Protein kinase C β inhibition by enzastaurin leads to mitotic missegregation and preferential cytotoxicity toward colorectal cancer cells with chromosomal instability (CIN)
about
Chronic chemotherapeutic stress promotes evolution of stemness and WNT/beta-catenin signaling in colorectal cancer cells: implications for clinical use of WNT-signaling inhibitors.Dual inhibition of ATR and ATM potentiates the activity of trabectedin and lurbinectedin by perturbing the DNA damage response and homologous recombination repair.Molecular mechanism underlying the pharmacological interactions of the protein kinase C-β inhibitor enzastaurin and erlotinib in non-small cell lung cancer cells
P2860
Protein kinase C β inhibition by enzastaurin leads to mitotic missegregation and preferential cytotoxicity toward colorectal cancer cells with chromosomal instability (CIN)
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
Protein kinase C β inhibition ...... chromosomal instability (CIN)
@ast
Protein kinase C β inhibition ...... chromosomal instability (CIN)
@en
type
label
Protein kinase C β inhibition ...... chromosomal instability (CIN)
@ast
Protein kinase C β inhibition ...... chromosomal instability (CIN)
@en
prefLabel
Protein kinase C β inhibition ...... chromosomal instability (CIN)
@ast
Protein kinase C β inhibition ...... chromosomal instability (CIN)
@en
P2860
P1433
P1476
Protein kinase C β inhibition ...... chromosomal instability (CIN)
@en
P2093
Annette K Larsen
Djamila Ouaret
P2860
P304
P356
10.4161/15384101.2015.945383
P577
2014-01-01T00:00:00Z