Enhancement of 26S proteasome functionality connects oxidative stress and vascular endothelial inflammatory response in diabetes mellitus.
about
Priming the proteasome by protein kinase G: a novel cardioprotective mechanism of sildenafilIdentification of nitric oxide as an endogenous inhibitor of 26S proteasomes in vascular endothelial cells.Proteasomal degradation of O-GlcNAc transferase elevates hypoxia-induced vascular endothelial inflammatory response†.Recent highlights of ATVB: diabetes mellitus.Downregulation of type II diabetes mellitus and maturity onset diabetes of young pathways in human pancreatic islets from hyperglycemic donors.Upregulation of Unc-51-like kinase 1 by nitric oxide stabilizes SIRT1, independent of autophagy.Participation of proteasome-ubiquitin protein degradation in autophagy and the activation of AMP-activated protein kinase.Resveratrol Rescues Kidney Mitochondrial Function Following Hemorrhagic Shock.High glucose and diabetes modulate cellular proteasome function: Implications in the pathogenesis of diabetes complications.Preventive and therapeutic effects of MG132 by activating Nrf2-ARE signaling pathway on oxidative stress-induced cardiovascular and renal injuryTherapeutic effect of MG132 on the aortic oxidative damage and inflammatory response in OVE26 type 1 diabetic mice.Nitric oxide differentially affects proteasome activator 28 after arterial injury in type 1 and type 2 diabetic ratsChitosan oligosaccharides block LPS-induced O-GlcNAcylation of NF-κB and endothelial inflammatory response.Role of mitochondrial dysfunction and altered autophagy in cardiovascular aging and disease: from mechanisms to therapeutics.Activation of activator protein 2 alpha by aspirin alleviates atherosclerotic plaque growth and instability in vivoLovastatin upregulates microRNA-29b to reduce oxidative stress in rats with multiple cardiovascular risk factors.Impaired proteostasis: role in the pathogenesis of diabetes mellitus.Therapeutic effect of MG-132 on diabetic cardiomyopathy is associated with its suppression of proteasomal activities: roles of Nrf2 and NF-κB.1,4-Dihydropyridine derivatives without Ca2+-antagonist activity up-regulate Psma6 mRNA expression in kidneys of intact and diabetic rats.
P2860
Q28084015-F55950F9-1016-49F3-ABD5-33445B4E393DQ33650608-39FEEC68-C078-4777-82DC-33E416F30CD8Q34048268-04E1F75E-ADA7-4F8F-ADB9-F9365FA95D19Q34088564-99EFE8EF-977E-4F9F-907F-189801D1D4FEQ34419372-424A9211-F6C2-46DD-8C64-8CC77DADD8B5Q34777068-ACC1E9AA-3AEB-4A54-A671-5DE29AA434C0Q35235799-12B6B5B4-6EA4-48F4-BEF0-AEE713513B28Q35858550-7044E2F2-F62A-4966-9DC2-2A6E1B85944CQ36677830-58809E50-285D-495E-8616-6855B3D54DECQ36711841-12AACBC3-5F5A-488B-9FCB-1AC9B4681CB5Q36754958-BD4B4836-157F-4F1D-ABAF-7A59727E9CC0Q36951713-35B2A304-87C6-4095-8FFD-5DA95B5BBD18Q37349166-CB821CA2-5D42-4DDF-BF2E-AB41F3277C2DQ37476530-69DCC719-C39C-4EFC-9ABA-F463115735ECQ37619656-1BF3F7FF-E7D9-4F16-921E-101BF75C4D9CQ37705906-3FEC5741-AB7A-4960-B5CD-FF117BA66F4FQ38210860-D0D34DBC-646A-48CF-9924-E8CD55F8ACEBQ44010395-F3E5ED2B-2D92-409B-A6C6-DCAC7668B6E3Q53393393-2086AE4B-74F1-4550-B693-E1934575BA70
P2860
Enhancement of 26S proteasome functionality connects oxidative stress and vascular endothelial inflammatory response in diabetes mellitus.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Enhancement of 26S proteasome ...... response in diabetes mellitus.
@ast
Enhancement of 26S proteasome ...... response in diabetes mellitus.
@en
type
label
Enhancement of 26S proteasome ...... response in diabetes mellitus.
@ast
Enhancement of 26S proteasome ...... response in diabetes mellitus.
@en
prefLabel
Enhancement of 26S proteasome ...... response in diabetes mellitus.
@ast
Enhancement of 26S proteasome ...... response in diabetes mellitus.
@en
P2093
P2860
P1476
Enhancement of 26S proteasome ...... response in diabetes mellitus.
@en
P2093
P2860
P304
P356
10.1161/ATVBAHA.112.253385
P407
P577
2012-07-05T00:00:00Z