Expression of N-cadherin by human squamous carcinoma cells induces a scattered fibroblastic phenotype with disrupted cell-cell adhesion
about
Epithelial cell adhesion molecule (Ep-CAM) modulates cell-cell interactions mediated by classic cadherinsCadherin-mediated cell sorting not determined by binding or adhesion specificityAdhesion-independent mechanism for suppression of tumor cell invasion by E-cadherin.The juxtamembrane region of the cadherin cytoplasmic tail supports lateral clustering, adhesive strengthening, and interaction with p120ctnDifferential localization of VE- and N-cadherins in human endothelial cells: VE-cadherin competes with N-cadherin for junctional localizationTransforming growth factor-beta and breast cancer: Tumor promoting effects of transforming growth factor-betaN-cadherin dependent collective cell invasion of prostate cancer cells is regulated by the N-terminus of α-cateninThe epidermal growth factor receptor modulates the interaction of E-cadherin with the actin cytoskeletonIQ-domain GTPase-activating protein 1 regulates beta-catenin at membrane ruffles and its role in macropinocytosis of N-cadherin and adenomatous polyposis coliIdentification of an invasive, N-cadherin-expressing epithelial cell type in endometriosis using a new cell culture modelCollagen I-mediated up-regulation of N-cadherin requires cooperative signals from integrins and discoidin domain receptor 1NHERF links the N-cadherin/catenin complex to the platelet-derived growth factor receptor to modulate the actin cytoskeleton and regulate cell motility.EGFR and ADAMs cooperate to regulate shedding and endocytic trafficking of the desmosomal cadherin desmoglein 2.N- and E-cadherins in Xenopus are specifically required in the neural and non-neural ectoderm, respectively, for F-actin assembly and morphogenetic movements.Hepatocyte growth factor/scatter factor induces not only scattering but also cohort migration of human colorectal-adenocarcinoma cells.Computational cell model based on autonomous cell movement regulated by cell-cell signalling successfully recapitulates the "inside and outside" pattern of cell sorting.Interplay between neural-cadherin and vascular endothelial-cadherin in breast cancer progression.Effects of PPAR gamma ligands on TGF-beta1-induced epithelial-mesenchymal transition in alveolar epithelial cells.Vimentin is a potential prognostic factor for tongue squamous cell carcinoma among five epithelial-mesenchymal transition-related proteins.Transendothelial migration of melanoma cells involves N-cadherin-mediated adhesion and activation of the beta-catenin signaling pathwayThe cytoplasmic domain of N-cadherin modulates MMP‑9 induction in oral squamous carcinoma cells.Cadherin and catenin alterations in human cancer.Identification of cadherin 11 as a mediator of dermal fibrosis and possible role in systemic sclerosis.The cadherin switch in ovarian high-grade serous carcinoma is associated with disease progression.Smad7 restricts melanoma invasion by restoring N-cadherin expression and establishing heterotypic cell-cell interactions in vivo.R-cadherin influences cell motility via Rho family GTPases.CLDN3 inhibits cancer aggressiveness via Wnt-EMT signaling and is a potential prognostic biomarker for hepatocellular carcinomaCadherins in neural crest cell development and transformation.Surface expression of precursor N-cadherin promotes tumor cell invasionType 2 cadherins in the human endometrium and placenta: their putative roles in human implantation and placentation.Intrinsic optical biomarkers associated with the invasive potential of tumor cells in engineered tissue models.Cell adhesion molecules and oral cancer.Adhesive mechanisms regulating invasion and metastasis in oral cancer.Phosphoinositide-3 kinase-Rac1-c-Jun NH2-terminal kinase signaling mediates collagen I-induced cell scattering and up-regulation of N-cadherin expression in mouse mammary epithelial cells.N-cadherin and keratinocyte growth factor receptor mediate the functional interplay between Ki-RASG12V and p53V143A in promoting pancreatic cell migration, invasion, and tissue architecture disruption.EMT and stem cell-like properties associated with miR-205 and miR-200 epigenetic silencing are early manifestations during carcinogen-induced transformation of human lung epithelial cells.Mechanisms of tumour invasion and metastasis: emerging targets for therapy.Cell phenotype in normal epithelial cell lines with high endogenous N-cadherin: comparison of RPE to an MDCK subclone.Adhesion-mediated squamous cell carcinoma survival through ligand-independent activation of epidermal growth factor receptor.Changing neighbours, changing behaviour: cell adhesion molecule-mediated signalling during tumour progression.
P2860
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P2860
Expression of N-cadherin by human squamous carcinoma cells induces a scattered fibroblastic phenotype with disrupted cell-cell adhesion
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
Expression of N-cadherin by hu ...... h disrupted cell-cell adhesion
@ast
Expression of N-cadherin by hu ...... h disrupted cell-cell adhesion
@en
type
label
Expression of N-cadherin by hu ...... h disrupted cell-cell adhesion
@ast
Expression of N-cadherin by hu ...... h disrupted cell-cell adhesion
@en
prefLabel
Expression of N-cadherin by hu ...... h disrupted cell-cell adhesion
@ast
Expression of N-cadherin by hu ...... h disrupted cell-cell adhesion
@en
P2093
P2860
P356
P1476
Expression of N-cadherin by hu ...... h disrupted cell-cell adhesion
@en
P2093
P2860
P304
P356
10.1083/JCB.135.6.1643
P407
P433
P577
1996-12-01T00:00:00Z