Impairment of synaptic plasticity by the stress mediator CRH involves selective destruction of thin dendritic spines via RhoA signaling - Wikidata - wikimedia - TriplyDB

Impairment of synaptic plasticity by the stress mediator CRH involves selective destruction of thin dendritic spines via RhoA signaling

Impairment of synaptic plasticity by the stress mediator CRH involves selective destruction of thin dendritic spines via RhoA signaling

http://www.wikidata.org/entity/Q36230118

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Dendritic Spines in Depression: What We Learned from Animal Models Rapid effects of oestrogen on synaptic plasticity: interactions with actin and its signalling proteins Galanin mediates features of neural and behavioral stress resilience afforded by exercise.Preferential loss of dorsal-hippocampus synapses underlies memory impairments provoked by short, multimodal stress.Early life stress leads to developmental and sex selective effects on performance in a novel object placement task Trophic Mechanisms for Exercise-Induced Stress Resilience: Potential Role of Interactions between BDNF and Galanin Effect of sulfonylureas administered centrally on the blood glucose level in immobilization stress model Estrogen promotes learning-related plasticity by modifying the synaptic cytoskeleton.Corticotropin-Releasing Hormone Receptor Type 1 (CRHR1) Clustering with MAGUKs Is Mediated via Its C-Terminal PDZ Binding Motif.Elimination of dendritic spines with long-term memory is specific to active circuits Toward Understanding How Early-Life Stress Reprograms Cognitive and Emotional Brain Networks.Short-term modern life-like stress exacerbates Aβ-pathology and synapse loss in 3xTg-AD mice The neuron-specific chromatin regulatory subunit BAF53b is necessary for synaptic plasticity and memory NMDA receptor activation and calpain contribute to disruption of dendritic spines by the stress neuropeptide CRH.Converging, Synergistic Actions of Multiple Stress Hormones Mediate Enduring Memory Impairments after Acute Simultaneous Stresses.Early-life stress impairs recognition memory and perturbs the functional maturation of prefrontal-hippocampal-perirhinal networks Early life stress accelerates behavioral and neural maturation of the hippocampus in male mice.Corticotropin releasing factor in neuroplasticity.The adaptive and maladaptive continuum of stress responses - a hippocampal perspective.Lack of CRH Affects the Behavior but Does Not Affect the Formation of Short-Term Memory.The CRF family of neuropeptides and their receptors - mediators of the central stress response.Drebrin depletion alters neurotransmitter receptor levels in protein complexes, dendritic spine morphogenesis and memory-related synaptic plasticity in the mouse hippocampus.Emotional learning, stress, and development: An ever-changing landscape shaped by early-life experience.Morphine-Associated Contextual Cues Induce Structural Plasticity in Hippocampal CA1 Pyramidal Neurons.Tuning synaptic transmission in the hippocampus by stress: the CRH system.Mechanism based approaches for rescuing and enhancing cognition.Nectin-3 links CRHR1 signaling to stress-induced memory deficits and spine loss.Stress and Seizures: Space, Time and Hippocampal Circuits.Early motor phenotype detection in a female mouse model of Rett syndrome is improved by cross-fostering.2.45 GHz Microwave radiation impairs hippocampal learning and spatial memory: Involvement of local stress mechanism induced suppression of iGluR/ERK/CREB signaling.Hippocampal corticotropin-releasing hormone neurons support recognition memory and modulate hippocampal excitability.Early postnatal stress suppresses the developmental trajectory of hippocampal pyramidal neurons: the role of CRHR1.Involvement of α(2)-adrenergic receptor in the regulation of the blood glucose level induced by immobilization stress.Genome-wide DNA hydroxymethylation identifies potassium channels in the nucleus accumbens as discriminators of methamphetamine addiction and abstinence.Effect of histamine receptors agonists or antagonists administered intracerebroventricularly and intrathecally on the blood glucose level in immobilization stress model

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P2860

Impairment of synaptic plasticity by the stress mediator CRH involves selective destruction of thin dendritic spines via RhoA signaling

http://www.wikidata.org/entity/Q36230118

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Impairment of synaptic plastic ...... itic spines via RhoA signaling

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Impairment of synaptic plastic ...... itic spines via RhoA signaling

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Impairment of synaptic plastic ...... itic spines via RhoA signaling

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Impairment of synaptic plastic ...... itic spines via RhoA signaling

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A H Babayan

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A L Andres

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C M Gall

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E A Kramár

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P2860

Displacement of corticotropin releasing factor from its binding protein as a possible treatment for Alzheimer's disease

AMPA receptor incorporation into synapses during LTP: the role of lateral movement and exocytosis.

The medial temporal lobe and recognition memory

The stressed synapse: the impact of stress and glucocorticoids on glutamate transmission

Novel and transient populations of corticotropin-releasing hormone-expressing neurons in developing hippocampus suggest unique functional roles: a quantitative spatiotemporal analysis

Trafficking and surface expression of hyperpolarization-activated cyclic nucleotide-gated channels in hippocampal neurons

Modulation of learning and anxiety by corticotropin-releasing factor (CRF) and stress: differential roles of CRF receptors 1 and 2

The Rho-specific GEF Lfc interacts with neurabin and spinophilin to regulate dendritic spine morphology

AMPA receptor trafficking and synaptic plasticity

Morphological changes in dendritic spines associated with long-term synaptic plasticity

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10.1038/MP.2012.17

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22411227

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