The immunology of experimental Chagas' disease. 3. Rejection of allogeneic heart cells in vitro. - Wikidata - wikimedia - TriplyDB

The immunology of experimental Chagas' disease. 3. Rejection of allogeneic heart cells in vitro.

The immunology of experimental Chagas' disease. 3. Rejection of allogeneic heart cells in vitro.

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Pathogenesis of chagas' disease: parasite persistence and autoimmunity Chagas disease Heat-killed Trypanosoma cruzi induces acute cardiac damage and polyantigenic autoimmunity Trypanosoma cruzi in the chicken model: Chagas-like heart disease in the absence of parasitism.Immunization of mice with Trypanosoma cruzi polyribosomes.Thymus-dependent control of host defense mechanisms against Trypanosoma cruzi infection.Adoptive transfer of protection against Trypanosoma cruzi with lymphocytes and macrophages.Chagas disease. What is known and what should be improved: a systemic review.The effect of interleukin-2 on parasitemia and myocarditis in experimental Chagas' disease.Inhibition of autoimmune Chagas-like heart disease by bone marrow transplantation Autoimmunity in Chagas' disease: specific inhibition of reactivity of CD4+ T cells against myosin in mice chronically infected with Trypanosoma cruzi.Tropical diseases encountered in Canada: 1. Chagas' disease.Enhancement of chronic Trypanosoma cruzi myocarditis in dogs treated with low doses of cyclophosphamide.The immunology of experimental Chagas' disease. IV. Production of lesions in rabbits similar to those of chronic Chagas' disease in man.Group A streptococcal antigens cross-reactive with myocardium. Purification of heart-reactive antibody and isolation and characterization of the streptococcal antigen.Induction of cardiac autoimmunity in Chagas heart disease: a case for molecular mimicry.Chagasic cardiopathy. Immunopathologic and morphologic studies in myocardial biopsies.Prevalence of gallstones in 1,229 patients submitted to surgical laparoscopic treatment of GERD and esophageal achalasia: associated cholecystectomy was a safe procedure.Evolution and pathology in chagas disease--a review.Antiheart antibody-dependent cytotoxicity in the sera of mice chronically infected with Trypanosoma cruzi.Do self-heart-reactive T cells expand in Trypanosoma cruzi-immune hosts?Elaboration by mammalian mesenchymal cells infected with Trypanosoma cruzi of a fibroblast-stimulating factor that may contribute to chagasic cardiomyopathy.Acquired cell-mediated immunodepression in acute Chagas' disease Do lymphocytes from Chagasic patients respond to heart antigens?Longitudinal study of immune response in human Chagas' disease.Chagas heart disease pathogenesis: one mechanism or many?Cellular immunity in chronic Chagas' disease.Chronic Trypanosoma cruzi-elicited cardiomyopathy: from the discovery to the proposal of rational therapeutic interventions targeting cell adhesion molecules and chemokine receptors--how to make a dream come true.Autoimmune pathogenesis of Chagas heart disease: looking back, looking ahead.Chagas' disease: trends in immunological research and prospects for immunoprophylaxis.Immunopathologic and morphologic studies of skeletal muscle in Chagas' disease.Delayed hypersensitivity to heart antigens in Chagas' disease as measured by in vitro lymphocyte stimulation.Immunopathology of experimental Chagas' disease: binding of T cells to Trypanosoma cruzi-infected heart tissue.Depletion of regulatory T cells decreases cardiac parasitosis and inflammation in experimental Chagas disease.Chronic chagasic cardiopathy: the product of a turbulent host-parasite relationship.Cardiac M(2) muscarinic cholinoceptor activation by human chagasic autoantibodies: association with bradycardia.Cardiac damage induced by immunization with heat-killed Trypanosoma cruzi is not antibody mediated.Effects of severe protein restriction in levels of parasitemia and in mortality of mice acutely infected with Trypanosoma cruzi.Polyclonal Lymphocyte Responses to Murine Trypanosoma cruzi Infection Polyclonal Lymphocyte Responses to Murine Trypanosoma cruzi Infection

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The immunology of experimental Chagas' disease. 3. Rejection of allogeneic heart cells in vitro.

http://www.wikidata.org/entity/Q36273596

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1974 nî lūn-bûn

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Santos-Buch CA

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Protein measurement with the Folin phenol reagent

IMMUNOLOGIC RELATION OF STREPTOCOCCAL AND TISSUE ANTIGENS. III. PRESENCE IN HUMAN SERA OF STREPTOCOCCAL ANTIBODY CROSS-REACTIVE WITH HEART TISSUE. ASSOCIATION WITH STREPTOCOCCAL INFECTION, RHEUMATIC FEVER, AND GLOMERULONEPHRITIS.

QUANTITATIVE STUDIES ON THE BEHAVIOR OF SENSITIZED LYMPHOCYTES IN VITRO. II. INHIBITORY INFLUENCE OF THE IMMUNE SUPPRESSOR, IMURAN, ON THE DESTRUCTIVE REACTION OF SENSITIZED LYMPHOID CELLS AGAINST HOMOLOGOUS TARGET CELLS

Suppression of delayed hypersensitivity in vitro by inhibition of protein synthesis

Studies on the mechanism of the cytolytic effect of sensitized lymphocytes.

Lysis of homologous cells by sensitized lymphocytes in tissue culture.

Cytotoxicity of human lymphocytes: antagonism between inducing processes.

Studies of allograft immunity in mice. II. Mechanism of target cell inactivation in vitro by sensitized lymphocytes.

Lymphocyte in vitro cytotoxicity: specific release of lymphotoxin-like materials from tuberculin-sensitive lymphoid cells.

Quantitative assay of the lytic action of immune lymphoid cells on 51-Cr-labelled allogeneic target cells in vitro; inhibition by isoantibody and by drugs.

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