Ultrastructural bases for metabolically linked mechanical activity in mitochondria. I. Reversible ultrastructural changes with change in metabolic steady state in isolated liver mitochondria.
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The molecular mechanism of Noxa-induced mitochondrial dysfunction in p53-mediated cell deathCaspase-dependent processing activates the proapoptotic activity of deleted in breast cancer-1 during tumor necrosis factor-alpha-mediated death signalingHuman ind1, an iron-sulfur cluster assembly factor for respiratory complex ILight scattering from intact cells reports oxidative-stress-induced mitochondrial swelling.The mitochondrial inner membrane protein mitofilin controls cristae morphologyThe connection between inner membrane topology and mitochondrial functionMorphological control of mitochondrial bioenergeticsGlucose stimulation induces dynamic change of mitochondrial morphology to promote insulin secretion in the insulinoma cell line INS-1EChanging perspective on oncometabolites: from metabolic signature of cancer to tumorigenic and immunosuppressive agentsMechanism of action of reagents that uncouple oxidative phosphorylationOPA1-dependent cristae modulation is essential for cellular adaptation to metabolic demandThe OPA1-dependent mitochondrial cristae remodeling pathway controls atrophic, apoptotic, and ischemic tissue damageImbalanced OPA1 processing and mitochondrial fragmentation cause heart failure in miceFactors affecting the susceptibility of staphylococci to killing by the cationic proteins from rabbit polymorphonuclear leucocytes: the effects of alteration of cellular energetics and of various iron compoundsShaping the role of mitochondria in the pathogenesis of Huntington's diseaseSLP-2 is required for stress-induced mitochondrial hyperfusionCardiolipin, a critical determinant of mitochondrial carrier protein assembly and function.Mitochondrial Ca2+ dynamics reveals limited intramitochondrial Ca2+ diffusionNitric oxide-induced mitochondrial fission is regulated by dynamin-related GTPases in neurons.Electron tomographic analysis of cytoskeletal cross-bridges in the paranodal region of the node of Ranvier in peripheral nerves.Elevated hydrostatic pressure triggers release of OPA1 and cytochrome C, and induces apoptotic cell death in differentiated RGC-5 cells.Localization of the enzymes of ketogenesis in rat liver mitochondria.A reduction in ATP demand and mitochondrial activity with neural differentiation of human embryonic stem cells.Electron tomography of paranodal septate-like junctions and the associated axonal and glial cytoskeletons in the central nervous system.Transient contraction of mitochondria induces depolarization through the inner membrane dynamin OPA1 protein.Altered mitochondrial structure and motion dynamics in living cells with energy metabolism defects revealed by real time microscope imaging.Spatiotemporal regulation of ATP and Ca2+ dynamics in vertebrate rod and cone ribbon synapses.Mitochondrial viability in mouse and human postmortem brainIon transport underlying metabolically controlled volume changes of isolated mitochondriaMigration of mitochondria to viral assembly sites in African swine fever virus-infected cells.Closure of VDAC causes oxidative stress and accelerates the Ca(2+)-induced mitochondrial permeability transition in rat liver mitochondriaTopological and functional organization of the mitochondrion.Abnormalities of Mitochondrial Dynamics in Neurodegenerative Diseases.Contribution of the mitochondrial compartment to the optical properties of the rat liver: a theoretical and practical approach.Mitochondrial shape changes: orchestrating cell pathophysiology.Calcium-induced alterations in mitochondrial morphology quantified in situ with optical scatter imagingMitochondrial respiratory control is lost during growth factor deprivation.The large-scale separation of peroxisomes, mitochondria, and lysosomes from the livers of rats injected with triton WR-1339. Improved isolation procedures, automated analysis, biochemical and morphological properties of fractions.Increased production of reactive oxygen species in hyperglycemic conditions requires dynamic change of mitochondrial morphology.Flow cytometry of isolated mitochondria during development and under some pathological conditions.
P2860
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P2860
Ultrastructural bases for metabolically linked mechanical activity in mitochondria. I. Reversible ultrastructural changes with change in metabolic steady state in isolated liver mitochondria.
description
1966 nî lūn-bûn
@nan
1966年の論文
@ja
1966年論文
@yue
1966年論文
@zh-hant
1966年論文
@zh-hk
1966年論文
@zh-mo
1966年論文
@zh-tw
1966年论文
@wuu
1966年论文
@zh
1966年论文
@zh-cn
name
Ultrastructural bases for meta ...... n isolated liver mitochondria.
@ast
Ultrastructural bases for meta ...... n isolated liver mitochondria.
@en
type
label
Ultrastructural bases for meta ...... n isolated liver mitochondria.
@ast
Ultrastructural bases for meta ...... n isolated liver mitochondria.
@en
prefLabel
Ultrastructural bases for meta ...... n isolated liver mitochondria.
@ast
Ultrastructural bases for meta ...... n isolated liver mitochondria.
@en
P2860
P356
P1476
Ultrastructural bases for meta ...... n isolated liver mitochondria.
@en
P2093
Hackenbrock CR
P2860
P304
P356
10.1083/JCB.30.2.269
P407
P577
1966-08-01T00:00:00Z