Rapid, estrogen receptor-mediated signaling: why is the endothelium so special? - Wikidata - wikimedia - TriplyDB

Rapid, estrogen receptor-mediated signaling: why is the endothelium so special?

Rapid, estrogen receptor-mediated signaling: why is the endothelium so special?

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Splice isoform estrogen receptors as integral transmembrane proteins Estrogen receptors in gastric cancer: Advances and perspectives Endothelial estrogen receptor isoforms and cardiovascular disease Estrogen receptor alpha 46 is reduced in tamoxifen resistant breast cancer cells and re-expression inhibits cell proliferation and estrogen receptor alpha 66-regulated target gene transcription.Vascular endothelial growth factor receptor-2 in breast cancer.Na+K+-ATPase activity and K+ channels differently contribute to vascular relaxation in male and female rats Sex steroid signaling: implications for lung diseases.Estrogen signaling multiple pathways to impact gene transcription.S-nitrosylation: NO-related redox signaling to protect against oxidative stress.Transit of hormonal and EGF receptor-dependent signals through cholesterol-rich membranes Estrogen-dependent enhancement of NO production in the nucleus tractus solitarius contributes to ethanol-induced hypotension in conscious female rats.ER-α36-mediated gastric cancer cell proliferation via the c-Src pathway Membrane-initiated actions of estrogen on the endothelium.Estrogen Regulation of MicroRNA Expression Maternal uterine vascular remodeling during pregnancy Estrogen and the Ca2+-mobilizing agonist ATP evoke acute NO synthesis via distinct pathways in an individual human vascular endothelium-derived cell.Role of GPER in estrogen-dependent nitric oxide formation and vasodilation.

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Rapid, estrogen receptor-mediated signaling: why is the endothelium so special?

http://www.wikidata.org/entity/Q36163230

description

2005 nî lūn-bûn

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Rapid, estrogen receptor-mediated signaling: why is the endothelium so special?

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Rapid, estrogen receptor-mediated signaling: why is the endothelium so special?

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Rapid, estrogen receptor-mediated signaling: why is the endothelium so special?

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Rapid, estrogen receptor-mediated signaling: why is the endothelium so special?

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Rapid, estrogen receptor-mediated signaling: why is the endothelium so special?

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Rapid, estrogen receptor-mediated signaling: why is the endothelium so special?

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Science's STKE : signal transduction knowledge environment

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Rapid, estrogen receptor-mediated signaling: why is the endothelium so special?

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Jeffrey R Bender

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Kyung Hee Kim

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P2860

Characterization of the interactions of estrogen receptor and MNAR in the activation of cSrc

Striatin assembles a membrane signaling complex necessary for rapid, nongenomic activation of endothelial NO synthase by estrogen receptor alpha

Risks and benefits of estrogen plus progestin in healthy postmenopausal women: principal results From the Women's Health Initiative randomized controlled trial

Cardiovascular disease outcomes during 6.8 years of hormone therapy: Heart and Estrogen/progestin Replacement Study follow-up (HERS II)

Randomized trial of estrogen plus progestin for secondary prevention of coronary heart disease in postmenopausal women. Heart and Estrogen/progestin Replacement Study (HERS) Research Group

Estrogen induces the Akt-dependent activation of endothelial nitric-oxide synthase in vascular endothelial cells.

Estradiol accelerates reendothelialization in mouse carotid artery through estrogen receptor-alpha but not estrogen receptor-beta.

Rapid nitric oxide-mediated S-nitrosylation of estrogen receptor: regulation of estrogen-dependent gene transcription

The AF-1 activation-function of ERalpha may be dispensable to mediate the effect of estradiol on endothelial NO production in mice

Hormone therapy to prevent disease and prolong life in postmenopausal women.

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