A common cause for a common phenotype: the gatekeeper hypothesis in fetal programming.
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PPARs Link Early Life Nutritional Insults to Later Programmed Hypertension and Metabolic SyndromeLow-grade inflammation, diet composition and health: current research evidence and its translationImpaired hypothalamic mTOR activation in the adult rat offspring born to mothers fed a low-protein dietEx vivo modeling of chemical synergy in prenatal kidney cystogenesisBoth food restriction and high-fat diet during gestation induce low birth weight and altered physical activity in adult rat offspring: the "Similarities in the Inequalities" modelMethylating micronutrient supplementation during pregnancy influences foetal hepatic gene expression and IGF signalling and increases foetal weight.Intrauterine growth restriction alters term fetal baboon hypothalamic appetitive peptide balance.Genome-wide methylation and gene expression changes in newborn rats following maternal protein restriction and reversal by folic acidTranscriptome analysis in rat kidneys: importance of genes involved in programmed hypertension.Fetal exposure to placental corticotropin-releasing hormone (pCRH) programs developmental trajectories.Elevated glucocorticoids during ovine pregnancy increase appetite and produce glucose dysregulation and adiposity in their granddaughters in response to ad libitum feeding at 1 year of age.Fetal malnutrition and long-term outcomes.Exercise in obese female rats has beneficial effects on maternal and male and female offspring metabolism.Developmental Programming of Adult Disease: Reprogramming by Melatonin?Fetal programming of CVD and renal disease: animal models and mechanistic considerations.Long term effects of pre- and early postnatal nutrition and environment on the gut.Mechanisms by which maternal obesity programs offspring for obesity: evidence from animal studies.Iron deficiency during pregnancy: the consequences for placental function and fetal outcome.Adaptation or malignant transformation: the two faces of epigenetically mediated response to stress.Mechanisms involved in developmental programming of hypertension and renal diseases. Gender differences.Prenatal Maternal Cortisol Has Sex-Specific Associations with Child Brain Network Properties.Physiology's impact: discovering life.Transplacental Nutrient Transport Mechanisms of Intrauterine Growth Restriction in Rodent Models and Humans.Patterns of age-related change in reproductive effort differ in the pre-natal and post-natal periods in a long-lived mammal.Sex-specific effects of low protein diet on in utero programming of renal G-protein coupled receptors.High- and low-protein gestation diets do not provoke common transcriptional responses representing universal target-pathways in muscle and liver of porcine progeny.
P2860
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P2860
A common cause for a common phenotype: the gatekeeper hypothesis in fetal programming.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
A common cause for a common phenotype: the gatekeeper hypothesis in fetal programming.
@ast
A common cause for a common phenotype: the gatekeeper hypothesis in fetal programming.
@en
type
label
A common cause for a common phenotype: the gatekeeper hypothesis in fetal programming.
@ast
A common cause for a common phenotype: the gatekeeper hypothesis in fetal programming.
@en
prefLabel
A common cause for a common phenotype: the gatekeeper hypothesis in fetal programming.
@ast
A common cause for a common phenotype: the gatekeeper hypothesis in fetal programming.
@en
P2093
P2860
P1433
P1476
A common cause for a common phenotype: the gatekeeper hypothesis in fetal programming
@en
P2093
P2860
P356
10.1016/J.MEHY.2011.09.047
P577
2011-11-01T00:00:00Z