"Autoimmune rejection" of neonatal heart transplants in experimental Chagas disease is a parasite-specific response to infected host tissue.
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VNI cures acute and chronic experimental Chagas diseaseCysteine protease inhibitors cure an experimental Trypanosoma cruzi infectionPathogenesis of chagas' disease: parasite persistence and autoimmunityGenes from Chagas susceptibility loci that are differentially expressed in T. cruzi-resistant mice are candidates accounting for impaired immunity.Chagas' disease and the autoimmunity hypothesis.DNA-Based immunization with Trypanosoma cruzi complement regulatory protein elicits complement lytic antibodies and confers protection against Trypanosoma cruzi infectionCoronary microvascular disease in chronic Chagas cardiomyopathy including an overview on history, pathology, and other proposed pathogenic mechanisms.Vaccination with trypomastigote surface antigen 1-encoding plasmid DNA confers protection against lethal Trypanosoma cruzi infection.Influence of acute-phase parasite load on pathology, parasitism, and activation of the immune system at the late chronic phase of Chagas' disease.Detection of live Trypanosoma cruzi in tissues of infected mice by using histochemical stain for beta-galactosidase.During Trypanosoma cruzi infection CD1d-restricted NK T cells limit parasitemia and augment the antibody response to a glycophosphoinositol-modified surface proteinCruzipain induces both mucosal and systemic protection against Trypanosoma cruzi in mice.Inducible nitric oxide synthase in heart tissue and nitric oxide in serum of Trypanosoma cruzi-infected rhesus monkeys: association with heart injury.Genetic dissection of drug resistance in trypanosomesCell-specific activation of nuclear factor-kappaB by the parasite Trypanosoma cruzi promotes resistance to intracellular infection.Inhibition of autoimmune Chagas-like heart disease by bone marrow transplantationA human type 5 adenovirus-based Trypanosoma cruzi therapeutic vaccine re-programs immune response and reverses chronic cardiomyopathy.Modulation of chagasic cardiomyopathy by interleukin-4: dissociation between inflammation and tissue parasitism.Innate and acquired immunity in the pathogenesis of Chagas disease.Oxidative stress and intracellular infections: more iron to the fire.The Trypanosoma cruzi-host-cell interplay: location, invasion, retention.Induction of cardiac autoimmunity in Chagas heart disease: a case for molecular mimicry.Oral vaccination with Salmonella enterica as a cruzipain-DNA delivery system confers protective immunity against Trypanosoma cruzi.Dominant T- and B-cell epitopes in an autoantigen linked to Chagas' diseaseChagas heart disease pathogenesis: one mechanism or many?Human infection with Trypanosoma cruzi induces parasite antigen-specific cytotoxic T lymphocyte responses.Design or screening of drugs for the treatment of Chagas disease: what shows the most promise?Chronic Trypanosoma cruzi-elicited cardiomyopathy: from the discovery to the proposal of rational therapeutic interventions targeting cell adhesion molecules and chemokine receptors--how to make a dream come true.Trypanocidal drugs: mechanisms, resistance and new targets.Host cell invasion by Trypanosoma cruzi: a unique strategy that promotes persistence.The emerging role of amiodarone and dronedarone in Chagas disease.Gene-deleted live-attenuated Trypanosoma cruzi parasites as vaccines to protect against Chagas disease.Nanomedicines against Chagas disease: an update on therapeutics, prophylaxis and diagnosis.Autoimmune pathogenesis of Chagas heart disease: looking back, looking ahead.Murine susceptibility to Chagas' disease maps to chromosomes 5 and 17.NK cells contribute to the control of Trypanosoma cruzi infection by killing free parasites by perforin-independent mechanisms.Infection of endothelial cells with Trypanosoma cruzi activates NF-kappaB and induces vascular adhesion molecule expression.Pathogenesis of Chagas disease: time to move on.Antitrypanosomal Activity of Sterol 14α-Demethylase (CYP51) Inhibitors VNI and VFV in the Swiss Mouse Models of Chagas Disease Induced by the Trypanosoma cruzi Y Strain.A heart-specific CD4+ T-cell line obtained from a chronic chagasic mouse induces carditis in heart-immunized mice and rejection of normal heart transplants in the absence of Trypanosoma cruzi.
P2860
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P2860
"Autoimmune rejection" of neonatal heart transplants in experimental Chagas disease is a parasite-specific response to infected host tissue.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
"Autoimmune rejection" of neon ...... ponse to infected host tissue.
@ast
"Autoimmune rejection" of neon ...... ponse to infected host tissue.
@en
type
label
"Autoimmune rejection" of neon ...... ponse to infected host tissue.
@ast
"Autoimmune rejection" of neon ...... ponse to infected host tissue.
@en
prefLabel
"Autoimmune rejection" of neon ...... ponse to infected host tissue.
@ast
"Autoimmune rejection" of neon ...... ponse to infected host tissue.
@en
P2093
P2860
P356
P1476
"Autoimmune rejection" of neon ...... ponse to infected host tissue.
@en
P2093
P2860
P304
P356
10.1073/PNAS.94.8.3932
P407
P577
1997-04-01T00:00:00Z