Molecular mechanisms of neuropathic pain-phenotypic switch and initiation mechanisms.
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Biological roles of lysophospholipid receptors revealed by genetic null mice: an update.Antinociceptive effect of cyclic phosphatidic acid and its derivative on animal models of acute and chronic pain.Kinin B1 receptors contributes to acute pain following minor surgery in humans.Forced exercise attenuates neuropathic pain in chronic constriction injury of male rat: an investigation of oxidative stress and inflammationPre-emptive morphine treatment abolishes nerve injury-induced lysophospholipid synthesis in mass spectrometrical analysis.Targeting TRPV1 as an alternative approach to narcotic analgesics to treat chronic pain conditions.Parathyroid hormone 2 receptor is a functional marker of nociceptive myelinated fibers responsible for neuropathic pain.Surgical decompression of painful diabetic peripheral neuropathy: the role of pain distribution.Aiming drug discovery at lysophosphatidic acid targets.Distribution of endogenous farnesyl pyrophosphate and four species of lysophosphatidic acid in rodent brain.An LPA species (18:1 LPA) plays key roles in the self-amplification of spinal LPA production in the peripheral neuropathic pain model.σ1 receptors activate astrocytes via p38 MAPK phosphorylation leading to the development of mechanical allodynia in a mouse model of neuropathic pain.Dorsal root ganglion - a potential new therapeutic target for neuropathic painProgesterone produces antinociceptive and neuroprotective effects in rats with microinjected lysophosphatidic acid in the trigeminal nerve root.Blockage of lysophosphatidic acid signaling improves spinal cord injury outcomes.Effect of the cannabinoid CB1 receptor antagonist rimonabant on nociceptive responses and adjuvant-induced arthritis in obese and lean rats.Long-range regulatory synergy is required to allow control of the TAC1 locus by MEK/ERK signalling in sensory neurones.Peripheral mechanisms of neuropathic pain - involvement of lysophosphatidic acid receptor-mediated demyelination.Animal models of cancer pain.Schwann cell LRP1 regulates remak bundle ultrastructure and axonal interactions to prevent neuropathic pain.Lysophosphatidic acid-3 receptor-mediated feed-forward production of lysophosphatidic acid: an initiator of nerve injury-induced neuropathic pain.Lysophosphatidic acid induces vasodilation mediated by LPA1 receptors, phospholipase C, and endothelial nitric oxide synthase.Cutting-edge issues in primary biliary cirrhosis.Targeting pain mediators induced by injured nerve-derived COX2 and PGE2 to treat neuropathic pain.Ephrins and pain.Challenges to develop novel anti-inflammatory and analgesic drugs.Functional up-regulation of Nav1.8 sodium channel in Aβ afferent fibers subjected to chronic peripheral inflammation.The molecular mechanism of cholestatic pruritus.PKC-NF-κB are involved in CCL2-induced Nav1.8 expression and channel function in dorsal root ganglion neuronsInvolvement of LPA1 receptor signaling in the reorganization of spinal input through Abeta-fibers in mice with partial sciatic nerve injury.Pharmacological switch in Abeta-fiber stimulation-induced spinal transmission in mice with partial sciatic nerve injury.Distinct calcitonin gene-related peptide expression pattern in primary afferents contribute to different neuropathic symptoms following chronic constriction or crush injuries to the rat sciatic nerve.Characterization of three different sensory fibers by use of neonatal capsaicin treatment, spinal antagonism and a novel electrical stimulation-induced paw flexion test.Autotaxin and lysophosphatidic acid1 receptor-mediated demyelination of dorsal root fibers by sciatic nerve injury and intrathecal lysophosphatidylcholine.The control of alternative splicing by SRSF1 in myelinated afferents contributes to the development of neuropathic pain.Dynamic response to peripheral nerve injury detected by in situ hybridization of IL-6 and its receptor mRNAs in the dorsal root ganglia is not strictly correlated with signs of neuropathic pain.Calpain-mediated down-regulation of myelin-associated glycoprotein in lysophosphatidic acid-induced neuropathic painEvidence for regulatory diversity and auto-regulation at the TAC1 locus in sensory neurones.Activation of TRESK channels by the inflammatory mediator lysophosphatidic acid balances nociceptive signalling.HDAC inhibitors restore C-fibre sensitivity in experimental neuropathic pain model.
P2860
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P2860
Molecular mechanisms of neuropathic pain-phenotypic switch and initiation mechanisms.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Molecular mechanisms of neuropathic pain-phenotypic switch and initiation mechanisms.
@ast
Molecular mechanisms of neuropathic pain-phenotypic switch and initiation mechanisms.
@en
type
label
Molecular mechanisms of neuropathic pain-phenotypic switch and initiation mechanisms.
@ast
Molecular mechanisms of neuropathic pain-phenotypic switch and initiation mechanisms.
@en
prefLabel
Molecular mechanisms of neuropathic pain-phenotypic switch and initiation mechanisms.
@ast
Molecular mechanisms of neuropathic pain-phenotypic switch and initiation mechanisms.
@en
P1476
Molecular mechanisms of neuropathic pain-phenotypic switch and initiation mechanisms.
@en
P2093
Hiroshi Ueda
P356
10.1016/J.PHARMTHERA.2005.06.003
P577
2005-07-15T00:00:00Z