Highly lytic CD8+, alpha beta T-cell receptor cytotoxic T cells with major histocompatibility complex (MHC) class I antigen-directed cytotoxicity in beta 2-microglobulin, MHC class I-deficient mice
about
Generation of alphabeta T-cell receptor+ CD4- CD8+ cells in major histocompatibility complex class I-deficient mice upon activation of the aryl hydrocarbon receptor by 2,3,7,8-tetrachlorodibenzo-p-dioxinLethal host-versus-graft disease and hypereosinophilia in the absence of MHC I-T-cell interactions.Gene knockout mice establish a primary protective role for major histocompatibility complex class II-restricted responses in Chlamydia trachomatis genital tract infection.Positive selection of self- and alloreactive CD8+ T cells in Tap-1 mutant mice.Virus-specific, CD8+ major histocompatibility complex class I-restricted cytotoxic T lymphocytes in lymphocytic choriomeningitis virus-infected beta2-microglobulin-deficient mice.Evidence for a single-niche model of positive selection.Transient up-regulation of P2Y2 nucleotide receptor mRNA expression is an immediate early gene response in activated thymocytesPurified hematopoietic stem cell engraftment of rare niches corrects severe lymphoid deficiencies without host conditioning.Emergence of NK1.1+ cells as effectors of IFN-gamma dependent immunity to Toxoplasma gondii in MHC class I-deficient mice.Increase in positive selection of CD8+ T cells in TAP1-mutant mice by human beta 2-microglobulin transgeneMajor histocompatibility complex (MHC) class I KbDb -/- deficient mice possess functional CD8+ T cells and natural killer cellsPhosphorylation of extracellular domains of T-lymphocyte surface proteins. Constitutive serine and threonine phosphorylation of the T cell antigen receptor ectodomains.The CD8+ T cell repertoire in beta 2-microglobulin-deficient mice is biased towards reactivity against self-major histocompatibility class I.Vaccination with DNA encoding internal proteins of influenza virus does not require CD8(+) cytotoxic T lymphocytes: either CD4(+) or CD8(+) T cells can promote survival and recovery after challenge.Analysis of graft-versus-host disease (GVHD) and graft rejection using MHC class I-deficient mice.Autoreactivity to self H-2Kb peptides in TAP1 mice. Intravenous administration of H-2Kb class I-derived peptides induces long-term survival of grafts from C57BL/6 donors.
P2860
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P2860
Highly lytic CD8+, alpha beta T-cell receptor cytotoxic T cells with major histocompatibility complex (MHC) class I antigen-directed cytotoxicity in beta 2-microglobulin, MHC class I-deficient mice
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Highly lytic CD8+, alpha beta ...... in, MHC class I-deficient mice
@ast
Highly lytic CD8+, alpha beta ...... in, MHC class I-deficient mice
@en
type
label
Highly lytic CD8+, alpha beta ...... in, MHC class I-deficient mice
@ast
Highly lytic CD8+, alpha beta ...... in, MHC class I-deficient mice
@en
prefLabel
Highly lytic CD8+, alpha beta ...... in, MHC class I-deficient mice
@ast
Highly lytic CD8+, alpha beta ...... in, MHC class I-deficient mice
@en
P2860
P356
P1476
Highly lytic CD8+, alpha beta ...... in, MHC class I-deficient mice
@en
P2093
P2860
P304
P356
10.1073/PNAS.90.7.2837
P407
P577
1993-04-01T00:00:00Z