SkM1 and Cx32 improve conduction in canine myocardial infarcts yet only SkM1 is antiarrhythmic
about
Deranged sodium to sudden death.Gene therapy to restore electrophysiological function in heart failure.Can heart function lost to disease be regenerated by therapeutic targeting of cardiac scar tissue?Gene Therapy for Cardiac Arrhythmias.Emerging molecular therapies targeting myocardial infarction-related arrhythmias.Ventricular repolarization time, location of pacing stimulus and current pulse amplitude conspire to determine arrhythmogenicity in mice.Restoring heart function and electrical integrity: closing the circuit.
P2860
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P2860
SkM1 and Cx32 improve conduction in canine myocardial infarcts yet only SkM1 is antiarrhythmic
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
SkM1 and Cx32 improve conducti ...... et only SkM1 is antiarrhythmic
@ast
SkM1 and Cx32 improve conducti ...... et only SkM1 is antiarrhythmic
@en
type
label
SkM1 and Cx32 improve conducti ...... et only SkM1 is antiarrhythmic
@ast
SkM1 and Cx32 improve conducti ...... et only SkM1 is antiarrhythmic
@en
prefLabel
SkM1 and Cx32 improve conducti ...... et only SkM1 is antiarrhythmic
@ast
SkM1 and Cx32 improve conducti ...... et only SkM1 is antiarrhythmic
@en
P2093
P2860
P356
P1476
SkM1 and Cx32 improve conducti ...... et only SkM1 is antiarrhythmic
@en
P2093
David H Lau
Eugene A Sosunov
Evgeny P Anyukhovsky
Gerard J J Boink
Heather S Duffy
Helen E Driessen
Ira S Cohen
Iryna N Shlapakova
Michael R Rosen
P2860
P304
P356
10.1093/CVR/CVS107
P577
2012-02-27T00:00:00Z