about
Constitutive heterochromatin formation and transcription in mammalsMicronuclei in genotoxicity assessment: from genetics to epigenetics and beyondLsh is involved in de novo methylation of DNALsh mediated RNA polymerase II stalling at HoxC6 and HoxC8 involves DNA methylationLSH and G9a/GLP complex are required for developmentally programmed DNA methylation.Lymphoid-specific helicase (HELLS) is essential for meiotic progression in mouse spermatocytes.DNA hypomethylation caused by Lsh deletion promotes erythroleukemia development.The ATP binding site of the chromatin remodeling homolog Lsh is required for nucleosome density and de novo DNA methylation at repeat sequencesLsh participates in DNA methylation and silencing of stem cell genesThe LSH/DDM1 Homolog MUS-30 Is Required for Genome Stability, but Not for DNA Methylation in Neurospora crassa.Lsh controls Hox gene silencing during developmentVezf1 regulates genomic DNA methylation through its effects on expression of DNA methyltransferase Dnmt3bThe SNF2 family ATPase LSH promotes cell-autonomous de novo DNA methylation in somatic cells.Hypomethylation of repeated DNA sequences in cancer.Lsh Is Essential for Maintaining Global DNA Methylation Levels in Amphibia and Fish and Interacts Directly with Dnmt1.Allosteric control of mammalian DNA methyltransferases - a new regulatory paradigm.Enzymology of Mammalian DNA Methyltransferases.The Role of p16INK4a Pathway in Human Epidermal Stem Cell Self-Renewal, Aging and CancerMethylating the DNA of the most repressed: special access required.Identification of multiple distinct Snf2 subfamilies with conserved structural motifs.ΔNp63α is an oncogene that targets chromatin remodeler Lsh to drive skin stem cell proliferation and tumorigenesis.Hypomethylation and genome instability in the germline of exposed parents and their progeny is associated with altered miRNA expression.HELLS and CDCA7 comprise a bipartite nucleosome remodeling complex defective in ICF syndrome.Tethering of Lsh at the Oct4 locus promotes gene repression associated with epigenetic changes.Lsh is required for meiotic chromosome synapsis and retrotransposon silencing in female germ cells.
P2860
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P2860
description
2005 nî lūn-bûn
@nan
2005年の論文
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2005年論文
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2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
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2005年論文
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2005年论文
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2005年论文
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name
Lsh, a guardian of heterochromatin at repeat elements.
@ast
Lsh, a guardian of heterochromatin at repeat elements.
@en
type
label
Lsh, a guardian of heterochromatin at repeat elements.
@ast
Lsh, a guardian of heterochromatin at repeat elements.
@en
prefLabel
Lsh, a guardian of heterochromatin at repeat elements.
@ast
Lsh, a guardian of heterochromatin at repeat elements.
@en
P2860
P356
P1476
Lsh, a guardian of heterochromatin at repeat elements.
@en
P2093
Kathrin Muegge
P2860
P304
P356
10.1139/O05-119
P577
2005-08-01T00:00:00Z