Gut mucosal injury in neonates is marked by macrophage infiltration in contrast to pleomorphic infiltrates in adult: evidence from an animal model - Wikidata - wikimedia - TriplyDB

Gut mucosal injury in neonates is marked by macrophage infiltration in contrast to pleomorphic infiltrates in adult: evidence from an animal model

Gut mucosal injury in neonates is marked by macrophage infiltration in contrast to pleomorphic infiltrates in adult: evidence from an animal model

http://www.wikidata.org/entity/Q36114925

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Haematological abnormalities in neonatal necrotizing enterocolitis Immunologic and Hematological Abnormalities in Necrotizing Enterocolitis.Neonatal mice with necrotizing enterocolitis-like injury develop thrombocytopenia despite increased megakaryopoiesis.Commensal and probiotic bacteria may prevent NEC by maturing intestinal host defenses.CXCL5 is required for angiogenesis, but not structural adaptation after small bowel resection.Cytokines associated with necrotizing enterocolitis in extremely-low-birth-weight infants Acute drop in blood monocyte count differentiates NEC from other causes of feeding intolerance.A clinical perspective of necrotizing enterocolitis: past, present, and future Smad7 inhibits autocrine expression of TGF-β2 in intestinal epithelial cells in baboon necrotizing enterocolitis The effect of impaired angiogenesis on intestinal function following massive small bowel resection.M1 to M2 macrophage polarization in heparin-binding epidermal growth factor-like growth factor therapy for necrotizing enterocolitis.CXCL5 signaling is a shared pathway of neuroinflammation and blood-brain barrier injury contributing to white matter injury in the immature brain.Preterm human milk contains a large pool of latent TGF-β, which can be activated by exogenous neuraminidase.The immunological landscape in necrotising enterocolitis Trinitrobenzene sulfonic acid-induced intestinal injury in neonatal mice activates transcriptional networks similar to those seen in human necrotizing enterocolitis.Amniotic fluid-borne hepatocyte growth factor protects rat pups against experimental necrotizing enterocolitis The Role of Mucosal Immunity in the Pathogenesis of Necrotizing Enterocolitis.The role of growth factors in intestinal regeneration and repair in necrotizing enterocolitis.Pathogenesis of necrotizing enterocolitis: modeling the innate immune response.Cytokines and growth factors in the developing intestine and during necrotizing enterocolitis.A co-culture model of the developing small intestine offers new insight in the early immunomodulation of enterocytes and macrophages by Lactobacillus spp. through STAT1 and NF-kB p65 translocation.Pathogenesis of NEC: Role of the innate and adaptive immune response.The viral dsRNA analogue poly (I:C) induces necrotizing enterocolitis in neonatal mice.Dextran sodium sulfate (DSS) induces necrotizing enterocolitis-like lesions in neonatal mice Smad7 interrupts TGF-β signaling in intestinal macrophages and promotes inflammatory activation of these cells during necrotizing enterocolitis.The science and necessity of using animal models in the study of necrotizing enterocolitis.Increased monocytes and bands following a red blood cell transfusion.

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Gut mucosal injury in neonates is marked by macrophage infiltration in contrast to pleomorphic infiltrates in adult: evidence from an animal model

http://www.wikidata.org/entity/Q36114925

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Gut mucosal injury in neonates ...... evidence from an animal model

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Gut mucosal injury in neonates ...... evidence from an animal model

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Gut mucosal injury in neonates ...... evidence from an animal model

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American Journal of Physiology - Gastrointestinal and Liver Physiology

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Gut mucosal injury in neonates ...... evidence from an animal model

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Akhil Maheshwari

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Anchal Bansal

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Ashish R Kurundkar

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C Rekha Bandepalli

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Joern-Hendrik Weitkamp

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Juan I Remon

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Kopperuncholan Namachivayam

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Krishnan MohanKumar

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Niroop Kaza

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Ramasamy Jagadeeswaran

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P2860

TGF-β2 suppresses macrophage cytokine production and mucosal inflammatory responses in the developing intestine

FGF-10 is decreased in bronchopulmonary dysplasia and suppressed by Toll-like receptor activation

Epithelial cells in fetal intestine produce chemerin to recruit macrophages

Inflammation in the developing human intestine: A possible pathophysiologic contribution to necrotizing enterocolitis

Enterocytes are the primary source of the chemokine ENA-78 in normal colon and ulcerative colitis

Regulation and function of the CXC chemokine ENA-78 in monocytes and its role in disease

Role of T lymphocytes in rat 2,4,6-trinitrobenzene sulphonic acid (TNBS) induced colitis: increased mortality after gammadelta T cell depletion and no effect of alphabeta T cell depletion

CXCL5 regulates chemokine scavenging and pulmonary host defense to bacterial infection

Age-dependent maturation of Toll-like receptor-mediated cytokine responses in Gambian infants

Identification of protein coding regions by database similarity search

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