Wild-type NRas and KRas perform distinct functions during transformation. - Wikidata - wikimedia - TriplyDB

Wild-type NRas and KRas perform distinct functions during transformation.

Wild-type NRas and KRas perform distinct functions during transformation.

http://www.wikidata.org/entity/Q36176614

about

Activated Ras as a Therapeutic Target: Constraints on Directly Targeting Ras Isoforms and Wild-Type versus Mutated Proteins Inferring tumor progression from genomic heterogeneity Ras, an actor on many stages: posttranslational modifications, localization, and site-specified events.Ras regulates kinesin 13 family members to control cell migration pathways in transformed human bronchial epithelial cells.Clinical relevance of the transcriptional signature regulated by CDC42 in colorectal cancer.Elevated phosphate activates N-ras and promotes cell transformation and skin tumorigenesis.Ras transformation uncouples the kinesin-coordinated cellular nutrient response MicroRNAs 206 and 21 cooperate to promote RAS-extracellular signal-regulated kinase signaling by suppressing the translation of RASA1 and SPRED1 KRAS Mouse Models: Modeling Cancer Harboring KRAS Mutations.Sos-mediated cross-activation of wild-type Ras by oncogenic Ras is essential for tumorigenesis.How to Target Activated Ras Proteins: Direct Inhibition vs. Induced Mislocalization.The role of wild type RAS isoforms in cancer.Time-resolved cellular effects induced by TcdA from Clostridium difficile.The differential effects of wild-type and mutated K-Ras on MST2 signaling are determined by K-Ras activation kinetics.Abrogation of PIK3CA or PIK3R1 reduces proliferation, migration, and invasion in glioblastoma multiforme cells.Prostaglandin E2-Dependent Phosphorylation of RAS Inhibition 1 (RIN1) at Ser 291 and 292 Inhibits Transforming Growth Factor-β-Induced RAS Activation Pathway in Human Synovial Fibroblasts: Role in Cell Migration.The miR-18a* microRNA functions as a potential tumor suppressor by targeting on K-Ras.Absence of K-Ras Reduces Proliferation and Migration But Increases Extracellular Matrix Synthesis in Fibroblasts.Tumour maintenance is mediated by eNOS.Functional specificity of ras isoforms: so similar but so different.Rare codons regulate KRas oncogenesis.CBP501 inhibits EGF-dependent cell migration, invasion and epithelial-to-mesenchymal transition of non-small cell lung cancer cells by blocking KRas to calmodulin binding.KRAS induces lung tumorigenesis through microRNAs modulation.

P2860

Q27027458-8F05002A-3F51-418F-92F1-98E0818F643C Q28749457-E3AA9FDC-AD81-4932-AEC0-8B5BCF3C305A Q33351823-D2C2555F-DBC3-4E54-8650-2A13BF9ACC8C Q33631023-93B156EA-3DF0-43B9-B198-EAC6941B95DD Q33688815-7D569278-7E15-4C5D-87EC-CBBDD498210A Q33708051-CC42F825-7492-4DE7-B2FE-6853B90BDCC1 Q33972110-A5C1C980-E03C-462B-856F-2C8656F5EFD0 Q34592684-98E6FC87-56EA-4465-910A-873777E42113 Q35083740-FDF61FD6-3590-4F61-AA9D-EEA65C6FDC54 Q36808753-BEE0614B-376E-48E4-B67D-90E914AE732E Q37113976-40892F13-C390-450D-B07A-7EA4A54626C2 Q38898397-96725F96-F688-4C00-9828-BD4B32F4B6B0 Q39006683-298C273F-1C62-4BBA-A0DF-F864BB5BC34F Q39185488-25E6F63B-099F-4932-A126-611F4B59E2B0 Q39446680-A53C4D73-CA45-496E-A93E-88C556DBD438 Q39801563-D99A4BF6-B5CF-47B6-8146-186CF634AF22 Q39860132-649EF1B3-CC39-44AE-908F-FC7449B11B94 Q39999843-26EFDACF-11E2-4820-ABF5-C72262C78C7D Q40001801-59CBC030-40FD-4D98-BDDB-58BF7AA7102E Q42784309-1B0389AD-AFAC-41BC-90B9-E092DBA39525 Q43220010-085CDD43-1E76-441C-A5C4-57773E612220 Q45897176-45990748-3A8E-4D7A-814A-36914D9A4116 Q50025863-8ECD1AAD-524A-45E3-9BD8-D353642DD1D9

P2860

Wild-type NRas and KRas perform distinct functions during transformation.

http://www.wikidata.org/entity/Q36176614

description

2007 nî lūn-bûn

@nan

2007年の論文

@ja

2007年論文

@yue

2007年論文

@zh-hant

2007年論文

@zh-hk

2007年論文

@zh-mo

2007年論文

@zh-tw

2007年论文

@wuu

2007年论文

@zh

2007年论文

@zh-cn

name

Wild-type NRas and KRas perform distinct functions during transformation.

