in Y chromosome multicopy genes is linked to a paternal parent-of-origin effect on CNS autoimmune disease in female offspringHistamine H(1) receptor signaling regulates effector T cell responses and susceptibility to coxsackievirus B3-induced myocarditisHistamine H4 receptor optimizes T regulatory cell frequency and facilitates anti-inflammatory responses within the central nervous system.Cutting edge: histamine is required for IL-4-driven eosinophilic allergic responses.Exacerbation of autoimmune neuroinflammation by dietary sodium is genetically controlled and sex specific.H(1)R expression by CD11B(+) cells is not required for susceptibility to experimental allergic encephalomyelitisExpression and function of histamine and its receptors in atopic dermatitisCombinatorial roles for histamine H1-H2 and H3-H4 receptors in autoimmune inflammatory disease of the central nervous system.IL-17 producing mast cells promote the expansion of myeloid-derived suppressor cells in a mouse allergy model of colorectal cancerAutoimmune disease-associated histamine receptor H1 alleles exhibit differential protein trafficking and cell surface expression.Nod1 Limits Colitis-Associated Tumorigenesis by Regulating IFN-γ Production.Systemic lack of canonical histamine receptor signaling results in increased resistance to autoimmune encephalomyelitis.G protein-coupled receptors as therapeutic targets for multiple sclerosis.Cutting edge: the Y chromosome controls the age-dependent experimental allergic encephalomyelitis sexual dimorphism in SJL/J miceThe Y chromosome as a regulatory element shaping immune cell transcriptomes and susceptibility to autoimmune disease.Accelerated remyelination during inflammatory demyelination prevents axonal loss and improves functional recoveryG proteins Gαi1/3 are critical targets for Bordetella pertussis toxin-induced vasoactive amine sensitization.Sex-specific control of central nervous system autoimmunity by p38 mitogen-activated protein kinase signaling in myeloid cells.Histamine H₂ receptor signaling × environment interactions determine susceptibility to experimental allergic encephalomyelitis.Histamine and neuroinflammation: insights from murine experimental autoimmune encephalomyelitis.The emerging role of p38 mitogen-activated protein kinase in multiple sclerosis and its models.TLR-induced activation of neutrophils promotes histamine production via a PI3 kinase dependent mechanism.Histamine deficiency promotes accumulation of immunosuppressive immature myeloid cells and growth of murine gliomasSynergy of TLR2 and H1R on Cox-2 Activation in Pulpal Cells.Molecular Genetic and Epigenetic Basis of Multiple Sclerosis.Modeling month-season of birth as a risk factor in mouse models of chronic disease: from multiple sclerosis to autoimmune encephalomyelitis.Histamine and Histamine Receptors in Allergic Dermatitis.Histamine regulates autoreactive T cell activation and adhesiveness in inflamed brain microcirculation.Polyacrylic acid-coated and non-coated iron oxide nanoparticles induce cytokine activation in human blood cells through TAK1, p38 MAPK and JNK pro-inflammatory pathways.Advances in the molecular imaging of multiple sclerosis
P2860
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P2860
Histamine receptor H1 is required for TCR-mediated p38 MAPK activation and optimal IFN-gamma production in mice.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Histamine receptor H1 is requi ...... IFN-gamma production in mice.
@ast
Histamine receptor H1 is requi ...... IFN-gamma production in mice.
@en
type
label
Histamine receptor H1 is requi ...... IFN-gamma production in mice.
@ast
Histamine receptor H1 is requi ...... IFN-gamma production in mice.
@en
prefLabel
Histamine receptor H1 is requi ...... IFN-gamma production in mice.
@ast
Histamine receptor H1 is requi ...... IFN-gamma production in mice.
@en
P2093
P2860
P356
P1476
Histamine receptor H1 is requi ...... IFN-gamma production in mice.
@en
P2093
Cory Teuscher
Elizabeth P Blankenhorn
Graeme Milligan
James F Zachary
Mercedes Rincon
Rajkumar Noubade
Roxana del Rio
P2860
P304
P356
10.1172/JCI32792
P407
P577
2007-11-01T00:00:00Z