Insulin-like growth factor binding protein-5 modulates muscle differentiation through an insulin-like growth factor-dependent mechanism.
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Netrins and neogenin promote myotube formationFunctional roles and clinical values of insulin-like growth factor-binding protein-5 in different types of cancersLongevity and skeletal muscle mass: the role of IGF signalling, the sirtuins, dietary restriction and protein intakeInsulin-like growth factor-binding protein 5 (Igfbp5) compromises survival, growth, muscle development, and fertility in miceInsulin-like growth factor 1 signaling regulates cytosolic sialidase Neu2 expression during myoblast differentiation and hypertrophyIGFBP-5 regulates muscle cell differentiation by binding to IGF-II and switching on the IGF-II auto-regulation loopGene expression studies of developing bovine longissimus muscle from two different beef cattle breeds.A calcineurin-NFATc3-dependent pathway regulates skeletal muscle differentiation and slow myosin heavy-chain expression.Permissive roles of phosphatidyl inositol 3-kinase and Akt in skeletal myocyte maturationmicroRNA-206 promotes skeletal muscle regeneration and delays progression of Duchenne muscular dystrophy in mice.Role of IGF-I in follistatin-induced skeletal muscle hypertrophy.Insulin-like growth factor (IGF) binding protein-5 blocks skeletal muscle differentiation by inhibiting IGF actionsAge-related changes in miR-143-3p:Igfbp5 interactions affect muscle regeneration.The influence of skeletal muscle on systemic aging and lifespan.Acute mechanical overload increases IGF-I and MMP-9 mRNA in 3D tissue-engineered skeletal muscle.Reduction of myoblast differentiation following multiple population doublings in mouse C2 C12 cells: a model to investigate ageing?Beneficial synergistic interactions of TNF-alpha and IL-6 in C2 skeletal myoblasts--potential cross-talk with IGF system.Mammalian target of rapamycin (mTOR) signaling is required for a late-stage fusion process during skeletal myotube maturation.SOCS-3 induces myoblast differentiation.Insulin-like growth factors (IGF-I and IGF-II) inhibit C2 skeletal myoblast differentiation and enhance TNF alpha-induced apoptosis.Tumor necrosis factor-alpha-induced apoptosis is associated with suppression of insulin-like growth factor binding protein-5 secretion in differentiating murine skeletal myoblasts.IGF-I and retinoic acid regulate the distribution pattern of IGFBPs synthesized by the canine mammary tumor cell line CMT-U335.Decreased steady-state insulin-like growth factor binding protein-3 (IGFBP-3) mRNA level is associated with differentiation of cultured porcine myogenic cells.Maternal vitamin D deficiency causes smaller muscle fibers and altered transcript levels of genes involved in protein degradation, myogenesis, and cytoskeleton organization in the newborn rat.Neprilysin participates in skeletal muscle regeneration and is accumulated in abnormal muscle fibres of inclusion body myositis.
P2860
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P2860
Insulin-like growth factor binding protein-5 modulates muscle differentiation through an insulin-like growth factor-dependent mechanism.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Insulin-like growth factor bin ...... th factor-dependent mechanism.
@ast
Insulin-like growth factor bin ...... th factor-dependent mechanism.
@en
type
label
Insulin-like growth factor bin ...... th factor-dependent mechanism.
@ast
Insulin-like growth factor bin ...... th factor-dependent mechanism.
@en
prefLabel
Insulin-like growth factor bin ...... th factor-dependent mechanism.
@ast
Insulin-like growth factor bin ...... th factor-dependent mechanism.
@en
P2093
P2860
P356
P1476
Insulin-like growth factor bin ...... th factor-dependent mechanism.
@en
P2093
P2860
P304
P356
10.1083/JCB.133.3.683
P407
P577
1996-05-01T00:00:00Z