Heparin selectively inhibits a protein kinase C-dependent mechanism of cell cycle progression in calf aortic smooth muscle cells
about
Human fibroblast growth factor 1 gene expression in vascular smooth muscle cells is modulated via an alternate promoter in response to serum and phorbol esterCCN5 is a growth arrest-specific gene that regulates smooth muscle cell proliferation and motilityPlatelet-derived growth factor modulates rat vascular smooth muscle cell responses on laminin-5 via mitogen-activated protein kinase-sensitive pathwaysCCN5 modulates the antiproliferative effect of heparin and regulates cell motility in vascular smooth muscle cellsThe S18 ribosomal protein is a putative substrate for Ca2+/calmodulin-activated protein kinase IIHeparin inhibits c-fos and c-myc mRNA expression in vascular smooth muscle cellsInhibitory effects of antisense oligodeoxynucleotides targeting c-myc mRNA on smooth muscle cell proliferation and migrationCOP-1, a member of the CCN family, is a heparin-induced growth arrest specific gene in vascular smooth muscle cells.Heparin inhibits the induction of three matrix metalloproteinases (stromelysin, 92-kD gelatinase, and collagenase) in primate arterial smooth muscle cells.Characterization of [3H]-heparin binding in human vascular smooth muscle cells and its relationship to the inhibition of DNA synthesis.Proteoglycans in cell regulation.Heparin inhibits phosphorylation and autonomous activity of Ca(2+)/calmodulin-dependent protein kinase II in vascular smooth muscle cells.Characterization of human aortic smooth muscle cells expressing HPV16 E6 and E7 open reading frames.Basic fibroblast growth factor promotes proliferation of rat glomerular visceral epithelial cells in vitro.Elevated expression of basic fibroblast growth factor in an immortalized rabbit smooth muscle cell linePathogenic factors in vascular dementia and Alzheimer's disease. Multiple actions of heparin that probably are beneficial.The heparins and cancer: review of clinical trials and biological properties.Smooth Muscle Stiffness Sensitivity is Driven by Soluble and Insoluble ECM ChemistryIsolation of vascular smooth muscle cell cultures with altered responsiveness to the antiproliferative effect of heparin.Delivery of large biopharmaceuticals from cardiovascular stents: a reviewInterleukin 1 regulates heparin-binding growth factor 2 gene expression in vascular smooth muscle cells.The Ca2(+)-binding glycoprotein SPARC modulates cell cycle progression in bovine aortic endothelial cells.Regulation of vascular smooth muscle proliferation by heparin: inhibition of cyclin-dependent kinase 2 activity by p27(kip1).Heparin sensitive and resistant vascular smooth muscle cells: biology and role in restenosis.Inhibition of phosphatidylinositol 4-phosphate kinase by heparin. A possible mechanism for the antiproliferative effects of heparinHeparin responses in vascular smooth muscle cells involve cGMP-dependent protein kinase (PKG)Inhibition of endothelial cell proliferation by platelet factor-4 involves a unique action on S phase progressionEntry and distribution of fluorescent antiproliferative heparin derivatives into rat vascular smooth muscle cells: comparison between heparin-sensitive and heparin-resistant cultures.Heparin inhibits mitogen-activated protein kinase-dependent and -independent c-fos induction in mesangial cells.Heparin alters epidermal growth factor metabolism in cultured rat glomerular epithelial cellsHeparin inhibits Ca2+/calmodulin-dependent kinase II activation and c-fos induction in mesangial cells.Heparin-like molecules inhibit pulmonary vascular pericyte proliferation in vitro.Antibodies against a putative heparin receptor slow cell proliferation and decrease MAPK activation in vascular smooth muscle cells.Atrial natriuretic factor inhibits mitogen-induced growth in aortic smooth muscle cells.Basic fibroblast growth factor inhibits cell proliferation in cultured avian inner ear sensory epithelia.Maintenance of contractility in dissociated smooth muscle: low-density cultures in a defined medium.Heparin down-regulates the phorbol ester-induced protein kinase C gene expression in human endothelial cells: enzyme-mediated autoregulation of protein kinase C-alpha and -delta genes.Characterization of rat aortic smooth muscle cells resistant to the antiproliferative activity of heparin following long-term heparin treatment.Cyclosporine A inhibits induction of DNA synthesis by PDGF and other peptide mitogens in cultured rat aortic smooth muscle cells and dermal fibroblasts.
P2860
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P2860
Heparin selectively inhibits a protein kinase C-dependent mechanism of cell cycle progression in calf aortic smooth muscle cells
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
1989年论文
@zh
1989年论文
@zh-cn
name
Heparin selectively inhibits a ...... alf aortic smooth muscle cells
@ast
Heparin selectively inhibits a ...... alf aortic smooth muscle cells
@en
type
label
Heparin selectively inhibits a ...... alf aortic smooth muscle cells
@ast
Heparin selectively inhibits a ...... alf aortic smooth muscle cells
@en
prefLabel
Heparin selectively inhibits a ...... alf aortic smooth muscle cells
@ast
Heparin selectively inhibits a ...... alf aortic smooth muscle cells
@en
P2093
P2860
P356
P1476
Heparin selectively inhibits a ...... alf aortic smooth muscle cells
@en
P2093
J J Castellot
M J Karnovsky
T C Wright
P2860
P304
P356
10.1083/JCB.109.6.3147
P407
P433
P577
1989-12-01T00:00:00Z