Role of Toll-like receptor 4 in induction of cell-mediated immunity and resistance to Brucella abortus infection in mice
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The role of TLR4 896 A>G and 1196 C>T in susceptibility to infections: a review and meta-analysis of genetic association studiesCrystal Structures of the Toll/Interleukin-1 Receptor (TIR) Domains from the Brucella Protein TcpB and Host Adaptor TIRAP Reveal Mechanisms of Molecular MimicryToll-Like Receptors 2 and 4 Cooperate in the Control of the Emerging Pathogen Brucella microtiBrucella abortus uses a stealthy strategy to avoid activation of the innate immune system during the onset of infection.Regulation of the mitogen-activated protein kinases by Brucella spp. expressing a smooth and rough phenotype: relationship to pathogen invasiveness.MyD88 and STING signaling pathways are required for IRF3-mediated IFN-β induction in response to Brucella abortus infectionGut microbiota is a key modulator of insulin resistance in TLR 2 knockout mice.Deep-sequencing analysis of the mouse transcriptome response to infection with Brucella melitensis strains of differing virulence.Toll-like receptor (TLR) 2 and TLR9 expressed in trigeminal ganglia are critical to viral control during herpes simplex virus 1 infectionIntestinal ischemia-reperfusion of macaques triggers a strong innate immune responseToll-like receptor 4 contributes to colitis development but not to host defense during Citrobacter rodentium infection in mice.The Attenuated Brucella abortus Strain 19 Invades, Persists in, and Activates Human Dendritic Cells, and Induces the Secretion of IL-12p70 but Not IL-23Lethal encephalitis in myeloid differentiation factor 88-deficient mice infected with herpes simplex virus 1.High-mobility group box-1 impairs memory in mice through both toll-like receptor 4 and Receptor for Advanced Glycation End Products.Pathogenesis and immunobiology of brucellosis: review of Brucella-host interactionsTrypanosoma cruzi and its components as exogenous mediators of inflammation recognized through Toll-like receptors.Role of the Toll/interleukin-1 receptor signaling pathway in host resistance and pathogenesis during infection with protozoan parasites.What have we learned from brucellosis in the mouse model?Toll-like receptors are critical for clearance of Brucella and play different roles in development of adaptive immunity following aerosol challenge in miceBrucella spp noncanonical LPS: structure, biosynthesis, and interaction with host immune system.Modulation of the bovine trophoblastic innate immune response by Brucella abortus.Subversion of innate immune responses by Brucella through the targeted degradation of the TLR signaling adapter, MALToll-like receptor 4-linked Janus kinase 2 signaling contributes to internalization of Brucella abortus by macrophagesEfficient production of pronuclear embryos in breeding and nonbreeding season for generating transgenic sheep overexpressing TLR4Key role of Toll-like receptor 2 in the inflammatory response and major histocompatibility complex class ii downregulation in Brucella abortus-infected alveolar macrophages.Confronting the barriers to develop novel vaccines against brucellosis.Serving the new masters - dendritic cells as hosts for stealth intracellular bacteria.Brucella melitensis triggers time-dependent modulation of apoptosis and down-regulation of mitochondrion-associated gene expression in mouse macrophages.Temporal Role for MyD88 in a Model of Brucella-Induced Arthritis and Musculoskeletal Inflammation.Nucleotide-binding oligomerization domain-1 and -2 play no role in controlling Brucella abortus infection in mice.NLRP12 negatively regulates proinflammatory cytokine production and host defense against Brucella abortus.Interleukin-1 receptor-associated kinase 4 is essential for initial host control of Brucella abortus infection.Toll-like receptor 6 plays an important role in host innate resistance to Brucella abortus infection in miceTLR2 and TLR4 signaling pathways are required for recombinant Brucella abortus BCSP31-induced cytokine production, functional upregulation of mouse macrophages, and the Th1 immune response in vivo and in vitroBtp Proteins from Brucella abortus Modulate the Lung Innate Immune Response to Infection by the Respiratory Route.Interaction of Brucella suis and Brucella abortus rough strains with human dendritic cells.MyD88, but not toll-like receptors 4 and 2, is required for efficient clearance of Brucella abortus.Macrophage-elicited osteoclastogenesis in response to Brucella abortus infection requires TLR2/MyD88-dependent TNF-α production.Immunoproteasome subunits are required for CD8+ T cell function and host resistance to Brucella abortus infection in mice.Characterization of biological activities of Brucella melitensis lipopolysaccharide.
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P2860
Role of Toll-like receptor 4 in induction of cell-mediated immunity and resistance to Brucella abortus infection in mice
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Role of Toll-like receptor 4 i ...... ella abortus infection in mice
@ast
Role of Toll-like receptor 4 i ...... ella abortus infection in mice
@en
type
label
Role of Toll-like receptor 4 i ...... ella abortus infection in mice
@ast
Role of Toll-like receptor 4 i ...... ella abortus infection in mice
@en
prefLabel
Role of Toll-like receptor 4 i ...... ella abortus infection in mice
@ast
Role of Toll-like receptor 4 i ...... ella abortus infection in mice
@en
P2093
P2860
P1476
Role of Toll-like receptor 4 i ...... ella abortus infection in mice
@en
P2093
Bruce W Jarvis
Gary A Splitter
Gracia M S Rosinha
Igor C Almeida
Marco A Campos
Nilofer Qureshi
Oscar Bruna-Romero
Ricardo T Gazzinelli
Sergio C Oliveira
Xirlene S Salgueiro
P2860
P304
P356
10.1128/IAI.72.1.176-186.2004
P407
P577
2004-01-01T00:00:00Z