Poliovirus infection transiently increases COPII vesicle budding - Wikidata - wikimedia - TriplyDB

Poliovirus infection transiently increases COPII vesicle budding

Poliovirus infection transiently increases COPII vesicle budding

http://www.wikidata.org/entity/Q36246268

about

The Virus-Host Interplay: Biogenesis of +RNA Replication Complexes Endoplasmic Reticulum: The Favorite Intracellular Niche for Viral Replication and Assembly Generation of unique poliovirus RNA replication organelles Long-Range Communication between Different Functional Sites in the Picornaviral 3C Protein.A role for endoplasmic reticulum exit sites in foot-and-mouth disease virus infection Opportunistic intruders: how viruses orchestrate ER functions to infect cells.Rewiring of cellular membrane homeostasis by picornaviruses.Poliovirus-induced changes in cellular membranes throughout infection.The anti-obesity drug orlistat reveals anti-viral activity.A Novel Mechanism Underlying the Innate Immune Response Induction upon Viral-Dependent Replication of Host Cell mRNA: A Mistake of +sRNA Viruses' Replicases.Trafficking and other regulatory mechanisms for organic anion transporting polypeptides and organic anion transporters that modulate cellular drug and xenobiotic influx and that are dysregulated in disease.Enterovirus 71 induces dsRNA/PKR-dependent cytoplasmic redistribution of GRP78/BiP to promote viral replication.ADP ribosylation factor 1 plays an essential role in the replication of a plant RNA virus.

P2860

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P2860

Poliovirus infection transiently increases COPII vesicle budding

http://www.wikidata.org/entity/Q36246268

description

2012 nî lūn-bûn

@nan

2012年の論文

@ja

2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

2012年论文

@zh

2012年论文

@zh-cn

name

Poliovirus infection transiently increases COPII vesicle budding

@ast

Poliovirus infection transiently increases COPII vesicle budding

@en

type

label

Poliovirus infection transiently increases COPII vesicle budding

@ast

Poliovirus infection transiently increases COPII vesicle budding

@en

prefLabel

Poliovirus infection transiently increases COPII vesicle budding

@ast

Poliovirus infection transiently increases COPII vesicle budding

@en

P1433

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P2860

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P356

P1433

Journal of Virology

P1476

Poliovirus infection transiently increases COPII vesicle budding

@en

P2093

Hyung Suk Oh

http://www.w3.org/2001/XMLSchema#string

Jesse C Hay

http://www.w3.org/2001/XMLSchema#string

Meg Trahey

http://www.w3.org/2001/XMLSchema#string

P2860

Mammalian homologues of yeast sec31p. An ubiquitously expressed form is localized to endoplasmic reticulum (ER) exit sites and is essential for ER-Golgi transport

Sec16B is involved in the endoplasmic reticulum export of the peroxisomal membrane biogenesis factor peroxin 16 (Pex16) in mammalian cells

Assembly, organization, and function of the COPII coat

Sec16 defines endoplasmic reticulum exit sites and is required for secretory cargo export in mammalian cells

Hijacking components of the cellular secretory pathway for replication of poliovirus RNA

Two mammalian Sec16 homologues have nonredundant functions in endoplasmic reticulum (ER) export and transitional ER organization

Cellular COPII proteins are involved in production of the vesicles that form the poliovirus replication complex.

Yeast SEC16 gene encodes a multidomain vesicle coat protein that interacts with Sec23p

COPII subunit interactions in the assembly of the vesicle coat.

Sec16p potentiates the action of COPII proteins to bud transport vesicles

P304

9675-9682

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1128/JVI.01159-12

http://www.w3.org/2001/XMLSchema#string

P407

P433

18

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P478

86

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P50

Craig E. Cameron

P577

2012-06-27T00:00:00Z

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P5875

228082515

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22740409

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P932

3446582

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