Memory T cells are anergic to the superantigen staphylococcal enterotoxin B.
about
Defective T cell receptor-mediated signal transduction in memory CD4 T lymphocytes exposed to superantigen or anti-T cell receptor antibodiesStaphylococcal superantigens in colonization and diseaseStaphylococcal enterotoxin B primes cytokine secretion and lytic activity in response to native bacterial antigensThe cell wall and membrane of Cryptococcus neoformans possess a mitogen for human T lymphocytes.Mycobacterium bovis BCG-infected mice are more susceptible to staphylococcal enterotoxin B-mediated toxic shock than uninfected mice despite reduced in vitro splenocyte responses to superantigensBacterial superantigen exposure after resolution of influenza virus infection perturbs the virus-specific memory CD8(+)-T-cell repertoireTolerance to staphylococcal enterotoxin B initiated Th1 cell differentiation in mice infected with Candida albicansStimulation with specific antigen can block superantigen-mediated deletion of T cells in vivo.Proteins in the cell wall and membrane of Cryptococcus neoformans stimulate lymphocytes from both adults and fetal cord blood to proliferateNitric oxide regulates clonal expansion and activation-induced cell death triggered by staphylococcal enterotoxin BSuperantigen-induced CD4 memory T cell anergy. I. Staphylococcal enterotoxin B induces Fyn-mediated negative signalingAnergy in CD4 memory T lymphocytes. II. Abrogation of TCR-induced formation of membrane signaling complexesSelective anergy of V beta 8+ T cells in human immunodeficiency virus-infected individuals.Staphylococcal Enterotoxin B (SEB) Induces Memory CD4 T Cell Anergy in vivo and Impairs Recall Immunity to Unrelated Antigens.The distinct role of CD4+ and CD8+ T-cells during the anti-tumour effects of targeted superantigens.Transcriptional modulation of TCR, Notch and Wnt signaling pathways in SEB-anergized CD4+ T cells.Zinc-binding sites in the N terminus of Mycoplasma arthritidis-derived mitogen permit the dimer formation required for high affinity binding to HLA-DR and for T cell activation.Peripheral T-cell reactivity to bacterial superantigens in vivo: the response/anergy paradox.T-cell activation or tolerization: the Yin and Yang of bacterial superantigens.Proliferation is a prerequisite for bacterial superantigen-induced T cell apoptosis in vivo.Non-responsiveness of antigen-experienced CD4 T cells reflects more stringent co-stimulatory requirements.
P2860
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P2860
Memory T cells are anergic to the superantigen staphylococcal enterotoxin B.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Memory T cells are anergic to the superantigen staphylococcal enterotoxin B.
@ast
Memory T cells are anergic to the superantigen staphylococcal enterotoxin B.
@en
type
label
Memory T cells are anergic to the superantigen staphylococcal enterotoxin B.
@ast
Memory T cells are anergic to the superantigen staphylococcal enterotoxin B.
@en
prefLabel
Memory T cells are anergic to the superantigen staphylococcal enterotoxin B.
@ast
Memory T cells are anergic to the superantigen staphylococcal enterotoxin B.
@en
P2860
P356
P1476
Memory T cells are anergic to the superantigen staphylococcal enterotoxin B.
@en
P2093
P2860
P304
P356
10.1084/JEM.176.2.575
P407
P577
1992-08-01T00:00:00Z