Polyoma middle T-induced vascular tumor formation: the role of the plasminogen activator/plasmin system.
about
Common and specific effects of TIE2 mutations causing venous malformationsParallel in vivo and in vitro selection using phage display identifies protease-dependent tumor-targeting peptides.Mechanisms of cell transformation induced by polyomavirus.PAI-1 leads to G1-phase cell-cycle progression through cyclin D3/cdk4/6 upregulationTargeting plasminogen activator inhibitor-1 inhibits angiogenesis and tumor growth in a human cancer xenograft model.Differential sensitivity of mouse epithelial tissues to the polyomavirus middle T oncogene.Membrane associated proteases and their inhibitors in tumour angiogenesis.The development of bleomycin-induced pulmonary fibrosis in mice deficient for components of the fibrinolytic systemReceptor-independent role of urokinase-type plasminogen activator in pericellular plasmin and matrix metalloproteinase proteolysis during vascular wound healing in mice.Development of a Mammalian suspension culture for expression of active recombinant human urokinase-type plasminogen activator.New functions of the fibrinolytic system in bone marrow cell-derived angiogenesis.PF573,228 inhibits vascular tumor cell growth, migration as well as angiogenesis, induces apoptosis and abrogates PRAS40 and S6RP phosphorylation.Glucocorticoids increase VE-cadherin expression and cause cytoskeletal rearrangements in murine brain endothelial cEND cells.Dexamethasone induces the expression of metalloproteinase inhibitor TIMP-1 in the murine cerebral vascular endothelial cell line cEND.Glucocorticoid effects on mouse microvascular endothelial barrier permeability are brain specific.Species-specific urokinase receptor ligands reduce glioma growth and increase survival primarily by an antiangiogenesis mechanism.Suppression of metastatic hemangiosarcoma by a parvovirus MVMp vector transducing the IP-10 chemokine into immunocompetent mice.Human endothelial cells expressing polyoma middle T induce tumors.HT-1080 fibrosarcoma cell matrix degradation and invasion are inhibited by the matrix-associated serine protease inhibitor TFPI-2/33 kDa MSPI.Inhibition of hemangioma development in a syngeneic mouse model correlates with bcl-2 suppression and the inhibition of Akt kinase activity.Formation of transformed endothelial cells in the absence of VEGFR-2/Flk-1 by Polyoma middle T oncogene.
P2860
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P2860
Polyoma middle T-induced vascular tumor formation: the role of the plasminogen activator/plasmin system.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh
1997年學術文章
@zh-hant
name
Polyoma middle T-induced vascu ...... ogen activator/plasmin system.
@ast
Polyoma middle T-induced vascu ...... ogen activator/plasmin system.
@en
type
label
Polyoma middle T-induced vascu ...... ogen activator/plasmin system.
@ast
Polyoma middle T-induced vascu ...... ogen activator/plasmin system.
@en
prefLabel
Polyoma middle T-induced vascu ...... ogen activator/plasmin system.
@ast
Polyoma middle T-induced vascu ...... ogen activator/plasmin system.
@en
P2093
P2860
P356
P1476
Polyoma middle T-induced vascu ...... ogen activator/plasmin system.
@en
P2093
Carmeliet P
Montesano R
Möhle-Steinlein U
Sabapathy KT
Tacchini-Cottier F
P2860
P304
P356
10.1083/JCB.137.4.953
P407
P577
1997-05-01T00:00:00Z