IGF1R Derived PI3K/AKT Signaling Maintains Growth in a Subset of Human T-Cell Acute Lymphoblastic Leukemias.
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Combined targeting of SET and tyrosine kinases provides an effective therapeutic approach in human T-cell acute lymphoblastic leukemia.ALL-TRANS-RETINOIC ACID-MEDIATED CYTOPROTECTION IN RENAL EPITHELIAL CELLS IS COUPLED TO p-ERK ACTIVATION IN A ROS-INDEPENDENT MANNER.Advances in studies of tyrosine kinase inhibitors and their acquired resistance.Therapeutic Targeting of mTOR in T-Cell Acute Lymphoblastic Leukemia: An Update
P2860
IGF1R Derived PI3K/AKT Signaling Maintains Growth in a Subset of Human T-Cell Acute Lymphoblastic Leukemias.
description
2016 nî lūn-bûn
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2016年の論文
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2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
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2016年论文
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name
IGF1R Derived PI3K/AKT Signali ...... Acute Lymphoblastic Leukemias.
@ast
IGF1R Derived PI3K/AKT Signali ...... Acute Lymphoblastic Leukemias.
@en
type
label
IGF1R Derived PI3K/AKT Signali ...... Acute Lymphoblastic Leukemias.
@ast
IGF1R Derived PI3K/AKT Signali ...... Acute Lymphoblastic Leukemias.
@en
prefLabel
IGF1R Derived PI3K/AKT Signali ...... Acute Lymphoblastic Leukemias.
@ast
IGF1R Derived PI3K/AKT Signali ...... Acute Lymphoblastic Leukemias.
@en
P2093
P2860
P1433
P1476
IGF1R Derived PI3K/AKT Signali ...... Acute Lymphoblastic Leukemias.
@en
P2093
Andrew P Weng
Catherine E Jenkins
Michael Pollak
Samuel Gusscott
Sonya H Lam
Vincenzo Giambra
P2860
P304
P356
10.1371/JOURNAL.PONE.0161158
P407
P577
2016-08-17T00:00:00Z