@ast

Wild-type NRas and KRas perform distinct functions during transformation.

@en

type

label

Wild-type NRas and KRas perform distinct functions during transformation.

@ast

Wild-type NRas and KRas perform distinct functions during transformation.

@en

prefLabel

Wild-type NRas and KRas perform distinct functions during transformation.

@ast

Wild-type NRas and KRas perform distinct functions during transformation.

@en

P1433

Q36176614-A0CCD55C-CBED-430D-9FA9-F2B7CCA204FB

P1476

Q36176614-F5219912-422A-4B47-9A1B-9A356359B4CA

P2093

Q36176614-87DC4654-32DB-4030-8FA2-B8F882E5935D

Q36176614-9DA8B087-5DC8-4EBB-BF94-4115BFF15076

Q36176614-BE367720-9A2B-4D04-94B0-B0C0300AAA99

Q36176614-D45618D1-AB1F-40FD-9135-7838F2B1B599

P2860

Q36176614-0152FD04-B7DB-4E75-AA97-705E41531C95

Q36176614-121E2275-A011-4399-992D-1C71F7EBA7FF

Q36176614-12D44AF5-47AC-46C4-B5C6-D646B9E08070

Q36176614-1386061A-DB69-4456-B2B1-B8E12D57696E

Q36176614-1614B59A-AC7E-43E1-90B3-341E93581940

Q36176614-19D00AE4-D5BD-4E43-A22D-55D88F3942DF

Q36176614-2443F7AE-ED2D-4083-807B-F3AFEB3C0E7B

Q36176614-3166FAF6-7CC6-485F-ABE0-0BA8CF877BCC

Q36176614-3277038B-5BAE-45D9-9CF8-7D2FD21B1184

Q36176614-34219C93-22B8-4751-AB45-436D064D9391

P304

Q36176614-47106D7A-973B-4823-BC82-50789623475E

P31

Q36176614-89321A9E-212C-4AAA-84B3-2D3827C1773B

P356

Q36176614-C4CA6D4A-AAF6-47FB-A251-C305F8D37321

P407

Q36176614-6E9AA788-F3E7-4251-BF8E-F20E48E4BF22

P433

Q36176614-91348479-94B5-45BF-AEBE-E468C5DBABBA

P478

Q36176614-5BDC8C20-49E9-49BF-810F-54DC900C730B

P577

Q36176614-5F24203E-72AE-496F-B5B8-1F5EA23A358E

P698

Q36176614-031B0898-D60C-453C-9CBB-EF24A6E92BDA

P932

Q36176614-C11F90CF-3903-471C-A6CE-775F49B48082

P356

P1433

Molecular and Cellular Biology

P1476

Wild-type NRas and KRas perform distinct functions during transformation.

@en

P2093

Chiaki Takahashi

http://www.w3.org/2001/XMLSchema#string

Hasan N Rajabi

http://www.w3.org/2001/XMLSchema#string

Mark E Ewen

http://www.w3.org/2001/XMLSchema#string

Poppy P Fotiadou

http://www.w3.org/2001/XMLSchema#string

P2860

The Hallmarks of Cancer

Rho, rac, and cdc42 GTPases regulate the assembly of multimolecular focal complexes associated with actin stress fibers, lamellipodia, and filopodia

Endomembrane trafficking of ras: the CAAX motif targets proteins to the ER and Golgi

EPS8 and E3B1 transduce signals from Ras to Rac

mDia mediates Rho-regulated formation and orientation of stable microtubules

Cdc42-MRCK and Rho-ROCK signalling cooperate in myosin phosphorylation and cell invasion

Genetic interaction between Rb and K-ras in the control of differentiation and tumor suppression

Nuclear movement regulated by Cdc42, MRCK, myosin, and actin flow establishes MTOC polarization in migrating cells

Rho and Rac take center stage

ras oncogenes in human cancer: a review

P304

6742-6755

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1128/MCB.00234-07

http://www.w3.org/2001/XMLSchema#string

P407

P433

19

http://www.w3.org/2001/XMLSchema#string

P478

27

http://www.w3.org/2001/XMLSchema#string

P577

2007-07-16T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

17636015

http://www.w3.org/2001/XMLSchema#string

P932

2099215

http://www.w3.org/2001/XMLSchema#string