Transforming growth factor-beta1 suppresses airway hyperresponsiveness in allergic airway disease.
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Chronic OVA allergen challenged Siglec-F deficient mice have increased mucus, remodeling, and epithelial Siglec-F ligands which are up-regulated by IL-4 and IL-13Genetic variants in TNFa, TGFB1, PTGS1 and PTGS2 genes are associated with diisocyanate-induced asthmaTreatment of allergic asthma: modulation of Th2 cells and their responsesStrain-dependent activation of NF-kappaB in the airway epithelium and its role in allergic airway inflammationTgf-Beta isoform specific regulation of airway inflammation and remodelling in a murine model of asthma.The presence of CD14 overcomes evasion of innate immune responses by virulent Francisella tularensis in human dendritic cells in vitro and pulmonary cells in vivo.Absence of c-Jun NH2-terminal kinase 1 protects against house dust mite-induced pulmonary remodeling but not airway hyperresponsiveness and inflammation.Airway hyperresponsiveness is associated with airway remodeling but not inflammation in aging Cav1-/- mice.CTGF upregulation correlates with MMP-9 level in airway remodeling in a murine model of asthma.Functional effects of TGF-β1 on mesenchymal stem cell mobilization in cockroach allergen-induced asthmaInhaled salmeterol and/or fluticasone alters structure/function in a murine model of allergic airways disease.Toll-like receptor-9 agonist inhibits airway inflammation, remodeling and hyperreactivity in mice exposed to chronic environmental tobacco smoke and allergenSystems biology approaches to understanding Epithelial Mesenchymal Transition (EMT) in mucosal remodeling and signaling in asthmaDeficiency of endothelial heparan sulfates attenuates allergic airway inflammation.Bone morphogenetic protein (BMP)-4 and BMP-7 regulate differentially transforming growth factor (TGF)-beta1 in normal human lung fibroblasts (NHLF).Bronchial epithelial cells produce IL-5: implications for local immune responses in the airways.The complex relationship between inflammation and lung function in severe asthmaBone marrow-derived mesenchymal stromal cells inhibit Th2-mediated allergic airways inflammation in miceAllergen-induced coexpression of bFGF and TGF-β1 by macrophages in a mouse model of airway remodeling: bFGF induces macrophage TGF-β1 expression in vitro.Aldose reductase inhibition prevents allergic airway remodeling through PI3K/AKT/GSK3β pathway in mice.Surfactant protein D attenuates sub-epithelial fibrosis in allergic airways disease through TGF-βA protective role for periostin and TGF-β in IgE-mediated allergy and airway hyperresponsivenessPulmonary receptor for advanced glycation end-products promotes asthma pathogenesis through IL-33 and accumulation of group 2 innate lymphoid cellsPathophysiological features of asthma develop in parallel in house dust mite-exposed neonatal mice.CD4+CD25+ regulatory T cells reverse established allergic airway inflammation and prevent airway remodeling.Pharyngeal microflora disruption by antibiotics promotes airway hyperresponsiveness after respiratory syncytial virus infection.The immunoregulatory and fibrotic roles of activin A in allergic asthmaEpistatic interactions between Tgfb1 and genetic loci, Tgfbm2 and Tgfbm3, determine susceptibility to an asthmatic stimulus.Molecular Mechanisms of Airway Hyperresponsiveness in a Murine Model of Steroid-Resistant Airway Inflammation.Resolvin D1 and aspirin-triggered resolvin D1 promote resolution of allergic airways responses.Airway Hyperresponsiveness and Inflammation: Causation, Correlation, or No Relation?Acute exercise decreases airway inflammation, but not responsiveness, in an allergic asthma model.Primary murine airway smooth muscle cells exposed to poly(I,C) or tunicamycin synthesize a leukocyte-adhesive hyaluronan matrixPI3K gamma-deficient mice have reduced levels of allergen-induced eosinophilic inflammation and airway remodeling.Variants in TGFB1, dust mite exposure, and disease severity in children with asthma.Healthy but not RSV-infected lung epithelial cells profoundly inhibit T cell activationImmunization against TGF-β1 reduces collagen deposition but increases sustained inflammation in a murine asthma modelUpdate in asthma 2007.Chronic alcohol induces M2 polarization enhancing pulmonary disease caused by exposure to particulate air pollutionTGF-beta can leave you breathless
P2860
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P2860
Transforming growth factor-beta1 suppresses airway hyperresponsiveness in allergic airway disease.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Transforming growth factor-bet ...... ss in allergic airway disease.
@ast
Transforming growth factor-bet ...... ss in allergic airway disease.
@en
type
label
Transforming growth factor-bet ...... ss in allergic airway disease.
@ast
Transforming growth factor-bet ...... ss in allergic airway disease.
@en
prefLabel
Transforming growth factor-bet ...... ss in allergic airway disease.
@ast
Transforming growth factor-bet ...... ss in allergic airway disease.
@en
P2093
P2860
P1476
Transforming growth factor-bet ...... ss in allergic airway disease.
@en
P2093
Charles G Irvin
Elizabeth F Jaffe
Jason H T Bates
John F Alcorn
Lisa M Rinaldi
Mirjam van Loon
P2860
P304
P356
10.1164/RCCM.200702-334OC
P407
P577
2007-08-29T00:00:00Z