Reduced knee extensor function in heart failure is not explained by inactivity
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Discerning primary and secondary factors responsible for clinical fatigue in multisystem diseasesSkeletal muscle myofilament adaptations to aging, disease, and disuse and their effects on whole muscle performance in older adult humansRandom myosin loss along thick-filaments increases myosin attachment time and the proportion of bound myosin heads to mitigate force decline in skeletal muscle.Age-related structural alterations in human skeletal muscle fibers and mitochondria are sex specific: relationship to single-fiber functionMyofilament protein alterations promote physical disability in aging and disease.Chronic heart failure reduces Akt phosphorylation in human skeletal muscle: relationship to muscle size and functionEffect of acute dietary nitrate intake on maximal knee extensor speed and power in healthy men and women.Chronic disuse and skeletal muscle structure in older adults: sex-specific differences and relationships to contractile function.Skeletal muscle mitochondrial density, gene expression, and enzyme activities in human heart failure: minimal effects of the disease and resistance training.Acute Dietary Nitrate Intake Improves Muscle Contractile Function in Patients With Heart Failure: A Double-Blind, Placebo-Controlled, Randomized Trial.Effect of resistance training on physical disability in chronic heart failure.Reduced rate of knee extensor torque development in older adults with knee osteoarthritis is associated with intrinsic muscle contractile deficits.Skeletal muscle ultrastructure and function in statin-tolerant individualsResearch on cachexia, sarcopenia and skeletal muscle in cardiology.Molecular mechanisms underlying skeletal muscle weakness in human cancer: reduced myosin-actin cross-bridge formation and kinetics.Analysis of Skeletal Muscle Torque Capacity and Circulating Ceramides in Patients with Advanced Heart Failure.Age-related slowing of myosin actin cross-bridge kinetics is sex specific and predicts decrements in whole skeletal muscle performance in humans.Muscle size explains low passive skeletal muscle force in heart failure patients.Mechanisms underlying skeletal muscle weakness in human heart failure: alterations in single fiber myosin protein content and function.Intrinsic skeletal muscle alterations in chronic heart failure patients: a disease-specific myopathy or a result of deconditioning?Skeletal muscle protein metabolism in human heart failure.Dietary Nitrate and Skeletal Muscle Contractile Function in Heart Failure.Moderate-intensity resistance exercise alters skeletal muscle molecular and cellular structure and function in inactive older adults with knee osteoarthritis.Resistance training alters skeletal muscle structure and function in human heart failure: effects at the tissue, cellular and molecular levels.Therapeutic Targets for the Multi-system Pathophysiology of Heart Failure: Exercise Training.High- versus moderate-intensity aerobic exercise training effects on skeletal muscle of infarcted rats.
P2860
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P2860
Reduced knee extensor function in heart failure is not explained by inactivity
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2009 nî lūn-bûn
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2009年の論文
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2009年学术文章
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2009年学术文章
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Reduced knee extensor function in heart failure is not explained by inactivity
@ast
Reduced knee extensor function in heart failure is not explained by inactivity
@en
type
label
Reduced knee extensor function in heart failure is not explained by inactivity
@ast
Reduced knee extensor function in heart failure is not explained by inactivity
@en
prefLabel
Reduced knee extensor function in heart failure is not explained by inactivity
@ast
Reduced knee extensor function in heart failure is not explained by inactivity
@en
P2093
P2860
P1476
Reduced knee extensor function in heart failure is not explained by inactivity
@en
P2093
Anthony O Shaw
David W Maughan
Mark S Miller
Martin M LeWinter
Michael J Toth
Peter VanBuren
Philip A Ades
P2860
P304
P356
10.1016/J.IJCARD.2009.02.040
P577
2009-03-27T00:00:00Z