Cyclophilins facilitate dissociation of the human papillomavirus type 16 capsid protein L1 from the L2/DNA complex following virus entry. - Wikidata - wikimedia - TriplyDB

Cyclophilins facilitate dissociation of the human papillomavirus type 16 capsid protein L1 from the L2/DNA complex following virus entry.

Cyclophilins facilitate dissociation of the human papillomavirus type 16 capsid protein L1 from the L2/DNA complex following virus entry.

http://www.wikidata.org/entity/Q36246390

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Tetraspanin CD151 mediates papillomavirus type 16 endocytosis Papillomavirus Infectious Pathways: A Comparison of Systems Concepts of papillomavirus entry into host cells Large scale RNAi reveals the requirement of nuclear envelope breakdown for nuclear import of human papillomaviruses Direct binding of retromer to human papillomavirus type 16 minor capsid protein L2 mediates endosome exit during viral infection A central region in the minor capsid protein of papillomaviruses facilitates viral genome tethering and membrane penetration for mitotic nuclear entry.Translocation of the papillomavirus L2/vDNA complex across the limiting membrane requires the onset of mitosis The nuclear retention signal of HPV16 L2 protein is essential for incoming viral genome to transverse the trans-Golgi network.Vesicular trafficking of incoming human papillomavirus 16 to the Golgi apparatus and endoplasmic reticulum requires γ-secretase activity.Cyclophilin A associates with enterovirus-71 virus capsid and plays an essential role in viral infection as an uncoating regulator Human papillomavirus species-specific interaction with the basement membrane-resident non-heparan sulfate receptor.Alpha-defensin HD5 inhibits furin cleavage of human papillomavirus 16 L2 to block infection.E6^E7, a novel splice isoform protein of human papillomavirus 16, stabilizes viral E6 and E7 oncoproteins via HSP90 and GRP78.Kallikrein-8 Proteolytically Processes Human Papillomaviruses in the Extracellular Space To Facilitate Entry into Host Cells Naturally Occurring Capsid Protein Variants of Human Papillomavirus Genotype 31 Represent a Single L1 Serotype.A Dual Role for the Nonreceptor Tyrosine Kinase Pyk2 during the Intracellular Trafficking of Human Papillomavirus 16.A Novel PDZ Domain Interaction Mediates the Binding between Human Papillomavirus 16 L2 and Sorting Nexin 27 and Modulates Virion Trafficking Topography of the Human Papillomavirus Minor Capsid Protein L2 during Vesicular Trafficking of Infectious Entry.Use of an in vivo animal model for assessing the role of integrin α(6)β(4) and syndecan-1 in early steps in papillomavirus infection.Identification of a role for the trans-Golgi network in human papillomavirus 16 pseudovirus infection.Is there a role for cyclophilin inhibitors in the management of primary biliary cirrhosis?Genome-wide siRNA screen identifies the retromer as a cellular entry factor for human papillomavirus.HPV virions hitchhike a ride on retromer complexes.The evolving field of human papillomavirus receptor research: a review of binding and entry.Human papillomavirus infections: warts or cancer?Incoming human papillomavirus type 16 genome resides in a vesicular compartment throughout mitosis.Furin Cleavage of L2 during Papillomavirus Infection: Minimal Dependence on Cyclophilins L2, the minor capsid protein of papillomavirus.The papillomavirus major capsid protein L1.Human Papillomavirus Entry: Hiding in a Bubble Multiple heparan sulfate binding site engagements are required for the infectious entry of human papillomavirus type 16.Implication of the Whitefly Bemisia tabaci Cyclophilin B Protein in the Transmission of Tomato yellow leaf curl virus.The Cytoskeletal Adaptor Obscurin-Like 1 Interacts with the Human Papillomavirus 16 (HPV16) Capsid Protein L2 and Is Required for HPV16 Endocytosis.The Role of DCT in HPV16 Infection of HaCaTs The tetraspanin CD151 in papillomavirus infection.Inhibition by cellular vacuolar ATPase impairs human papillomavirus uncoating and infection Host-cell factors involved in papillomavirus entry.Cyclophilins as modulators of viral replication.Virion factors that target Daxx to overcome intrinsic immunity.Coat as a dagger: the use of capsid proteins to perforate membranes during non-enveloped DNA viruses trafficking.

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P2860

Cyclophilins facilitate dissociation of the human papillomavirus type 16 capsid protein L1 from the L2/DNA complex following virus entry.

http://www.wikidata.org/entity/Q36246390

description

2012 nî lūn-bûn

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name

Cyclophilins facilitate dissoc ...... complex following virus entry.

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Cyclophilins facilitate dissoc ...... complex following virus entry.

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Cyclophilins facilitate dissoc ...... complex following virus entry.

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Carlyn Williams

http://www.w3.org/2001/XMLSchema#string

Malgorzata Bienkowska-Haba

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Martin Sapp

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Robert L Garcea

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Seong Man Kim

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P2860

Cyclophilin B is a functional regulator of hepatitis C virus RNA polymerase

Human papillomavirus infection requires cell surface heparan sulfate.

Cleavage of the papillomavirus minor capsid protein, L2, at a furin consensus site is necessary for infection

A membrane-destabilizing peptide in capsid protein L2 is required for egress of papillomavirus genomes from endosomes.

Establishment of papillomavirus infection is enhanced by promyelocytic leukemia protein (PML) expression

Endosome maturation

Entry of human papillomavirus type 16 by actin-dependent, clathrin- and lipid raft-independent endocytosis

Structure of small virus-like particles assembled from the L1 protein of human papillomavirus 16

Structural Basis of Oligosaccharide Receptor Recognition by Human Papillomavirus

Establishment of human papillomavirus infection requires cell cycle progression

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18

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86

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2012-07-03T00:00:00Z

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22761365

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3446629

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