Protection against Acute Hepatocellular Injury Afforded by Liver Fibrosis Is Independent of T Lymphocytes.

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Protection against Acute Hepatocellular Injury Afforded by Liver Fibrosis Is Independent of T Lymphocytes.

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2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

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Protection against Acute Hepat ...... Independent of T Lymphocytes.

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Protection against Acute Hepat ...... Independent of T Lymphocytes.

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Item

label

Protection against Acute Hepat ...... Independent of T Lymphocytes.

@ast

Protection against Acute Hepat ...... Independent of T Lymphocytes.

@en

prefLabel

Protection against Acute Hepat ...... Independent of T Lymphocytes.

@ast

Protection against Acute Hepat ...... Independent of T Lymphocytes.

@en

P1476

Protection against Acute Hepat ...... s Independent of T Lymphocytes

@en

P2093

Benoit Lacoste

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Marc Bilodeau

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Valérie-Ann Raymond

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P2860

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Quantitative evaluation and selection of reference genes in mouse oocytes and embryos cultured in vivo and in vitro.

An IL-13 inhibitor blocks the development of hepatic fibrosis during a T-helper type 2-dominated inflammatory response.

Fibrosis-dependent mechanisms of hepatocarcinogenesis

Metabolism and toxicity of thioacetamide and thioacetamide S-oxide in rat hepatocytes

Interleukin-5 (IL-5) augments the progression of liver fibrosis by regulating IL-13 activity.

Autophagy releases lipid that promotes fibrogenesis by activated hepatic stellate cells in mice and in human tissues.

P304

e0165360

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scholarly article

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10.1371/JOURNAL.PONE.0165360

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P407

English

P433

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P478

http://www.w3.org/2001/XMLSchema#string

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Pascal Lapierre

P577

2016-10-28T00:00:00Z

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P698

27792745

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P932

5085019

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