Receptor-independent actions of PPAR thiazolidinedione agonists: is mitochondrial function the key?
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Effect of pioglitazone treatment on behavioral symptoms in autistic childrenPioglitazone acutely reduces insulin secretion and causes metabolic deceleration of the pancreatic beta-cell at submaximal glucose concentrationsRosiglitazone increases dendritic spine density and rescues spine loss caused by apolipoprotein E4 in primary cortical neuronsMitoNEET is a uniquely folded 2Fe 2S outer mitochondrial membrane protein stabilized by pioglitazoneBreaking the mold: transcription factors in the anucleate platelet and platelet-derived microparticlesCrystal structure of human mitoNEET reveals distinct groups of iron sulfur proteinsCrystallographic studies of human MitoNEETPPARγ in Bacterial Infections: A Friend or Foe?Omega-3 Fatty Acids and PPARgamma in CancerComparative gene expression profiles induced by PPARγ and PPARα/γ agonists in human hepatocytesIdentification of a mitochondrial target of thiazolidinedione insulin sensitizers (mTOT)--relationship to newly identified mitochondrial pyruvate carrier proteinsMitoNEET is an iron-containing outer mitochondrial membrane protein that regulates oxidative capacityNutrient-deprivation autophagy factor-1 (NAF-1): biochemical properties of a novel cellular target for anti-diabetic drugsEffect of chronic treatment with Rosiglitazone on Leydig cell steroidogenesis in rats: in vivo and ex vivo studiesPPARγ Agonists Attenuate Trigeminal Neuropathic Pain.Pioglitazone enhances mitochondrial biogenesis and ribosomal protein biosynthesis in skeletal muscle in polycystic ovary syndrome.Mood, memory and movement: an age-related neurodegenerative complex?The PPARgamma agonist pioglitazone ameliorates aging-related progressive renal injury.Thiazolidinedione-independent activation of peroxisome proliferator-activated receptor γ is a potential target for diabetic macrovascular complications.Effects of PPAR gamma ligands on TGF-beta1-induced epithelial-mesenchymal transition in alveolar epithelial cells.Insulin resistance is not conserved in myotubes established from women with PCOS.Alzheimer's disease: relevant molecular and physiopathological events affecting amyloid-β brain balance and the putative role of PPARs.Chemical proteomics-based analysis of off-target binding profiles for rosiglitazone and pioglitazone: clues for assessing potential for cardiotoxicity.Thiazolidinediones and the promise of insulin sensitization in type 2 diabetes.Development of Therapeutics That Induce Mitochondrial Biogenesis for the Treatment of Acute and Chronic Degenerative Diseases.PPAR-γ activation increases insulin secretion through the up-regulation of the free fatty acid receptor GPR40 in pancreatic β-cellsPTB-associated splicing factor (PSF) is a PPARγ-binding protein and growth regulator of colon cancer cells.Targeting PPARγ Signaling Cascade for the Prevention and Treatment of Prostate Cancer.The complexities of obesity and diabetes with the development and progression of pancreatic cancer.Novel insulin sensitizer modulates nutrient sensing pathways and maintains β-cell phenotype in human islets.Hyper-inflammation and skin destruction mediated by rosiglitazone activation of macrophages in IL-6 deficiency.Pioglitazone restores phagocyte mitochondrial oxidants and bactericidal capacity in chronic granulomatous disease.The PPAR-γ agonist pioglitazone modulates inflammation and induces neuroprotection in parkinsonian monkeys.Adipocyte mitochondrial genes and the forkhead factor FOXC2 are decreased in type 2 diabetes patients and normalized in response to rosiglitazoneThymoquinone and curcumin attenuate gentamicin-induced renal oxidative stress, inflammation and apoptosis in rats.An insulin-sensitizing thiazolidinedione, which minimally activates PPARγ, does not cause bone loss.Rosiglitazone affects nitric oxide synthases and improves renal outcome in a rat model of severe ischemia/reperfusion injury.Troglitazone induced apoptosis of human pterygium fibroblasts through a mitochondrial-dependent pathway.Insulin resistance and metabolic derangements in obese mice are ameliorated by a novel peroxisome proliferator-activated receptor γ-sparing thiazolidinedione.Role of transcription factors in mediating post-ischemic cerebral inflammation and brain damage.
P2860
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P2860
Receptor-independent actions of PPAR thiazolidinedione agonists: is mitochondrial function the key?
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Receptor-independent actions o ...... itochondrial function the key?
@ast
Receptor-independent actions o ...... itochondrial function the key?
@en
type
label
Receptor-independent actions o ...... itochondrial function the key?
@ast
Receptor-independent actions o ...... itochondrial function the key?
@en
prefLabel
Receptor-independent actions o ...... itochondrial function the key?
@ast
Receptor-independent actions o ...... itochondrial function the key?
@en
P2093
P1476
Receptor-independent actions o ...... itochondrial function the key?
@en
P2093
P304
P356
10.1016/J.BCP.2005.03.033
P407
P577
2005-07-01T00:00:00Z