Activation of the PTEN/mTOR/STAT3 pathway in breast cancer stem-like cells is required for viability and maintenance - Wikidata - wikimedia - TriplyDB

Activation of the PTEN/mTOR/STAT3 pathway in breast cancer stem-like cells is required for viability and maintenance

Activation of the PTEN/mTOR/STAT3 pathway in breast cancer stem-like cells is required for viability and maintenance

http://www.wikidata.org/entity/Q36092560

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The role of PTEN/Akt/PI3K signaling in the maintenance and viability of prostate cancer stem-like cell populations Stem-like features of cancer cells on their way to metastasis Anti-Cancer Stem-like Cell Compounds in Clinical Development - An Overview and Critical Appraisal Cancer stem cells: a metastasizing menace!Redox Modulating NRF2: A Potential Mediator of Cancer Stem Cell Resistance Molecular Dimensions of Gastric Cancer: Translational and Clinical Perspectives Targeting colorectal cancer stem cells using curcumin and curcumin analogues: insights into the mechanism of the therapeutic efficacy Redox Regulation in Cancer Stem Cells PI3K/Akt/mTOR signaling pathway in cancer stem cells: from basic research to clinical application Side population cells as prototype of chemoresistant, tumor-initiating cells Identifying and targeting tumor-initiating cells in the treatment of breast cancer Oncogenic role and therapeutic target of leptin signaling in breast cancer and cancer stem cells Long non-coding RNAs in gastric cancer: versatile mechanisms and potential for clinical translation Glycerol-3-phosphate acyltransferase-1 upregulation by O-GlcNAcylation of Sp1 protects against hypoxia-induced mouse embryonic stem cell apoptosis via mTOR activation Judicious Toggling of mTOR Activity to Combat Insulin Resistance and Cancer: Current Evidence and Perspectives In vitro models of cancer stem cells and clinical applications Reverse phase protein microarrays advance to use in clinical trials Sustained proliferation in cancer: Mechanisms and novel therapeutic targets Regulation of mammary stem/progenitor cells by PTEN/Akt/beta-catenin signaling SALL4, a stem cell factor, affects the side population by regulation of the ATP-binding cassette drug transport genes Galiellalactone inhibits stem cell-like ALDH-positive prostate cancer cells Low concentrations of metformin selectively inhibit CD133⁺ cell proliferation in pancreatic cancer and have anticancer action SNAI1-mediated epithelial-mesenchymal transition confers chemoresistance and cellular plasticity by regulating genes involved in cell death and stem cell maintenance Metformin, a diabetes drug, eliminates tumor-initiating hepatocellular carcinoma cells Response of breast cancer cells and cancer stem cells to metformin and hyperthermia alone or combined The Synthetic β-Nitrostyrene Derivative CYT-Rx20 Inhibits Esophageal Tumor Growth and Metastasis via PI3K/AKT and STAT3 Pathways EMT, cancer stem cells and drug resistance: an emerging axis of evil in the war on cancer Niclosamide and its analogs are potent inhibitors of Wnt/β-catenin, mTOR and STAT3 signaling in ovarian cancer.Obesity and cancer pathogenesis.Towards the Biological Understanding of CTC: Capture Technologies, Definitions and Potential to Create Metastasis.Asymmetric cancer cell division regulated by AKT Studying cancer stem cell dynamics on PDMS surfaces for microfluidics device design.Cancer stem/progenitor cell active compound 8-quinolinol in combination with paclitaxel achieves an improved cure of breast cancer in the mouse model Detection of treatment-induced changes in signaling pathways in gastrointestinal stromal tumors using transcriptomic data.Down-regulation of the fetal stem cell factor SOX17 by H33342: a mechanism responsible for differential gene expression in breast cancer side population cells.Stemness in Cancer: Stem Cells, Cancer Stem Cells, and Their Microenvironment.A novel mouse model of inflammatory bowel disease links mammalian target of rapamycin-dependent hyperproliferation of colonic epithelium to inflammation-associated tumorigenesis Nuclear beta-catenin and CD44 upregulation characterize invasive cell populations in non-aggressive MCF-7 breast cancer cells.Dietary suppression of the mammary CD29(hi)CD24(+) epithelial subpopulation and its cytokine/chemokine transcriptional signatures modifies mammary tumor risk in MMTV-Wnt1 transgenic mice Stem cell-like transcriptional reprogramming mediates metastatic resistance to mTOR inhibition.

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Activation of the PTEN/mTOR/STAT3 pathway in breast cancer stem-like cells is required for viability and maintenance

http://www.wikidata.org/entity/Q36092560

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2007 nî lūn-bûn

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2007年論文

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Activation of the PTEN/mTOR/ST ...... for viability and maintenance

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Activation of the PTEN/mTOR/ST ...... for viability and maintenance

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Proceedings of the National Academy of Sciences of the United States of America

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Activation of the PTEN/mTOR/ST ...... for viability and maintenance

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Emanuel Petricoin

http://www.w3.org/2001/XMLSchema#string

Hao Zhang

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Jiangbing Zhou

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Jianghong Deng

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Julia Wulfkuhle

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Lance A Liotta

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Peihua Gu

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Yanqin Yang

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Ying Zhang

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P2860

A distinct "side population" of cells with high drug efflux capacity in human tumor cells

Prospective identification of tumorigenic breast cancer cells

Identification and expansion of human colon-cancer-initiating cells

A human colon cancer cell capable of initiating tumour growth in immunodeficient mice

Stem cells, cancer, and cancer stem cells

Identification of human brain tumour initiating cells

A cell initiating human acute myeloid leukaemia after transplantation into SCID mice

Serine phosphorylation and maximal activation of STAT3 during CNTF signaling is mediated by the rapamycin target mTOR

Identification of pancreatic cancer stem cells

Regulation of c-Met-dependent gene expression by PTEN

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104

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Joseph B Margolick

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5934335

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