Continuous treatment with FTS confers resistance to apoptosis and affects autophagy.

about

Continuous treatment with FTS confers resistance to apoptosis and affects autophagy.

Item

http://www.wikidata.org/entity/Q36267411

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh

2017年學術文章

@zh-hant

name

Continuous treatment with FTS confers resistance to apoptosis and affects autophagy.

@ast

Continuous treatment with FTS confers resistance to apoptosis and affects autophagy.

@en

type

Item

label

Continuous treatment with FTS confers resistance to apoptosis and affects autophagy.

@ast

Continuous treatment with FTS confers resistance to apoptosis and affects autophagy.

@en

prefLabel

Continuous treatment with FTS confers resistance to apoptosis and affects autophagy.

@ast

Continuous treatment with FTS confers resistance to apoptosis and affects autophagy.

@en

P1476

Continuous treatment with FTS confers resistance to apoptosis and affects autophagy.

@en

P2093

Eran Schmukler

http://www.w3.org/2001/XMLSchema#string

Eya Wolfson

http://www.w3.org/2001/XMLSchema#string

Yoel Kloog

http://www.w3.org/2001/XMLSchema#string

Zvulun Elazar

http://www.w3.org/2001/XMLSchema#string

P2860

Guidelines for the use and interpretation of assays for monitoring autophagy

Mitophagy: mechanisms, pathophysiological roles, and analysis

Cancer statistics, 2010

Targeting RAS signalling pathways in cancer therapy

5-fluorouracil: mechanisms of action and clinical strategies

Autophagy and signaling: their role in cell survival and cell death

Ras oncogenes: split personalities

Circumventing cancer drug resistance in the era of personalized medicine.

The Ras inhibitor farnesylthiosalicylic acid (Salirasib) disrupts the spatiotemporal localization of active Ras: a potential treatment for cancer.

Inhibition of mitotic Aurora kinase A by alisertib induces apoptosis and autophagy of human gastric cancer AGS and NCI-N78 cells.

P304

e0171351

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1371/JOURNAL.PONE.0171351

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Ronit Pinkas-Kramarski

P577

2017-02-02T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28151959

http://www.w3.org/2001/XMLSchema#string

P921

autophagy

P932

5289601

http://www.w3.org/2001/XMLSchema#string