Viral modulation of antigen presentation: manipulation of cellular targets in the ER and beyond.
about
A ubiquitin ligase-associated chaperone holdase maintains polypeptides in soluble states for proteasome degradationInhibition of p97-dependent protein degradation by Eeyarestatin IThe orthology of HLA-E and H2-Qa1 is hidden by their concerted evolution with other MHC class I molecules.Structural Mechanism of ER Retrieval of MHC Class I by CowpoxOrganizing principles of mammalian nonsense-mediated mRNA decayKaposi's sarcoma-associated herpesvirus K7 induces viral G protein-coupled receptor degradation and reduces its tumorigenicityTMEM129 is a Derlin-1 associated ERAD E3 ligase essential for virus-induced degradation of MHC-I.The structure of mouse cytomegalovirus m04 protein obtained from sparse NMR data reveals a conserved fold of the m02-m06 viral immune modulator family.Bovine herpesvirus 1 UL49.5 protein inhibits the transporter associated with antigen processing despite complex formation with glycoprotein M.Varicellovirus UL 49.5 proteins differentially affect the function of the transporter associated with antigen processing, TAP.Stage-specific inhibition of MHC class I presentation by the Epstein-Barr virus BNLF2a protein during virus lytic cycle.Immune evasion proteins of murine cytomegalovirus preferentially affect cell surface display of recently generated peptide presentation complexes.Cleavage by signal peptide peptidase is required for the degradation of selected tail-anchored proteins.Novel Drosophila viruses encode host-specific suppressors of RNAiHIV-1 Nef binds a subpopulation of MHC-I throughout its trafficking itinerary and down-regulates MHC-I by perturbing both anterograde and retrograde trafficking.Influenza A virus infection of human primary dendritic cells impairs their ability to cross-present antigen to CD8 T cells.Major histocompatibility complex class I downregulation induced by equine herpesvirus type 1 pUL56 is through dynamin-dependent endocytosis.Viral and cellular MARCH ubiquitin ligases and cancer.The C-terminal amino acid of the MHC-I heavy chain is critical for binding to Derlin-1 in human cytomegalovirus US11-induced MHC-I degradation.Cytomegalovirus encodes a positive regulator of antigen presentationInhibition of heavy chain and beta2-microglobulin synthesis as a mechanism of major histocompatibility complex class I downregulation during Epstein-Barr virus replicationHost shutoff during productive Epstein-Barr virus infection is mediated by BGLF5 and may contribute to immune evasion.A CD8+ T cell immune evasion protein specific to Epstein-Barr virus and its close relatives in Old World primates.Hiding lipid presentation: viral interference with CD1d-restricted invariant natural killer T (iNKT) cell activationIdentification of hot spots in the variola virus complement inhibitor (SPICE) for human complement regulation.Contribution of mass spectrometry-based proteomics to immunology.Inflammation in common variable immunodeficiency is associated with a distinct CD8(+) response to cytomegalovirus.The protective and destructive roles played by molecular chaperones during ERAD (endoplasmic-reticulum-associated degradation).The immune evasion paradox: immunoevasins of murine cytomegalovirus enhance priming of CD8 T cells by preventing negative feedback regulation.Decoupling the role of ubiquitination for the dislocation versus degradation of major histocompatibility complex (MHC) class I proteins during endoplasmic reticulum-associated degradation (ERAD)Dendritic cells: driving the differentiation programme of T cells in viral infections.Viral proteins interfering with antigen presentation target the major histocompatibility complex class I peptide-loading complex.Rat cytomegalovirus infection depletes MHC II in bone marrow derived dendritic cells.IL-15 functions as a danger signal to regulate tissue-resident T cells and tissue destructionIntracellular assembly and trafficking of MHC class I molecules.Structure and function of murine cytomegalovirus MHC-I-like molecules: how the virus turned the host defense to its advantage.Disulfide formation in the ER and mitochondria: two solutions to a common process.Molecular mechanisms of viral immune evasion proteins to inhibit MHC class I antigen processing and presentation.Plasticity in programming of effector and memory CD8 T-cell formation.Exploiting human herpesvirus immune evasion for therapeutic gain: potential and pitfalls.
P2860
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P2860
Viral modulation of antigen presentation: manipulation of cellular targets in the ER and beyond.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Viral modulation of antigen pr ...... targets in the ER and beyond.
@ast
Viral modulation of antigen pr ...... targets in the ER and beyond.
@en
type
label
Viral modulation of antigen pr ...... targets in the ER and beyond.
@ast
Viral modulation of antigen pr ...... targets in the ER and beyond.
@en
prefLabel
Viral modulation of antigen pr ...... targets in the ER and beyond.
@ast
Viral modulation of antigen pr ...... targets in the ER and beyond.
@en
P2860
P1476
Viral modulation of antigen pr ...... targets in the ER and beyond.
@en
P2093
Brendan N Lilley
Hidde L Ploegh
P2860
P304
P356
10.1111/J.0105-2896.2005.00318.X
P577
2005-10-01T00:00:00Z