Differential Effect of Endurance Training on Mitochondrial Protein Damage, Degradation, and Acetylation in the Context of Aging. - Wikidata - wikimedia - TriplyDB

Differential Effect of Endurance Training on Mitochondrial Protein Damage, Degradation, and Acetylation in the Context of Aging.

Differential Effect of Endurance Training on Mitochondrial Protein Damage, Degradation, and Acetylation in the Context of Aging.

http://www.wikidata.org/entity/Q36181495

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The Role of Mitochondrial DNA in Mediating Alveolar Epithelial Cell Apoptosis and Pulmonary Fibrosis Molecular mechanisms for mitochondrial adaptation to exercise training in skeletal muscle.Spermidine coupled with exercise rescues skeletal muscle atrophy from D-gal-induced aging rats through enhanced autophagy and reduced apoptosis via AMPK-FOXO3a signal pathway.Chronically endurance-trained individuals preserve skeletal muscle mitochondrial gene expression with age but differences within age groups remain.Combined training enhances skeletal muscle mitochondrial oxidative capacity independent of age Defects in mitochondrial efficiency and H2O2 emissions in obese women are restored to a lean phenotype with aerobic exercise training.Resistance training and redox homeostasis: Correlation with age-associated genomic changes Mitochondrial Aging and Physical Decline: Insights From Three Generations of Women Hypertrophy-Promoting Effects of Leucine Supplementation and Moderate Intensity Aerobic Exercise in Pre-Senescent Mice.The ageing neuromuscular system and sarcopenia: a mitochondrial perspective.The effect of acute and chronic sprint-interval training on LRP130, SIRT3, and PGC-1α expression in human skeletal muscle.Skeletal muscle aging: influence of oxidative stress and physical exercise.Radical Oxygen Species, Exercise and Aging: An Update.Omics and Exercise: Global Approaches for Mapping Exercise Biological Networks.The Antioxidant Effect of Exercise: A Systematic Review and Meta-Analysis.Beneficial effects of exercise on age-related mitochondrial dysfunction and oxidative stress in skeletal muscle.Short-term intense exercise training reduces stress markers and alters the transcriptional response to exercise in skeletal muscle.Sirtuin 3 Deregulation Promotes Pulmonary Fibrosis.Release of skeletal muscle peptide fragments identifies individual proteins degraded during insulin deprivation in type 1 diabetic humans and mice.High-intensity high-volume swimming induces more robust signaling through PGC-1α and AMPK activation than sprint interval swimming in m. triceps brachii.Cardiac Aging - Benefits of Exercise, Nrf2 Activation and Antioxidant Signaling.AMPK in skeletal muscle function and metabolism.Effect of scuba diving on the oxidant/antioxidant status, SIRT1 and SIRT3 expression in recreational divers after a winter nondive period.SIRT3 gene expression but not SIRT3 subcellular localization is altered in response to fasting and exercise in human skeletal muscle.

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Differential Effect of Endurance Training on Mitochondrial Protein Damage, Degradation, and Acetylation in the Context of Aging.

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2014 nî lūn-bûn

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The Journals of Gerontology

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Differential Effect of Enduran ...... ation in the Context of Aging.

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Adam R Konopka

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Carrie Heppelmann

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Daniel R Jakaitis

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Dawn M Morse

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Gregory C Henderson

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H Robert Bergen

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Jill M Schimke

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K Sreekumaran Nair

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Katherine A Klaus

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Matthew L Johnson

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P2860

Lon protease preferentially degrades oxidized mitochondrial aconitase by an ATP-stimulated mechanism

A mitochondrial protein compendium elucidates complex I disease biology

Aging: a theory based on free radical and radiation chemistry

Changes in rat liver mitochondria with aging. Lon protease-like reactivity and N(epsilon)-carboxymethyllysine accumulation in the matrix

Age-associated declines in mitochondrial biogenesis and protein quality control factors are minimized by exercise training

Sirt3 mediates reduction of oxidative damage and prevention of age-related hearing loss under caloric restriction

A PGC-1α isoform induced by resistance training regulates skeletal muscle hypertrophy

Aging-dependent large accumulation of point mutations in the human mtDNA control region for replication.

Decline in skeletal muscle mitochondrial function with aging in humans

Age and aerobic exercise training effects on whole body and muscle protein metabolism.

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1386-1393

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10.1093/GERONA/GLU221

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