Expression of human endogenous retrovirus type K (HML-2) is activated by the Tat protein of HIV-1.
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Human endogenous retrovirus K and cancer: Innocent bystander or tumorigenic accomplice?Vaccination against endogenous retrotransposable element consensus sequences does not protect rhesus macaques from SIVsmE660 infection and replicationHIV infection reveals widespread expansion of novel centromeric human endogenous retrovirusesEpigenetic Control of Human Endogenous Retrovirus Expression: Focus on Regulation of Long-Terminal Repeats (LTRs)A survey of endogenous retrovirus (ERV) sequences in the vicinity of multiple sclerosis (MS)-associated single nucleotide polymorphisms (SNPs).The retrovirus/superantigen hypothesis of multiple sclerosis."Reverse genomics" and human endogenous retroviruses.HIV-1 interacts with human endogenous retrovirus K (HML-2) envelopes derived from human primary lymphocytes.Cutting edge: An antibody recognizing ancestral endogenous virus glycoproteins mediates antibody-dependent cellular cytotoxicity on HIV-1-infected cells.Regulation of the human endogenous retrovirus K (HML-2) transcriptome by the HIV-1 Tat protein.Human endogenous retrovirus np9 gene is over expressed in chronic lymphocytic leukemia patients.HIV-1 infection leads to increased transcription of human endogenous retrovirus HERV-K (HML-2) proviruses in vivo but not to increased virion production.HERV-K(HML-2), a seemingly silent subtenant - but still waters run deep.Frequency of human endogenous retroviral sequences (HERV) K113 and K115 in the Polish population, and their effect on HIV infectionHuman Endogenous Retrovirus Type K (HERV-K) Particles Package and Transmit HERV-K-Related SequencesHuman endogenous retrovirus K Gag coassembles with HIV-1 Gag and reduces the release efficiency and infectivity of HIV-1.Further Evidence that Human Endogenous Retrovirus K102 is a Replication Competent Foamy Virus that may Antagonize HIV-1 ReplicationHuman endogenous retrovirus (HERV) expression is not induced by treatment with the histone deacetylase (HDAC) inhibitors in cellular models of HIV-1 latencyEndogenous retrovirus-K promoter: a landing strip for inflammatory transcription factors?HERV-K(HML-2), the Best Preserved Family of HERVs: Endogenization, Expression, and Implications in Health and Disease.HERV-K and LINE-1 DNA Methylation and Reexpression in Urothelial Carcinoma.Vaccination directed against the human endogenous retrovirus-K (HERV-K) gag protein slows HERV-K gag expressing cell growth in a murine model system.HIV-1 latency: an update of molecular mechanisms and therapeutic strategies.Anti-HERV-K (HML-2) capsid antibody responses in HIV elite controllers.The role of molecular mimicry and other factors in the association of Human Endogenous Retroviruses and autoimmunity.Modulation of human endogenous retrovirus (HERV) transcription during persistent and de novo HIV-1 infection.HBV X Protein induces overexpression of HERV-W env through NF-κB in HepG2 cells.Molecular mechanisms by which HERV-K Gag interferes with HIV-1 Gag assembly and particle infectivity.Expression patterns of endogenous avian retrovirus ALVE1 and its response to infection with exogenous avian tumour viruses.Expression of the env gene from the avian endogenous retrovirus ALVE and regulation by miR-155.Human endogenous retrovirus-K contributes to motor neuron disease.Natural and cross-inducible anti-SIV antibodies in Mauritian cynomolgus macaques.Cooccurrences of Putative Endogenous Retrovirus-Associated Diseases.Fluctuations in Tat copy number when it counts the most: a possible mechanism to battle the HIV latency.HIV-1 Infection of Primary CD4+ T Cells Regulates the Expression of Specific Human Endogenous Retrovirus HERV-K (HML-2) Elements.Human Endogenous Retrovirus-K and TDP-43 Expression Bridges ALS and HIV Neuropathology.Hypothetical endogenous SIV-like antigens in Mauritian cynomolgus macaques.Transcriptional Modulation of Human Endogenous Retroviruses in Primary CD4+ T Cells Following Vorinostat Treatment.Human Endogenous Retrovirus K in the Crosstalk Between Cancer Cells Microenvironment and Plasticity: A New Perspective for Combination Therapy.
P2860
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P2860
Expression of human endogenous retrovirus type K (HML-2) is activated by the Tat protein of HIV-1.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Expression of human endogenous ...... d by the Tat protein of HIV-1.
@ast
Expression of human endogenous ...... d by the Tat protein of HIV-1.
@en
type
label
Expression of human endogenous ...... d by the Tat protein of HIV-1.
@ast
Expression of human endogenous ...... d by the Tat protein of HIV-1.
@en
prefLabel
Expression of human endogenous ...... d by the Tat protein of HIV-1.
@ast
Expression of human endogenous ...... d by the Tat protein of HIV-1.
@en
P2093
P2860
P356
P1433
P1476
Expression of human endogenous ...... d by the Tat protein of HIV-1.
@en
P2093
David M Markovitz
Mark H Kaplan
Marta J Gonzalez-Hernandez
Michael D Swanson
Rafael Contreras-Galindo
Richard J Noel
Sarah Cookinham
Steven R King
P2860
P304
P356
10.1128/JVI.07215-11
P407
P577
2012-05-16T00:00:00Z