Ubiquitin-H2AX fusions render 53BP1 recruitment to DNA damage sites independent of RNF8 or RNF168
about
Choreographing the Double Strand Break Response: Ubiquitin and SUMO Control of Nuclear ArchitectureLow-dose ionizing irradiation triggers a 53BP1 response to DNA double strand breaks in mouse spermatogonial stem cells.Tumors overexpressing RNF168 show altered DNA repair and responses to genotoxic treatments, genomic instability and resistance to proteotoxic stress.Chronic oxidative stress promotes H2AX protein degradation and enhances chemosensitivity in breast cancer patients.Reading chromatin signatures after DNA double-strand breaks.Epigenomic Modifications Mediating Antibody Maturation.A bi-terminal protein ligation strategy to probe chromatin structure during DNA damage.
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Q26744843-EE7EDC9E-CD4E-4905-AB8F-6731F92C19B1Q35917737-3DE684E8-7503-4EE6-B426-123D3869A3E3Q36191522-81905E4D-2C97-4FCC-A264-65A1FE37285DQ37441289-E2FF83AB-63D0-47D4-A8EE-B642EF3C94A5Q50494189-0A27D4EA-71A1-4FED-BBB9-F4611D56B873Q55011560-3CC341FA-60C3-4284-914D-EDDDE1921B5AQ55251925-9A81105F-42E7-4F93-A24B-A4558049AC10
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Ubiquitin-H2AX fusions render 53BP1 recruitment to DNA damage sites independent of RNF8 or RNF168
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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name
Ubiquitin-H2AX fusions render ...... independent of RNF8 or RNF168
@ast
Ubiquitin-H2AX fusions render ...... independent of RNF8 or RNF168
@en
type
label
Ubiquitin-H2AX fusions render ...... independent of RNF8 or RNF168
@ast
Ubiquitin-H2AX fusions render ...... independent of RNF8 or RNF168
@en
prefLabel
Ubiquitin-H2AX fusions render ...... independent of RNF8 or RNF168
@ast
Ubiquitin-H2AX fusions render ...... independent of RNF8 or RNF168
@en
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P2860
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Ubiquitin-H2AX fusions render ...... independent of RNF8 or RNF168
@en
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Alix J Rey
Maciej K Kocyłowski
Thanos D Halazonetis
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P304
P356
10.1080/15384101.2015.1010918
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2015-01-01T00:00:00Z