Opposite Effects of M1 and M2 Macrophage Subtypes on Lung Cancer Progression. - Wikidata - wikimedia - TriplyDB

Opposite Effects of M1 and M2 Macrophage Subtypes on Lung Cancer Progression.

Opposite Effects of M1 and M2 Macrophage Subtypes on Lung Cancer Progression.

http://www.wikidata.org/entity/Q36099676

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Interplay between Cellular and Molecular Inflammatory Mediators in Lung Cancer Relationships between chronic obstructive pulmonary disease and lung cancer: biological insights Inferring condition-specific targets of human TF-TF complexes using ChIP-seq data.Inhibition of lung cancer growth by HangAmDan-B is mediated by macrophage activation to M1 subtype Diversity and Versatility of Phagocytosis: Roles in Innate Immunity, Tissue Remodeling, and Homeostasis.Targeting Tumor Microenvironment: Effects of Chinese Herbal Formulae on Macrophage-Mediated Lung Cancer in Mice.Negative Immune Regulator TIPE2 Promotes M2 Macrophage Differentiation through the Activation of PI3K-AKT Signaling Pathway Macrophages promote matrix protrusive and invasive function of breast cancer cells via MIP-1β dependent upregulation of MYO3A gene in breast cancer cells Breast cancers from black women exhibit higher numbers of immunosuppressive macrophages with proliferative activity and of crown-like structures associated with lower survival compared to non-black Latinas and Caucasians.IL-1α and IL-1β-producing macrophages populate lung tumor lesions in mice Cancer cells increase endothelial cell tube formation and survival by activating the PI3K/Akt signalling pathway.Hepatocellular carcinoma is accelerated by NASH involving M2 macrophage polarization mediated by hif-1αinduced IL-10 Macrophage Polarization Contributes to Glioblastoma Eradication by Combination Immunovirotherapy and Immune Checkpoint Blockade.Immune consequences of penfluridol treatment associated with inhibition of glioblastoma tumor growth.Melittin suppresses tumor progression by regulating tumor-associated macrophages in a Lewis lung carcinoma mouse model IL-33 blockade suppresses tumor growth of human lung cancer through direct and indirect pathways in a preclinical model.Macrophage Polarization Contributes to the Anti-Tumoral Efficacy of Mesoporous Nanovectors Loaded with Albumin-Bound Paclitaxel miR-708-5p: a microRNA with emerging roles in cancer.Generation and Functional In Vitro Analysis of Semliki Forest Virus Vectors Encoding TNF-α and IFN-γ.Danshen improves survival of patients with advanced lung cancer and targeting the relationship between macrophages and lung cancer cells.Mathematical modeling of tumor-associated macrophage interactions with the cancer microenvironment.NF-kappaB: Two Sides of the Same Coin.Clinical evaluation of macrophages in cancer: role in treatment, modulation and challenges.Targeting tumor microenvironment to curb chemoresistance via novel drug delivery strategies.Microscale Gene Expression Analysis of Tumor-Associated Macrophages.Characterization of cluster of differentiation 47 expression and its potential as a therapeutic target in esophageal squamous cell cancer.Macrophages: Key orchestrators of a tumor microenvironment defined by therapeutic resistance.Harnessing innate lung anti-cancer effector functions with a novel bacterial-derived immunotherapy.Matrix stiffness and tumor-associated macrophages modulate epithelial to mesenchymal transition of human adenocarcinoma cells.Regulation of Immune Cell Functions by Metabolic Reprogramming.Effect of colorectal cancer-derived extracellular vesicles on the immunophenotype and cytokine secretion profile of monocytes and macrophages.Epigenetic control of macrophage polarization: implications for targeting tumor-associated macrophages.Anti-PD-1 therapy redirects macrophages from an M2 to an M1 phenotype inducing regression of OS lung metastases.Engineered Tumor-Targeted T Cells Mediate Enhanced Anti-Tumor Efficacy Both Directly and through Activation of the Endogenous Immune System.Identification of an immune gene expression signature associated with favorable clinical features in Treg-enriched patient tumor samples.Role of Human Macrophage Polarization in Inflammation during Infectious Diseases.Endogenous retrovirus expression is associated with response to immune checkpoint blockade in clear cell renal cell carcinoma Identifying a Clinically Applicable Mutational Burden Threshold as a Potential Biomarker of Response to Immune Checkpoint Therapy in Solid Tumors Attenuation of CD47-SIRPα Signal in Cholangiocarcinoma Potentiates Tumor-Associated Macrophage-Mediated Phagocytosis and Suppresses Intrahepatic Metastasis Exploring immuno-regulatory mechanisms in the tumor microenvironment: Model and design of protocols for cancer remission

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Opposite Effects of M1 and M2 Macrophage Subtypes on Lung Cancer Progression.

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2015 nî lūn-bûn

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2015年論文

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2015年论文

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2015年论文

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Opposite Effects of M1 and M2 Macrophage Subtypes on Lung Cancer Progression.

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Opposite Effects of M1 and M2 Macrophage Subtypes on Lung Cancer Progression.

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Opposite Effects of M1 and M2 Macrophage Subtypes on Lung Cancer Progression.

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Opposite Effects of M1 and M2 Macrophage Subtypes on Lung Cancer Progression.

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Opposite Effects of M1 and M2 Macrophage Subtypes on Lung Cancer Progression.

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Scientific Reports

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Opposite Effects of M1 and M2 Macrophage Subtypes on Lung Cancer Progression

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Hsuan-Yu Chen

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Huei-Wen Chen

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Jeremy J W Chen

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Pan-Chyr Yang

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Tsai-Hsia Hong

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Yi-Chia Liu

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Yi-Jing Hsiao

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Yu-Yun Chen

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Hallmarks of Cancer: The Next Generation

Roles of CHOP/GADD153 in endoplasmic reticulum stress

Interaction of the p53-regulated protein Gadd45 with proliferating cell nuclear antigen

HRG inhibits tumor growth and metastasis by inducing macrophage polarization and vessel normalization through downregulation of PlGF

NF-kappa B-mediated repression of growth arrest- and DNA-damage-inducible proteins 45alpha and gamma is essential for cancer cell survival

Oncogene-induced senescence relayed by an interleukin-dependent inflammatory network.

Up-regulation of tumor interleukin-8 expression by infiltrating macrophages: its correlation with tumor angiogenesis and patient survival in non-small cell lung cancer

Cancer immunoediting: from immunosurveillance to tumor escape

Protein levels of p21, p27, cyclin E and Bax predict sensitivity to cisplatin and paclitaxel in head and neck squamous cell carcinomas

Cancer cells in transit: the vascular interactions of tumor cells

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10.1038/SREP14273

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26399191

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