IL-21R signaling is critical for induction of spontaneous experimental autoimmune encephalomyelitis.
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A Context-Dependent Role for IL-21 in Modulating the Differentiation, Distribution, and Abundance of Effector and Memory CD8 T Cell Subsets.Interleukin-21 is associated with the severity of psoriasis vulgaris through promoting CD4+ T cells to differentiate into Th17 cellsTh17 cells in neuromyelitis optica spectrum disorder: a review.IL-21 and T Cell Differentiation: Consider the Context.Deficiency of the immunostimulatory cytokine IL-21 promotes intestinal neoplasia via dysregulation of the Th1/Th17 axis.Monoclonal Antibodies in Preclinical EAE Models of Multiple Sclerosis: A Systematic Review.CTL-Promoting Effects of IL-21 Counteract Murine Lupus in the Parent→F1 Graft-versus-Host Disease Model.The Interleukin (IL)-23/T helper (Th)17 Axis in Experimental Autoimmune Encephalomyelitis and Multiple Sclerosis.
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P2860
IL-21R signaling is critical for induction of spontaneous experimental autoimmune encephalomyelitis.
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2015 nî lūn-bûn
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2015年学术文章
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name
IL-21R signaling is critical f ...... autoimmune encephalomyelitis.
@ast
IL-21R signaling is critical f ...... autoimmune encephalomyelitis.
@en
type
label
IL-21R signaling is critical f ...... autoimmune encephalomyelitis.
@ast
IL-21R signaling is critical f ...... autoimmune encephalomyelitis.
@en
prefLabel
IL-21R signaling is critical f ...... autoimmune encephalomyelitis.
@ast
IL-21R signaling is critical f ...... autoimmune encephalomyelitis.
@en
P2093
P2860
P356
P1476
IL-21R signaling is critical f ...... autoimmune encephalomyelitis.
@en
P2093
Guangxiang Gu
Meike Mitsdoerffer
Raymond A Sobel
Sheng Xiao
Vijay K Kuchroo
Youjin Lee
P2860
P304
P356
10.1172/JCI75933
P407
P577
2015-09-28T00:00:00Z