Sensitivity Analysis of the NPM-ALK Signalling Network Reveals Important Pathways for Anaplastic Large Cell Lymphoma Combination Therapy.

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Computer simulations of the signalling network in FLT3 +-acute myeloid leukaemia - indications for an optimal dosage of inhibitors against FLT3 and CDK6.A computational study of hedgehog signalling involved in basal cell carcinoma reveals the potential and limitation of combination therapy.

P2860

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Sensitivity Analysis of the NPM-ALK Signalling Network Reveals Important Pathways for Anaplastic Large Cell Lymphoma Combination Therapy.

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http://www.wikidata.org/entity/Q36145016

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

@zh-hant

2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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name

Sensitivity Analysis of the NP ...... Lymphoma Combination Therapy.

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Sensitivity Analysis of the NP ...... Lymphoma Combination Therapy.

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type

Item

label

Sensitivity Analysis of the NP ...... Lymphoma Combination Therapy.

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Sensitivity Analysis of the NP ...... Lymphoma Combination Therapy.

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prefLabel

Sensitivity Analysis of the NP ...... Lymphoma Combination Therapy.

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Sensitivity Analysis of the NP ...... Lymphoma Combination Therapy.

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P1476

Sensitivity Analysis of the NP ...... l Lymphoma Combination Therapy

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P2093

Thomas O'Hare

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P2860

The SH2/SH3 domain-containing protein GRB2 interacts with tyrosine-phosphorylated IRS1 and Shc: implications for insulin control of ras signalling

Vav3 mediates receptor protein tyrosine kinase signaling, regulates GTPase activity, modulates cell morphology, and induces cell transformation.

Cytoscape: a software environment for integrated models of biomolecular interaction networks

Nucleophosmin-anaplastic lymphoma kinase of large-cell anaplastic lymphoma is a constitutively active tyrosine kinase that utilizes phospholipase C-gamma to mediate its mitogenicity

Characterization of the transforming activity of p80, a hyperphosphorylated protein in a Ki-1 lymphoma cell line with chromosomal translocation t(2;5)

ALK and ROS1 as a joint target for the treatment of lung cancer: a review

Activating mutations in ALK provide a therapeutic target in neuroblastoma.

Multiplex reverse transcription-PCR screening for EML4-ALK fusion transcripts.

EML4-ALK mutations in lung cancer that confer resistance to ALK inhibitors.

A novel ALK secondary mutation and EGFR signaling cause resistance to ALK kinase inhibitors.

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e0163011

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scholarly article

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10.1371/JOURNAL.PONE.0163011

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English

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Antoine Buetti-Dinh

Ran Friedman

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2016-09-26T00:00:00Z

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27669408

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5036789

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Sensitivity Analysis of the NPM-ALK Signalling Network Reveals Important Pathways for Anaplastic Large Cell Lymphoma Combination Therapy.

about

P2860

P2860

Sensitivity Analysis of the NPM-ALK Signalling Network Reveals Important Pathways for Anaplastic Large Cell Lymphoma Combination Therapy.

description

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type

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P1433

P1476

P2093

P2860

P304

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P356

P407

P433

P478

P50

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P932