Paradoxical effect of rapamycin on inflammatory stress-induced insulin resistance in vitro and in vivo.

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Airborne PM2.5-Induced Hepatic Insulin Resistance by Nrf2/JNK-Mediated Signaling Pathway.Bidirectional modulation of insulin action by reactive oxygen species in 3T3‑L1 adipocytes.mTOR complexes differentially orchestrates eosinophil development in allergy.

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Paradoxical effect of rapamycin on inflammatory stress-induced insulin resistance in vitro and in vivo.

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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Paradoxical effect of rapamyci ...... sistance in vitro and in vivo.

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Paradoxical effect of rapamyci ...... sistance in vitro and in vivo.

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Paradoxical effect of rapamyci ...... sistance in vitro and in vivo.

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Paradoxical effect of rapamyci ...... sistance in vitro and in vivo.

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Paradoxical effect of rapamyci ...... sistance in vitro and in vivo.

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Paradoxical effect of rapamyci ...... sistance in vitro and in vivo.

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P1476

Paradoxical effect of rapamyci ...... sistance in vitro and in vivo.

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P2093

John F Moorhead

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Jun Yuan

http://www.w3.org/2001/XMLSchema#string

Lei Zhao

http://www.w3.org/2001/XMLSchema#string

Ping Yang

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Xiong Z Ruan

http://www.w3.org/2001/XMLSchema#string

Yao Chen

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Yaxi Chen

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Yunfei Zhao

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Zac Varghese

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Inflammation and insulin resistance

S6K directly phosphorylates IRS-1 on Ser-270 to promote insulin resistance in response to TNF-(alpha) signaling through IKK2

Nutrients suppress phosphatidylinositol 3-kinase/Akt signaling via raptor-dependent mTOR-mediated insulin receptor substrate 1 phosphorylation

Rapamycin-induced insulin resistance is mediated by mTORC2 loss and uncoupled from longevity

Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance

The c-Jun NH(2)-terminal kinase promotes insulin resistance during association with insulin receptor substrate-1 and phosphorylation of Ser(307)

Specificity and mechanism of action of some commonly used protein kinase inhibitors

Suppressor of cytokine signaling 1 (SOCS-1) and SOCS-3 cause insulin resistance through inhibition of tyrosine phosphorylation of insulin receptor substrate proteins by discrete mechanisms

MCP-1 contributes to macrophage infiltration into adipose tissue, insulin resistance, and hepatic steatosis in obesity

Local and systemic insulin resistance resulting from hepatic activation of IKK-beta and NF-kappaB

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scholarly article

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10.1038/SREP14959

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English

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2015-10-09T00:00:00Z

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Paradoxical effect of rapamycin on inflammatory stress-induced insulin resistance in vitro and in vivo.

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P2860

P2860

Paradoxical effect of rapamycin on inflammatory stress-induced insulin resistance in vitro and in vivo.

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P1476

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P2860

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