Overexpression of SUR2A generates a cardiac phenotype resistant to ischemia. - Wikidata - wikimedia - TriplyDB

Overexpression of SUR2A generates a cardiac phenotype resistant to ischemia.

Overexpression of SUR2A generates a cardiac phenotype resistant to ischemia.

http://www.wikidata.org/entity/Q36238917

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Infection with AV-SUR2A protects H9C2 cells against metabolic stress: a mechanism of SUR2A-mediated cytoprotection independent from the K(ATP) channel activity Cardiomyocyte sulfonylurea receptor 2-KATP channel mediates cardioprotection and ST segment elevation.3'Phosphoinositide-dependent kinase-1 is essential for ischemic preconditioning of the myocardium.Mg2+ protects adult beating cardiomyocytes against ischaemia Nicotinamide-rich diet protects the heart against ischaemia-reperfusion in mice: a crucial role for cardiac SUR2A Quaternary structure of KATP channel SUR2A nucleotide binding domains resolved by synchrotron radiation X-ray scattering.Exposure to 15% oxygen in vivo up-regulates cardioprotective SUR2A without affecting ERK1/2 and AKT: a crucial role for AMPK.Human K(ATP) channelopathies: diseases of metabolic homeostasis.Human oocytes express ATP-sensitive K(+) channels Exercise-induced expression of cardiac ATP-sensitive potassium channels promotes action potential shortening and energy conservation.Muscle KATP channels: recent insights to energy sensing and myoprotection Upregulation of cardioprotective SUR2A by sub-hypoxic drop in oxygen.Advances in cardiac ATP-sensitive K+ channelopathies from molecules to populations.Nicotinamide-rich diet improves physical endurance by up-regulating SUR2A in the heart Remodeling of atrial ATP-sensitive K⁺ channels in a model of salt-induced elevated blood pressure.Sirtuins, aging, and cardiovascular risks.Mild hypoxia in vivo regulates cardioprotective SUR2A: A role for Akt and LDH.AMP-activated protein kinase mediates preconditioning in cardiomyocytes by regulating activity and trafficking of sarcolemmal ATP-sensitive K(+) channels.KATP Channels in the Cardiovascular System.Phenylephrine preconditioning in embryonic heart H9c2 cells is mediated by up-regulation of SUR2B/Kir6.2: A first evidence for functional role of SUR2B in sarcolemmal KATP channels and cardioprotection.Testosterone protects female embryonic heart H9c2 cells against severe metabolic stress by activating estrogen receptors and up-regulating IES SUR2B.Ageing, gender and cardiac sarcolemmal K(ATP) channels.Anti-Apoptotic and Pro-Survival Effect of Alpinate Oxyphyllae Fructus (AOF) in a d-Galactose-Induced Aging Heart.ATP-sensitive potassium channels: metabolic sensing and cardioprotection.KCNJ11 knockout morula re-engineered by stem cell diploid aggregation.Kir6.2 limits Ca(2+) overload and mitochondrial oscillations of ventricular myocytes in response to metabolic stress.Interaction of asymmetric ABCC9-encoded nucleotide binding domains determines KATP channel SUR2A catalytic activity.Proteomic profiling of KATP channel-deficient hypertensive heart maps risk for maladaptive cardiomyopathic outcome.Mice lacking sulfonylurea receptor 2 (SUR2) ATP-sensitive potassium channels are resistant to acute cardiovascular stress.The role of ATP-sensitive potassium channels in cellular function and protection in the cardiovascular system Cardiac sarcolemmal K(ATP) channels: Latest twists in a questing tale!RNA SEQ Analysis Indicates that the AE3 Cl-/HCO3- Exchanger Contributes to Active Transport-Mediated CO2 Disposal in Heart.M-LDH physically associated with sarcolemmal K ATP channels mediates cytoprotection in heart embryonic H9C2 cells A patient suffering from hypokalemic periodic paralysis is deficient in skeletal muscle ATP-sensitive K channels.Limb remote ischemia per-conditioning protects the heart against ischemia-reperfusion injury through the opioid system in rats.Cholesterol-enriched diet inhibits cardioprotection by ATP-sensitive K+ channel activators cromakalim and diazoxide.The beneficial effect of repaglinide on in vitro maturation and development ability of immature mouse oocytes.Cardiomyocyte Specific Deletion of Sirt1 Gene Sensitizes Myocardium to Ischemia and Reperfusion Injury.KATP channel knockout worsens myocardial calcium stress load in vivo and impairs recovery in stunned heart.Ageing-induced decline in physical endurance in mice is associated with decrease in cardiac SUR2A and increase in cardiac susceptibility to metabolic stress: therapeutic prospects for up-regulation of SUR2A.

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P2860

Overexpression of SUR2A generates a cardiac phenotype resistant to ischemia.

http://www.wikidata.org/entity/Q36238917

description

2006 nî lūn-bûn

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2006年论文

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name

Overexpression of SUR2A generates a cardiac phenotype resistant to ischemia.

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Overexpression of SUR2A generates a cardiac phenotype resistant to ischemia.

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type

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Overexpression of SUR2A generates a cardiac phenotype resistant to ischemia.

@ast

Overexpression of SUR2A generates a cardiac phenotype resistant to ischemia.

@en

prefLabel

Overexpression of SUR2A generates a cardiac phenotype resistant to ischemia.

@ast

Overexpression of SUR2A generates a cardiac phenotype resistant to ischemia.

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Overexpression of SUR2A generates a cardiac phenotype resistant to ischemia.

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Aleksandar Jovanović

http://www.w3.org/2001/XMLSchema#string

Allyson Clelland

http://www.w3.org/2001/XMLSchema#string

Andrey Sukhodub

http://www.w3.org/2001/XMLSchema#string

Grant Budas

http://www.w3.org/2001/XMLSchema#string

Karen Phelan

http://www.w3.org/2001/XMLSchema#string

Lorraine Malone

http://www.w3.org/2001/XMLSchema#string

Qingyou Du

http://www.w3.org/2001/XMLSchema#string

Sofija Jovanović

http://www.w3.org/2001/XMLSchema#string

Victoria Murray-Tait

http://www.w3.org/2001/XMLSchema#string

P2860

M-LDH serves as a sarcolemmal K(ATP) channel subunit essential for cell protection against ischemia

Kir6.2 is required for adaptation to stress.

Chronic mild hypoxia protects heart-derived H9c2 cells against acute hypoxia/reoxygenation by regulating expression of the SUR2A subunit of the ATP-sensitive K+ channel

Comparing protein abundance and mRNA expression levels on a genomic scale

Heart mitochondria contain functional ATP-dependent K+ channels

Distribution of Kir6.0 and SUR2 ATP-sensitive potassium channel subunits in isolated ventricular myocytes

ATP-regulated K+ channels in cardiac muscle

Creatine kinase is physically associated with the cardiac ATP-sensitive K+ channel in vivo

A family of sulfonylurea receptors determines the pharmacological properties of ATP-sensitive K+ channels

Syntaxin-1A inhibits cardiac KATP channels by its actions on nucleotide binding folds 1 and 2 of sulfonylurea receptor 2A

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1131-1141

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10.1096/FJ.05-5483COM

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16770012

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2121651

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