Zinc dyshomeostasis during polymicrobial sepsis in mice involves zinc transporter Zip14 and can be overcome by zinc supplementation
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Role of Zinc in the Development/Progression of Alcoholic Liver Disease.Immune and metabolic responses in early and late sepsis during mild dietary zinc restriction.Zinc supplementation leads to immune modulation and improved survival in a juvenile model of murine sepsis.Aging amplifies multiple phenotypic defects in mice with zinc transporter Zip14 (Slc39a14) deletion.Zinc Signals and Immunity.Metal Transporter Zip14 (Slc39a14) Deletion in Mice Increases Manganese Deposition and Produces Neurotoxic Signatures and Diminished Motor Activity.Zinc as a Gatekeeper of Immune Function.Recent Advances in the Role of SLC39A/ZIP Zinc Transporters In Vivo.SLC39A14 deficiency alters manganese homeostasis and excretion resulting in brain manganese accumulation and motor deficits in mice.Polymicrobial sepsis influences NK-cell-mediated immunity by diminishing NK-cell-intrinsic receptor-mediated effector responses to viral ligands or infectionsEssential Role of Zinc and Zinc Transporters in Myeloid Cell Function and Host Defense against Infection
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Zinc dyshomeostasis during polymicrobial sepsis in mice involves zinc transporter Zip14 and can be overcome by zinc supplementation
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name
Zinc dyshomeostasis during pol ...... ercome by zinc supplementation
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Zinc dyshomeostasis during pol ...... ercome by zinc supplementation
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type
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Zinc dyshomeostasis during pol ...... ercome by zinc supplementation
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Zinc dyshomeostasis during pol ...... ercome by zinc supplementation
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prefLabel
Zinc dyshomeostasis during pol ...... ercome by zinc supplementation
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Zinc dyshomeostasis during pol ...... ercome by zinc supplementation
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P2860
P356
P1476
Zinc dyshomeostasis during pol ...... ercome by zinc supplementation
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P2093
Inga Wessels
Robert J Cousins
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P304
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10.1152/AJPGI.00179.2015
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2015-08-13T00:00:00Z