Immunodominant fragments of myelin basic protein initiate T cell-dependent pain. - Wikidata - wikimedia - TriplyDB

Immunodominant fragments of myelin basic protein initiate T cell-dependent pain.

Immunodominant fragments of myelin basic protein initiate T cell-dependent pain.

http://www.wikidata.org/entity/Q36156744

about

In vivo nanoneurotoxicity screening using oxidative stress and neuroinflammation paradigms Matrix metalloproteinase-14 both sheds cell surface neuronal glial antigen 2 (NG2) proteoglycan on macrophages and governs the response to peripheral nerve injury Active immunization with myelin-derived altered peptide ligand reduces mechanical pain hypersensitivity following peripheral nerve injury The alternatively spliced fibronectin CS1 isoform regulates IL-17A levels and mechanical allodynia after peripheral nerve injury.Matrix Metalloproteinase (MMP) Proteolysis of the Extracellular Loop of Voltage-gated Sodium Channels and Potential Alterations in Pain Signaling.Matrix metalloproteinases - From the cleavage data to the prediction tools and beyond.Pain in experimental autoimmune encephalitis: a comparative study between different mouse models.Role of myelin auto-antigens in pain: a female connection.Safety of human neural stem cell transplantation in chronic spinal cord injury.Molecules in pain and sex: a developing story.Molecules involved in the crosstalk between immune- and peripheral nerve Schwann cells.Myelin Basic Protein Citrullination in Multiple Sclerosis: A Potential Therapeutic Target for the Pathology.Reciprocal relationship between membrane type 1 matrix metalloproteinase and the algesic peptides of myelin basic protein contributes to chronic neuropathic pain.Spinal activity of interleukin 6 mediates myelin basic protein-induced allodynia.Schwann cells promote post-traumatic nerve inflammation and neuropathic pain through MHC class II.A sensitive and selective ELISA methodology quantifies a demyelination marker in experimental and clinical samples.Perineural expression of high-mobility group box-1 contributes to long-lasting mechanical hypersensitivity via matrix metalloproteinase-9 upregulation in mice with painful peripheral neuropathy.Acute- and late-phase matrix metalloproteinase (MMP)-9 activity is comparable in female and male rats after peripheral nerve injury.

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P2860

Immunodominant fragments of myelin basic protein initiate T cell-dependent pain.

http://www.wikidata.org/entity/Q36156744

description

2012 nî lūn-bûn

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2012年论文

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name

Immunodominant fragments of myelin basic protein initiate T cell-dependent pain.

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Immunodominant fragments of myelin basic protein initiate T cell-dependent pain.

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type

label

Immunodominant fragments of myelin basic protein initiate T cell-dependent pain.

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Immunodominant fragments of myelin basic protein initiate T cell-dependent pain.

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prefLabel

Immunodominant fragments of myelin basic protein initiate T cell-dependent pain.

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Immunodominant fragments of myelin basic protein initiate T cell-dependent pain.

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1742-2094-9-119

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Journal of Neuroinflammation

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Immunodominant fragments of myelin basic protein initiate T cell-dependent pain

@en

P2093

Albert G Remacle

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Andrei V Chernov

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Huaqing Liu

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Igor Shubayev

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Sergey A Shiryaev

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Svetlana Baranovskaya

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Veronica I Shubayev

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Vladislav S Golubkov

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Youngsoon Kim

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Ectopic discharge in Abeta afferents as a source of neuropathic pain

The role of neuroinflammation in neuropathic pain: mechanisms and therapeutic targets

Matrix metalloproteinase proteolysis of the myelin basic protein isoforms is a source of immunogenic peptides in autoimmune multiple sclerosis

Quantitative assessment of tactile allodynia in the rat paw

A new and sensitive method for measuring thermal nociception in cutaneous hyperalgesia

Structure and function of matrix metalloproteinases and TIMPs

Endoneurial remodeling by TNFalph- and TNFalpha-releasing proteases. A spatial and temporal co-localization study in painful neuropathy.

Proteomic identification of brainstem cytosolic proteins in a neuropathic pain model.

Sodium channel expression and localization at demyelinated sites in painful human dental pulp

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1742-2094-9-119

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9

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