The G protein-activating peptide, mastoparan, and the synthetic NH2-terminal ARF peptide, ARFp13, inhibit in vitro Golgi transport by irreversibly damaging membranes
about
Rab2 is essential for the maturation of pre-Golgi intermediatesStructure and function of ARF proteins: activators of cholera toxin and critical components of intracellular vesicular transport processesArf-1 (ADP-ribosylation factor-1) is involved in the activation of a mammalian Na+-selective currentCell-free transport to distinct Golgi cisternae is compartment specific and ARF independentADP ribosylation factor 1 is required for synaptic vesicle budding in PC12 cells.Intra-Golgi transport inhibition by megalomicin.Regulation of sorting and post-Golgi trafficking of rhodopsin by its C-terminal sequence QVS(A)PA.ADP-ribosylation factor-1 stimulates formation of nascent secretory vesicles from the trans-Golgi network of endocrine cells.Brefeldin A inhibits cell-free, de novo synthesis of poliovirus.Role of heterotrimeric GTP binding proteins in vesicular protein transport: indications for both classical and alternative G protein cycles.Potential roles of heterotrimeric G proteins of the endomembrane system.Regulation and recruitment of phosphatidylinositol 4-kinase on immature secretory granules is independent of ADP-ribosylation factor 1.Evidence that a low-molecular-mass GTP-binding protein is required for store-activated Ca2+ inflow in hepatocytes.Involvement of ADP-ribosylation factor 1 in cholera toxin-induced morphological changes of Chinese hamster ovary cells.In vitro transport on cis and trans sides of the Golgi involves two distinct types of coatomer and ADP-ribosylation factor-independent transport intermediates.Sec7p directs the transitions required for yeast Golgi biogenesis.Cytosolic ADP-ribosylation factors are not required for endosome-endosome fusion but are necessary for GTP gamma S inhibition of fusion.Sar1 translocation onto the ER-membrane for vesicle budding has different pathways for promotion and suppression of ER-to-Golgi transport mediated through H89-sensitive kinase and ER-resident G protein.ER-resident Gi2 protein controls sar1 translocation onto the ER during budding of transport vesicles.
P2860
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P2860
The G protein-activating peptide, mastoparan, and the synthetic NH2-terminal ARF peptide, ARFp13, inhibit in vitro Golgi transport by irreversibly damaging membranes
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年学术文章
@wuu
1994年学术文章
@zh-cn
1994年学术文章
@zh-hans
1994年学术文章
@zh-my
1994年学术文章
@zh-sg
1994年學術文章
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1994年學術文章
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1994年學術文章
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name
The G protein-activating pepti ...... rreversibly damaging membranes
@ast
The G protein-activating pepti ...... rreversibly damaging membranes
@en
type
label
The G protein-activating pepti ...... rreversibly damaging membranes
@ast
The G protein-activating pepti ...... rreversibly damaging membranes
@en
prefLabel
The G protein-activating pepti ...... rreversibly damaging membranes
@ast
The G protein-activating pepti ...... rreversibly damaging membranes
@en
P2860
P356
P1476
The G protein-activating pepti ...... rreversibly damaging membranes
@en
P2093
P2860
P304
P356
10.1083/JCB.127.6.1815
P407
P433
P577
1994-12-01T00:00:00Z