Mechanistic role for a novel glucocorticoid-KLF11 (TIEG2) protein pathway in stress-induced monoamine oxidase A expression - Wikidata - wikimedia - TriplyDB

Mechanistic role for a novel glucocorticoid-KLF11 (TIEG2) protein pathway in stress-induced monoamine oxidase A expression

Mechanistic role for a novel glucocorticoid-KLF11 (TIEG2) protein pathway in stress-induced monoamine oxidase A expression

http://www.wikidata.org/entity/Q36097919

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Molecular mechanism of monoamine oxidase A gene regulation under inflammation and ischemia-like conditions: key roles of the transcription factors GATA2, Sp1 and TBP Krüppel-like factor 11 regulates the expression of metabolic genes via an evolutionarily conserved protein interaction domain functionally disrupted in maturity onset diabetes of the young.Monoamine oxidase A upregulated by chronic intermittent hypoxia activates indoleamine 2,3-dioxygenase and neurodegeneration.Krüpple-like factors in the central nervous system: novel mediators in stroke.Role for Krüppel-like transcription factor 11 in mesenchymal cell function and fibrosis Feed-forward transcriptional programming by nuclear receptors: regulatory principles and therapeutic implications Krüppel-like factors are effectors of nuclear receptor signaling.Evidence revealing deregulation of the KLF11-MAO A pathway in association with chronic stress and depressive disorders Early life environmental and pharmacological stressors result in persistent dysregulations of the serotonergic system.Kruppel-like factor 4 is critical for transcriptional control of cardiac mitochondrial homeostasis.M30 Antagonizes Indoleamine 2,3-Dioxygenase Activation and Neurodegeneration Induced by Corticosterone in the Hippocampus Chronic Social Stress and Ethanol Increase Expression of KLF11, a Cell Death Mediator, in Rat Brain KLF11 mediates PPARγ cerebrovascular protection in ischaemic stroke.The glucocorticoid receptor and KLF15 regulate gene expression dynamics and integrate signals through feed-forward circuitry.Elevated Monoamine Oxidase-A Distribution Volume in Borderline Personality Disorder Is Associated With Severity Across Mood Symptoms, Suicidality, and Cognition Greater monoamine oxidase a binding in perimenopausal age as measured with carbon 11-labeled harmine positron emission tomography.Previous history of chronic stress changes the transcriptional response to glucocorticoid challenge in the dentate gyrus region of the male rat hippocampus.Effect of voluntary alcohol consumption on Maoa expression in the mesocorticolimbic brain of adult male rats previously exposed to prolonged maternal separation Elevated monoamine oxidase a binding during major depressive episodes is associated with greater severity and reversed neurovegetative symptoms.Modulation of monoamine oxidase (MAO) expression in neuropsychiatric disorders: genetic and environmental factors involved in type A MAO expression.Social defeat protocol and relevant biomarkers, implications for stress response physiology, drug abuse, mood disorders and individual stress vulnerability: a systematic review of the last decade.Binge ethanol exposure increases the Krüppel-like factor 11-monoamine oxidase (MAO) pathway in rats: Examining the use of MAO inhibitors to prevent ethanol-induced brain injury Systemic lipopolysaccharide-mediated alteration of cortical neuromodulation involves increases in monoamine oxidase-A and acetylcholinesterase activity Diabetes-causing gene, kruppel-like factor 11, modulates the antinociceptive response of chronic ethanol intake.Gene Expression Correlated with Severe Asthma Characteristics Reveals Heterogeneous Mechanisms of Severe Disease.Regulatory Pathways of Monoamine Oxidase A during Social Stress.Behavioral Activity and Some Markers of Posttraumatic Stress Disorder among Serotoninergic System Indicators and Glucocorticoid Metabolizing Enzymes in Rats with Different Duration of Hexenal Sleep.Type A and B monoamine oxidases distinctly modulate signal transduction pathway and gene expression to regulate brain function and survival of neurons.Repetitive transcranial magnetic stimulation inhibits Sirt1/MAO-A signaling in the prefrontal cortex in a rat model of depression and cortex-derived astrocytes.Potential Roles of microRNAs in the Regulation of Monoamine Oxidase A in the Brain

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Mechanistic role for a novel glucocorticoid-KLF11 (TIEG2) protein pathway in stress-induced monoamine oxidase A expression

http://www.wikidata.org/entity/Q36097919

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Journal of Biological Chemistry

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Mechanistic role for a novel g ...... monoamine oxidase A expression

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Craig A Stockmeier

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Deyin Lu

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Donald B Sittman

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Gwen Lomberk

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Ian A Paul

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Jeffrey H Meyer

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Junming Wang

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Klaus A Miczek

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Mark C Austin

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Matthew Grunewald

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P2860

Glucocorticoid and androgen activation of monoamine oxidase A is regulated differently by R1 and Sp1

Monoamine oxidase: from genes to behavior

Aggressive behavior and altered amounts of brain serotonin and norepinephrine in mice lacking MAOA

Functional interaction between coactivators CBP/p300, PCAF, and transcription factor FKLF2

Nitric oxide-induced nuclear GAPDH activates p300/CBP and mediates apoptosis

Monoamine oxidase A and repressor R1 are involved in apoptotic signaling pathway

High activity-related allele of MAO-A gene associated with depressed suicide in males

S-nitrosylated GAPDH initiates apoptotic cell death by nuclear translocation following Siah1 binding

Elevated monoamine oxidase a levels in the brain: an explanation for the monoamine imbalance of major depression

MAO-A gene polymorphisms are associated with major depression and sleep disturbance in males

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