Some strains of Plasmodium falciparum, a human malaria parasite, evade the complement-like system of Anopheles gambiae mosquitoes - Wikidata - wikimedia - TriplyDB

Some strains of Plasmodium falciparum, a human malaria parasite, evade the complement-like system of Anopheles gambiae mosquitoes

Some strains of Plasmodium falciparum, a human malaria parasite, evade the complement-like system of Anopheles gambiae mosquitoes

http://www.wikidata.org/entity/Q36094285

about

The CLIP-domain serine protease homolog SPCLIP1 regulates complement recruitment to microbial surfaces in the malaria mosquito Anopheles gambiae The remarkable journey of adaptation of the Plasmodium falciparum malaria parasite to New World anopheline mosquitoes The human malaria parasite Pfs47 gene mediates evasion of the mosquito immune system Multiple pathways for Plasmodium ookinete invasion of the mosquito midgut Immune Escape Strategies of Malaria Parasites An Evolution-Based Screen for Genetic Differentiation between Anopheles Sister Taxa Enriches for Detection of Functional Immune Factors Non-genetic determinants of mosquito competence for malaria parasites From genome-scale data to models of infectious disease: A Bayesian network-based strategy to drive model development.Immune resistance and tolerance strategies in malaria vector and non-vector mosquitoes NF-κB-Like Signaling Pathway REL2 in Immune Defenses of the Malaria Vector Anopheles gambiae.In depth annotation of the Anopheles gambiae mosquito midgut transcriptome.The Plasmodium bottleneck: malaria parasite losses in the mosquito vector Sex-partitioning of the Plasmodium falciparum stage V gametocyte proteome provides insight into falciparum-specific cell biology An impossible journey? The development of Plasmodium falciparum NF54 in Culex quinquefasciatus.The JNK pathway is a key mediator of Anopheles gambiae antiplasmodial immunity.Profile of Carolina Barillas-Mury Malaria parasite Pfs47 disrupts JNK signaling to escape mosquito immunity Biophysical analysis of anopheles gambiae leucine-rich repeat proteins APL1A1, APL1B [corrected] and APL1C and their interaction with LRIM1.An overview of malaria transmission from the perspective of Amazon Anopheles vectors.Plasmodium falciparum mating patterns and mosquito infectivity of natural isolates of gametocytes.A New Role of the Mosquito Complement-like Cascade in Male Fertility in Anopheles gambiae.A mosquito lipoxin/lipocalin complex mediates innate immune priming in Anopheles gambiae Plant-Mediated Effects on Mosquito Capacity to Transmit Human Malaria.Plasmodium yoelii nigeriensis (N67) Is a Robust Animal Model to Study Malaria Transmission by South American Anopheline Mosquitoes.Molecular Analysis of Pfs47-Mediated Plasmodium Evasion of Mosquito Immunity.Plasmodium evasion of mosquito immunity and global malaria transmission: The lock-and-key theory Variation in susceptibility of African Plasmodium falciparum malaria parasites to TEP1 mediated killing in Anopheles gambiae mosquitoes A serine protease homolog negatively regulates TEP1 consumption in systemic infections of the malaria vector Anopheles gambiae CLIPB8 is part of the prophenoloxidase activation system in Anopheles gambiae mosquitoes Measuring changes in Plasmodium falciparum transmission: precision, accuracy and costs of metrics.A compendium of molecules involved in vector-pathogen interactions pertaining to malaria.Culture-adapted Plasmodium falciparum isolates from UK travellers: in vitro drug sensitivity, clonality and drug resistance markers.Genome-block expression-assisted association studies discover malaria resistance genes in Anopheles gambiae The role of hemocytes in Anopheles gambiae antiplasmodial immunity.The Anopheles innate immune system in the defense against malaria infection.Artonin E induces p53-independent G1 cell cycle arrest and apoptosis through ROS-mediated mitochondrial pathway and livin suppression in MCF-7 cells.Plasmodium berghei P47 is essential for ookinete protection from the Anopheles gambiae complement-like response.Alice in microbes' land: adaptations and counter-adaptations of vector-borne parasitic protozoa and their hosts.The structure and function of thioester-containing proteins in arthropods.Late-phase immune responses limiting oocyst survival are independent of TEP1 function yet display strain specific differences in Anopheles gambiae.

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Some strains of Plasmodium falciparum, a human malaria parasite, evade the complement-like system of Anopheles gambiae mosquitoes

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2012 nî lūn-bûn

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PNAS.1121183109

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Proceedings of the National Academy of Sciences of the United States of America

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Some strains of Plasmodium fal ...... f Anopheles gambiae mosquitoes

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Alvaro Molina-Cruz

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Ashley Haile

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Carolina Barillas-Mury

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Corrie Ortega

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Ekua Abban

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Janneth Rodrigues

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Randall J DeJong

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P2860

Quantitative trait loci in Anopheles gambiae controlling the encapsulation response against Plasmodium cynomolgi Ceylon

Host genotype by parasite genotype interactions underlying the resistance of anopheline mosquitoes to Plasmodium falciparum

Human malaria parasites in continuous culture

Quantitative trait loci for refractoriness of Anopheles gambiae to Plasmodium cynomolgi B.

Caspar controls resistance to Plasmodium falciparum in diverse anopheline species

Mosquito immune responses and compatibility between Plasmodium parasites and anopheline mosquitoes

Fine pathogen discrimination within the APL1 gene family protects Anopheles gambiae against human and rodent malaria species

Complement-like protein TEP1 is a determinant of vectorial capacity in the malaria vector Anopheles gambiae.

Adaptive divergence between incipient species of Anopheles gambiae increases resistance to Plasmodium.

Two mosquito LRR proteins function as complement control factors in the TEP1-mediated killing of Plasmodium.

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28

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109

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Giovanna Jaramillo-Gutierrez

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2012-05-23T00:00:00Z

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225059183

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22623529

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Anopheles gambiae

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3396512

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